Bei Wu

1.7k total citations
23 papers, 1.3k citations indexed

About

Bei Wu is a scholar working on Molecular Biology, Physiology and Neurology. According to data from OpenAlex, Bei Wu has authored 23 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 6 papers in Physiology and 5 papers in Neurology. Recurrent topics in Bei Wu's work include Prion Diseases and Protein Misfolding (8 papers), Neurological diseases and metabolism (5 papers) and Alzheimer's disease research and treatments (5 papers). Bei Wu is often cited by papers focused on Prion Diseases and Protein Misfolding (8 papers), Neurological diseases and metabolism (5 papers) and Alzheimer's disease research and treatments (5 papers). Bei Wu collaborates with scholars based in United States, China and Australia. Bei Wu's co-authors include Jie Shen, Shumin Duan, Chen Zhang, Ioannis Dragatsis, Mary Wines-Samuelson, David A. Harris, Thomas C. Südhof, Dawei Zhang, Vassilios Beglopoulos and Hiroo Yamaguchi and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Bei Wu

23 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Bei Wu United States 16 650 458 430 196 193 23 1.3k
M. T. Ciotti Italy 19 1.0k 1.6× 442 1.0× 939 2.2× 169 0.9× 125 0.6× 28 1.8k
Melvin L. Billingsley United States 22 1.0k 1.6× 868 1.9× 694 1.6× 302 1.5× 277 1.4× 46 2.2k
Valérie Petegnief Spain 18 504 0.8× 531 1.2× 281 0.7× 134 0.7× 66 0.3× 31 1.3k
Rian de Laat United States 10 385 0.6× 456 1.0× 476 1.1× 149 0.8× 925 4.8× 11 1.6k
Takao Makifuchi Japan 21 929 1.4× 611 1.3× 613 1.4× 205 1.0× 554 2.9× 55 1.8k
Paula E. Jarvie Australia 13 794 1.2× 174 0.4× 772 1.8× 282 1.4× 78 0.4× 15 1.2k
Tetsuro Murakami Japan 27 784 1.2× 671 1.5× 504 1.2× 257 1.3× 937 4.9× 79 2.2k
Esteban Montejo de Garcini Spain 17 778 1.2× 856 1.9× 316 0.7× 343 1.8× 154 0.8× 24 1.3k
M L Billingsley United States 19 1.1k 1.6× 291 0.6× 584 1.4× 745 3.8× 154 0.8× 31 1.8k
Cinzia Galli Italy 10 920 1.4× 227 0.5× 590 1.4× 117 0.6× 46 0.2× 13 1.4k

Countries citing papers authored by Bei Wu

Since Specialization
Citations

This map shows the geographic impact of Bei Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bei Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bei Wu more than expected).

Fields of papers citing papers by Bei Wu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bei Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bei Wu. The network helps show where Bei Wu may publish in the future.

