Barton G. Weick

544 total citations
16 papers, 470 citations indexed

About

Barton G. Weick is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Physiology. According to data from OpenAlex, Barton G. Weick has authored 16 papers receiving a total of 470 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Cellular and Molecular Neuroscience, 6 papers in Molecular Biology and 5 papers in Physiology. Recurrent topics in Barton G. Weick's work include Neurotransmitter Receptor Influence on Behavior (8 papers), Receptor Mechanisms and Signaling (4 papers) and Parkinson's Disease Mechanisms and Treatments (4 papers). Barton G. Weick is often cited by papers focused on Neurotransmitter Receptor Influence on Behavior (8 papers), Receptor Mechanisms and Signaling (4 papers) and Parkinson's Disease Mechanisms and Treatments (4 papers). Barton G. Weick collaborates with scholars based in United States. Barton G. Weick's co-authors include Judith R. Walters, Raymond M. Quock, Sue Ritter, Thomas N. Chase, A. Horita, Justine R. Smith, Zvi Susel, Thomas M. Engber, Joanne H. Carlson and Debra A. Bergstrom and has published in prestigious journals such as Brain Research, Life Sciences and Neurobiology of Aging.

In The Last Decade

Barton G. Weick

16 papers receiving 454 citations

Peers

Countries citing papers authored by Barton G. Weick

Since Specialization

Citations

This map shows the geographic impact of Barton G. Weick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Barton G. Weick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Barton G. Weick more than expected).

Fields of papers citing papers by Barton G. Weick

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Barton G. Weick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Barton G. Weick. The network helps show where Barton G. Weick may publish in the future.

Co-authorship network of co-authors of Barton G. Weick

This figure shows the co-authorship network connecting the top 25 collaborators of Barton G. Weick. A scholar is included among the top collaborators of Barton G. Weick based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Barton G. Weick. Barton G. Weick is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

#	Title	Journal	Authors	Indexed citations
1	Intravenous administration of L-kynurenine to rhesus monkeys: Effect on quinolinate and kynurenate levels in serum and cerebrospinal fluid	Neuropharmacology	Vimala H. Sethy, Barton G. Weick et al.	23
2	Effects of chronic Levodopa on D1 and D2 receptor-mediated striatal output	Neurochemistry International	Thomas M. Engber, Zvi Susel et al.	4
3	2-Amino-3-(methylamino)propanoic acid (BMAA) bioavailability in the primate	Neurobiology of Aging	Mark W. Duncan, Barton G. Weick et al.	29
4	Responses of substantia nigra pars reticulata neurons to GABA and SKF 38393 in 6-hydroxydopamine-lesioned rats are differentially affected by continuous and intermittent levodopa administration	Brain Research	Barton G. Weick, Thomas M. Engber et al.	52
5	Neurophysiological Examination of the Role of D-1 Dopamine Receptors in the Regulation of Neuronal Activity in the Basal Ganglia	Advances in experimental medicine and biology	Judith R. Walters, Joanne H. Carlson et al.	2
6	Neurophysiological investigation of effects of the D‐1 agonist SKF 38393 on tonic activity of substantia nigra dopamine neurons	Synapse	Joanne H. Carlson, Debra A. Bergstrom et al.	37
7	D-1 dopamine receptor stimulation potentiates neurophysiological effects of bromocriptine in rats with lesions of the nigrostriatal dopamine pathway	Neuropharmacology	Barton G. Weick, Judith R. Walters	24
8	Effects of D1 and D2 dopamine receptor stimulation on the activity of substantia nigra pars reticulata neurons in 6-hydroxydopamine lesioned rats: D1/D2 coactivation induces potentiated responses	Brain Research	Barton G. Weick, Judith R. Walters	133
9	Stimulation of insulin release and suppression of feeding by hepatic portal glucagon infusion in rats	Physiology & Behavior	Barton G. Weick, Sue Ritter	8
10	Dose-related suppression of feeding by intraportal glucagon infusion in the rat	American Journal of Physiology-Regulatory, Integrative and Comparative Physiology	Barton G. Weick, Sue Ritter	22
11	Plasma catecholamines in fasted and sucrose supplemented rats	Physiology & Behavior	Barton G. Weick, Sue Ritter et al.	21
12	Fasting reduces cardiac beta-adrenergic receptor concentration and ornithine decarboxylase activity in rats	Journal of the Autonomic Nervous System	Barton G. Weick, Sue Ritter	5
13	Plasma catecholamines: Exaggerated elevation is associated with stress susceptibility	Physiology & Behavior	Barton G. Weick, Sue Ritter et al.	6
14	p-Chloroamphetamine-induced hyperthermia pharmacologically distinct from fenfluramine-induced hyperthermia	Journal of Pharmacy and Pharmacology	Raymond M. Quock, Barton G. Weick	15
15	Tryptamine-induced drug effects insensitive to serotoninergic antagonists: evidence of specific tryptaminergic receptor stimulation?	Journal of Pharmacy and Pharmacology	Raymond M. Quock, Barton G. Weick	41
16	Effects of thyrotropin-releasing hormone (TRH) microinjected into various brain areas of conscious and pentobarbital-pretreated rabbits	Life Sciences	Justine R. Smith, Barton G. Weick et al.	48

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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