Beate Leo‐Kottler

2.1k total citations · 1 hit paper
28 papers, 1.5k citations indexed

About

Beate Leo‐Kottler is a scholar working on Molecular Biology, Ophthalmology and Clinical Biochemistry. According to data from OpenAlex, Beate Leo‐Kottler has authored 28 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 7 papers in Ophthalmology and 5 papers in Clinical Biochemistry. Recurrent topics in Beate Leo‐Kottler's work include Mitochondrial Function and Pathology (21 papers), ATP Synthase and ATPases Research (14 papers) and Retinal Development and Disorders (6 papers). Beate Leo‐Kottler is often cited by papers focused on Mitochondrial Function and Pathology (21 papers), ATP Synthase and ATPases Research (14 papers) and Retinal Development and Disorders (6 papers). Beate Leo‐Kottler collaborates with scholars based in Germany, United Kingdom and Australia. Beate Leo‐Kottler's co-authors include Bernd Wissinger, Georg Auburger, Ulrich Kellner, Marcela Votruba, Christiane Alexander, Miguel Rodríguez, Dawn L. Thiselton, Anthony T. Moore, Simone Mayer and Dorothea Besch and has published in prestigious journals such as Nature Genetics, Biochemical and Biophysical Research Communications and American Journal of Ophthalmology.

In The Last Decade

Beate Leo‐Kottler

27 papers receiving 1.5k citations

Hit Papers

OPA1, encoding a dynamin-related GTPase, is mutated in au... 2000 2026 2008 2017 2000 250 500 750 1000

Peers

Beate Leo‐Kottler
Comparison fields: 5 of 66
  • Molecular Biology 1.4k
  • Clinical Biochemistry 469
  • Ophthalmology 203
  • Cellular and Molecular Neuroscience 169
  • Physiology 116
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Jean‐Michel Griffoin France View profile →
Citations per field, relative to Beate Leo‐Kottler
Beate Leo‐Kottler · 1×
Citations per year, relative to Beate Leo‐Kottler
Beate Leo‐Kottler · 1×

Countries citing papers authored by Beate Leo‐Kottler

Since Specialization
Citations

This map shows the geographic impact of Beate Leo‐Kottler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beate Leo‐Kottler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beate Leo‐Kottler more than expected).

Fields of papers citing papers by Beate Leo‐Kottler

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Beate Leo‐Kottler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beate Leo‐Kottler. The network helps show where Beate Leo‐Kottler may publish in the future.

Co-authorship network of co-authors of Beate Leo‐Kottler

This figure shows the co-authorship network connecting the top 25 collaborators of Beate Leo‐Kottler. A scholar is included among the top collaborators of Beate Leo‐Kottler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Beate Leo‐Kottler. Beate Leo‐Kottler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 16
2 14
3 3
4 15
5 42
6 0
7 3
8 18
9 1
10 22
11 13
12 22
13
OPA1, encoding a dynamin-related GTPase, is mutated in autosomal dominant optic atrophy linked to chromosome 3q28 breakdown →
1033
14 19
15 26
16 8
17 6
18 25
19 2
20 48

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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