Barbara Hobo

716 total citations
18 papers, 524 citations indexed

About

Barbara Hobo is a scholar working on Molecular Biology, Genetics and Physiology. According to data from OpenAlex, Barbara Hobo has authored 18 papers receiving a total of 524 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 7 papers in Genetics and 6 papers in Physiology. Recurrent topics in Barbara Hobo's work include Ubiquitin and proteasome pathways (8 papers), Alzheimer's disease research and treatments (6 papers) and Virus-based gene therapy research (4 papers). Barbara Hobo is often cited by papers focused on Ubiquitin and proteasome pathways (8 papers), Alzheimer's disease research and treatments (6 papers) and Virus-based gene therapy research (4 papers). Barbara Hobo collaborates with scholars based in Netherlands, United Kingdom and United States. Barbara Hobo's co-authors include David F. Fischer, Elly M. Hol, Fred W. van Leeuwen, Joost Verhaagen, Paula van Tijn, Rob A. I. de Vos, Wouter Kamphorst, M.A.F. Sonnemans, Alejandro Carnicer‐Lombarte and James W. Fawcett and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Neuroscience and PLoS ONE.

In The Last Decade

Barbara Hobo

17 papers receiving 519 citations

Peers

Barbara Hobo

PHYSI

CMN

GENET

Juliette M. Han United States

Haim Belinson Israel

Ann K. Wright United Kingdom

Niamh C. O’Sullivan Ireland

Hyun-Hee Ryu South Korea

Małgorzata Perycz Poland

Rosalind Norkett United Kingdom

Nathalie Higgs United Kingdom

So Yeon Koo United States

Francesca Pischedda Italy

Juliette M. Han United States

Barbara Hobo

447 ×1.4

90 ×0.7

215 ×1.6

PHYSI

180 ×1.5

CMN

95 ×0.9

GENET

Citations per year, relative to Barbara Hobo Barbara Hobo (= 1×) peers Juliette M. Han

Countries citing papers authored by Barbara Hobo

Since Specialization

Citations

This map shows the geographic impact of Barbara Hobo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Barbara Hobo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Barbara Hobo more than expected).

Fields of papers citing papers by Barbara Hobo

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Barbara Hobo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Barbara Hobo. The network helps show where Barbara Hobo may publish in the future.

Co-authorship network of co-authors of Barbara Hobo

This figure shows the co-authorship network connecting the top 25 collaborators of Barbara Hobo. A scholar is included among the top collaborators of Barbara Hobo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Barbara Hobo. Barbara Hobo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

Hobo, Barbara, et al.. (2024). Production of High-Yield Adeno Associated Vector Batches Using HEK293 Suspension Cells. Journal of Visualized Experiments. 1 indexed citations

Winter, Fred de, Isaac Francos-Quijorna, Emily R. Burnside, et al.. (2022). Characterization of an immune-evading doxycycline-inducible lentiviral vector for gene therapy in the spinal cord. Experimental Neurology. 355. 114120–114120. 3 indexed citations

Nieuwenhuis, Bart, Barbara Haenzi, Sam Hilton, et al.. (2020). Optimization of adeno-associated viral vector-mediated transduction of the corticospinal tract: comparison of four promoters. Gene Therapy. 28(1-2). 56–74. 79 indexed citations

Korecka, Joanna A., Elizabeth B. Moloney, Ruben Eggers, et al.. (2017). Repulsive Guidance Molecule a (RGMa) Induces Neuropathological and Behavioral Changes That Closely Resemble Parkinson's Disease. Journal of Neuroscience. 37(39). 9361–9379. 27 indexed citations

Verhaagen, Joost, Barbara Hobo, Erich Ehlert, et al.. (2017). Small Scale Production of Recombinant Adeno-Associated Viral Vectors for Gene Delivery to the Nervous System. Methods in molecular biology. 1715. 3–17. 11 indexed citations