Co-authorship network of co-authors of Bei Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Bei Wu. A scholar is included among the top collaborators of Bei Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bei Wu. Bei Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wu, Bei, et al.. (2024). Abnormal synaptic architecture in iPSC-derived neurons from a multi-generational family with genetic Creutzfeldt-Jakob disease. Stem Cell Reports. 19(10). 1474–1488. 3 indexed citations
2.
Li, Pengtao, Zhihao Sun, Yu Chen, et al.. (2024). Integrated Transcriptomic and Metabolomic Analysis of G. hirsutum and G. barbadense Responses to Verticillium Wilt Infection. International Journal of Molecular Sciences. 26(1). 28–28. 1 indexed citations
3.
Tao, Lizhi, Bei Wu, M. Jake Pushie, et al.. (2020). Both N-Terminal and C-Terminal Histidine Residues of the Prion Protein Are Essential for Copper Coordination and Neuroprotective Self-Regulation. Journal of Molecular Biology. 432(16). 4408–4425. 32 indexed citations
4.
Roseman, Graham, et al.. (2020). Intrinsic toxicity of the cellular prion protein is regulated by its conserved central region. The FASEB Journal. 34(6). 8734–8748. 5 indexed citations
5.
McDonald, Alex J., Deborah R. Leon, Bei Wu, et al.. (2019). Altered Domain Structure of the Prion Protein Caused by Cu2+ Binding and Functionally Relevant Mutations: Analysis by Cross-Linking, MS/MS, and NMR. Structure. 27(6). 907–922.e5. 27 indexed citations
6.
Cheng, Fang, et al.. (2018). Prions activate a p38 MAPK synaptotoxic signaling pathway. PLoS Pathogens. 14(9). e1007283–e1007283. 50 indexed citations
7.
Wu, Bei, et al.. (2018). Prion neurotoxicity. Brain Pathology. 29(2). 263–277. 23 indexed citations
8.
Wu, Bei, Alex J. McDonald, Celeste B. Rich, et al.. (2017). The N-terminus of the prion protein is a toxic effector regulated by the C-terminus. eLife. 6. 59 indexed citations
9.
Xia, Dan, Hirotaka Watanabe, Bei Wu, et al.. (2015). Presenilin-1 Knockin Mice Reveal Loss-of-Function Mechanism for Familial Alzheimer’s Disease. Neuron. 85(5). 967–981. 171 indexed citations
10.
Zhang, Zhijun, Sandra Almeida, Yubing Lu, et al.. (2013). Downregulation of MicroRNA-9 in iPSC-Derived Neurons of FTD/ALS Patients with TDP-43 Mutations. PLoS ONE. 8(10). e76055–e76055. 111 indexed citations
11.
Wu, Bei, Hiroo Yamaguchi, F. Anthony Lai, & Jie Shen. (2013). Presenilins regulate calcium homeostasis and presynaptic function via ryanodine receptors in hippocampal neurons. Proceedings of the National Academy of Sciences. 110(37). 15091–15096. 96 indexed citations
12.
Barth, Sandra, Uwe Konietzko, Bei Wu, et al.. (2013). Dysregulation of Hypoxia-Inducible Factor by Presenilin/γ-Secretase Loss-of-Function Mutations. Journal of Neuroscience. 33(5). 1915–1926. 20 indexed citations
13.
Zhang, Chen, Bei Wu, Vassilios Beglopoulos, et al.. (2009). Presenilins are essential for regulating neurotransmitter release. Nature. 460(7255). 632–636. 236 indexed citations
14.
Kitada, Tohru, Antonio Pisani, Marián Haburčák, et al.. (2009). Impaired dopamine release and synaptic plasticity in the striatum of Parkin−/− mice. Journal of Neurochemistry. 110(2). 613–621. 100 indexed citations
15.
Yang, Yue, Albert H. Kim, Tomoko Yamada, et al.. (2009). A Cdc20-APC Ubiquitin Signaling Pathway Regulates Presynaptic Differentiation. Science. 326(5952). 575–578. 100 indexed citations
16.
Shen, Wanhua, Bei Wu, Zhijun Zhang, et al.. (2006). Activity-Induced Rapid Synaptic Maturation Mediated by Presynaptic Cdc42 Signaling. Neuron. 50(3). 401–414. 108 indexed citations
17.
Martin, Lisandra L., Gary D. Fallon, & Bei Wu. (2004). Ammonium heptanitrosyltrithiotetraferrate monohydrate. Acta Crystallographica Section E Structure Reports Online. 60(3). i37–i39. 5 indexed citations
18.
Martin, Lisandra L., et al.. (2001). An extrusion strategy for the FeMo cofactor from nitrogenase.. European Journal of Biochemistry. 268(22). 5676–5686. 7 indexed citations
19.
McCarrick, Mary, Bei Wu, Stephen J. Harris, et al.. (1993). Chromogenic ligands for lithium based on calix[4]arene tetraesters bearing nitrophenol residues. Journal of the Chemical Society Perkin Transactions 2. 1963–1963. 23 indexed citations
20.
McCarrick, Mary, et al.. (1992). Novel chromogenic ligands for lithium and sodium based on calix[4]arene tetraesters. Journal of the Chemical Society Chemical Communications. 1287–1287. 35 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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