Barbara Belli
- Molecular Biology
- Hematology top 2%
- Infectious Diseases top 5%
- Immunology top 10%
- Genetics top 10%
- Co-authors
- Thomas W. DubenskyJohn M. PoloDavid A. DriverRobert C. ArmstrongRuwanthi N. GunawardaneSilvia PerriDaniel BrighamShripad S. Bhagwat
- Topics
- Chronic Lymphocytic Leukemia Research (8 papers)Acute Myeloid Leukemia Research (7 papers)Virus-based gene therapy research (5 papers)
- Partner nations
- United StatesUnited KingdomFrance
In The Last Decade
Barbara Belli
26 papers receiving 1.2k citations
Hit Papers
Peers
Comparison fields: 5 of 67
- Molecular Biology 525
- Hematology 418
- Infectious Diseases 366
- Immunology 303
- Genetics 239
Countries citing papers authored by Barbara Belli
This map shows the geographic impact of Barbara Belli's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Barbara Belli with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Barbara Belli more than expected).
Fields of papers citing papers by Barbara Belli
This network shows the impact of papers produced by Barbara Belli. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Barbara Belli. The network helps show where Barbara Belli may publish in the future.
Co-authorship network of co-authors of Barbara Belli
This figure shows the co-authorship network connecting the top 25 collaborators of Barbara Belli. A scholar is included among the top collaborators of Barbara Belli based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Barbara Belli. Barbara Belli is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 9 | |
| 3 | 16 | |
| 4 | 39 | |
| 5 | 13 | |
| 6 | 1 | |
| 7 | 1 | |
| 8 | Abstract #2335: AC220, a uniquely potent and selective FLT3 inhibitor, provides sustained protection in a FLT3 ITD driven model of acute myeloid leukemia | 0 |
| 9 | Alphavirus DNA and particle replicons for vaccines and gene therapy. | 37 |
| 10 | 100 | |
| 11 | 98 | |
| 12 | 109 | |
| 13 | 145 | |
| 14 | 8 | |
| 15 | 1 | |
| 16 | 14 | |
| 17 | 14 | |
| 18 | 19 | |
| 19 | 10 | |
| 20 | 24 |
About Barbara Belli
Barbara Belli is a scholar working on Genetics, Hematology and Virology, having authored 28 papers that have together received 1.3k indexed citations. Recurring topics across this work include Chronic Lymphocytic Leukemia Research (8 papers), Acute Myeloid Leukemia Research (7 papers) and Virus-based gene therapy research (5 papers). The work is most often cited by research in Hematology (418 citations), Infectious Diseases (366 citations) and Genetics (200 citations). Barbara Belli has collaborated with scholars based in United States, United Kingdom and France. Frequent co-authors include Thomas W. Dubensky, John M. Polo, David A. Driver, Robert C. Armstrong, Ruwanthi N. Gunawardane, Silvia Perri, Daniel Brigham, Shripad S. Bhagwat, Joyce James and Michael F. Gardner. Their work appears in journals such as Proceedings of the National Academy of Sciences, Blood and Cancer Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.