Moloney, Elizabeth B., Barbara Hobo, Fred de Winter, & Joost Verhaagen. (2017). Expression of a Mutant SEMA3A Protein with Diminished Signalling Capacity Does Not Alter ALS-Related Motor Decline, or Confer Changes in NMJ Plasticity after BotoxA-Induced Paralysis of Male Gastrocnemic Muscle. PLoS ONE. 12(1). e0170314–e0170314. 11 indexed citations

Giacobini, Paolo, Jyoti Parkash, Céline Campagne, et al.. (2014). Brain Endothelial Cells Control Fertility through Ovarian-Steroid–Dependent Release of Semaphorin 3A. PLoS Biology. 12(3). e1001808–e1001808. 50 indexed citations

Tijn, Paula van, Frank J.A. Dennissen, Barbara Hobo, et al.. (2012). Mutant ubiquitin decreases amyloid β plaque formation in a transgenic mouse model of Alzheimer’s disease. Neurochemistry International. 61(5). 739–748. 34 indexed citations

Tijn, Paula van, et al.. (2010). Low levels of mutant ubiquitin are degraded by the proteasome in vivo. Journal of Neuroscience Research. 88(11). 2325–2337. 14 indexed citations

10.

Tijn, Paula van, et al.. (2010). Alzheimer-associated mutant ubiquitin impairs spatial reference memory. Physiology & Behavior. 102(2). 193–200. 13 indexed citations

11.

Pril, Remko de, Barbara Hobo, Paula van Tijn, et al.. (2009). Modest proteasomal inhibition by aberrant ubiquitin exacerbates aggregate formation in a Huntington disease mouse model. Molecular and Cellular Neuroscience. 43(3). 281–286. 15 indexed citations

12.

Fischer, David F., Paula van Tijn, Barbara Hobo, et al.. (2008). Long-term proteasome dysfunction in the mouse brain by expression of aberrant ubiquitin. Neurobiology of Aging. 30(6). 847–863. 49 indexed citations

13.

Fischer, David F., Barbara Hobo, Rob A. I. de Vos, et al.. (2008). Proteasome subunit proteins and neuropathology in tauopathies and synucleinopathies: Consequences for proteomic analyses. PROTEOMICS. 8(6). 1221–1236. 22 indexed citations

14.

Leeuwen, F.W. van, Paula van Tijn, M.A.F. Sonnemans, et al.. (2006). Frameshift proteins in autosomal dominant forms of Alzheimer disease and other tauopathies. Neurology. 66(1_suppl_1). S86–92. 38 indexed citations

15.

Fischer, David F., Elly M. Hol, Barbara Hobo, & Fred W. van Leeuwen. (2005). Alzheimer-associated APP+1 transgenic mice: Frameshift β-amyloid precursor protein is secreted in cerebrospinal fluid without inducing neuropathology. Neurobiology of Aging. 27(10). 1445–1450. 7 indexed citations

16.

Fischer, David F., Jacqueline A. Sluijs, M.A.F. Sonnemans, et al.. (2004). Frame‐shifted amyloid precursor protein found in Alzheimer's disease and Down's syndrome increases levels of secreted amyloid β40. Journal of Neurochemistry. 90(3). 712–723. 10 indexed citations

17.

Hol, Elly M., Lisya Gerez, Jacqueline A. Sluijs, et al.. (2003). Frameshifted β-Amyloid Precursor Protein (APP+1) Is a Secretory Protein, and the Level of APP+1 in Cerebrospinal Fluid Is Linked to Alzheimer Pathology. Journal of Biological Chemistry. 278(41). 39637–39643. 12 indexed citations

18.

Fischer, David F., Rob A. I. de Vos, Femke M.S. de Vrij, et al.. (2003). Disease‐specific accumulation of mutant ubiquitin as a marker for proteasomal dysfunction in the brain. The FASEB Journal. 17(14). 2014–2024. 128 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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