Áurea Castilho

470 total citations
18 papers, 388 citations indexed

About

Áurea Castilho is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Ophthalmology. According to data from OpenAlex, Áurea Castilho has authored 18 papers receiving a total of 388 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 9 papers in Cellular and Molecular Neuroscience and 6 papers in Ophthalmology. Recurrent topics in Áurea Castilho's work include Neuroscience and Neuropharmacology Research (9 papers), Retinal Development and Disorders (8 papers) and Retinal Diseases and Treatments (6 papers). Áurea Castilho is often cited by papers focused on Neuroscience and Neuropharmacology Research (9 papers), Retinal Development and Disorders (8 papers) and Retinal Diseases and Treatments (6 papers). Áurea Castilho collaborates with scholars based in Portugal, Norway and Denmark. Áurea Castilho's co-authors include António Francisco Ambrósio, Filipa I. Baptista, Célia A. Aveleira, Joana M. Gaspar, Ermelindo C. Leal, Espen Hartveit, Margaret Lin Veruki, Joana Liberal, Joana Galvão and Rodrigo A. Cunha and has published in prestigious journals such as Journal of Neuroscience, PLoS ONE and Journal of Neurophysiology.

In The Last Decade

Áurea Castilho

17 papers receiving 384 citations

Peers

Áurea Castilho
Tora Sund Morken Norway
Camelia A. Danilov United States
M. Doly France
Thanh Thanh L. Nguyen United States
S. Berweck Germany
Esraa Shosha United States
Sandra Barral United States
D.T. Organisciak United States
Harry Twaalfhoven Netherlands
Tora Sund Morken Norway
Áurea Castilho
Citations per year, relative to Áurea Castilho Áurea Castilho (= 1×) peers Tora Sund Morken

Countries citing papers authored by Áurea Castilho

Since Specialization
Citations

This map shows the geographic impact of Áurea Castilho's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Áurea Castilho with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Áurea Castilho more than expected).

Fields of papers citing papers by Áurea Castilho

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Áurea Castilho. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Áurea Castilho. The network helps show where Áurea Castilho may publish in the future.

Co-authorship network of co-authors of Áurea Castilho

This figure shows the co-authorship network connecting the top 25 collaborators of Áurea Castilho. A scholar is included among the top collaborators of Áurea Castilho based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Áurea Castilho. Áurea Castilho is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Castilho, Áurea, et al.. (2025). Multimodal Imaging of Brain Metastasis-Derived Extracellular Vesicles Using Superparamagnetic Iron Oxide Nanoparticle Labeling. International Journal of Nanomedicine. Volume 20. 7501–7514.
2.
Castilho, Áurea, et al.. (2022). Inhibitory inputs to an inhibitory interneuron: Spontaneous postsynaptic currents and GABA A receptors of A17 amacrine cells in the rat retina. European Journal of Neuroscience. 55(6). 1442–1470. 3 indexed citations
3.
Veruki, Margaret Lin, Yifan Zhou, Áurea Castilho, Catherine W. Morgans, & Espen Hartveit. (2018). Extrasynaptic NMDA Receptors on Rod Pathway Amacrine Cells: Molecular Composition, Activation, and Signaling. Journal of Neuroscience. 39(4). 627–650. 13 indexed citations
4.
Baptista, Filipa I., Célia A. Aveleira, Áurea Castilho, & António Francisco Ambrósio. (2017). Elevated Glucose and Interleukin-1β Differentially Affect Retinal Microglial Cell Proliferation. Mediators of Inflammation. 2017. 1–11. 29 indexed citations
5.
Hartveit, Espen, et al.. (2017). AMPA receptors at ribbon synapses in the mammalian retina: kinetic models and molecular identity. Brain Structure and Function. 223(2). 769–804. 5 indexed citations
6.
Losnegård, Are, Lars A. R. Reisæter, Ole J. Halvorsen, et al.. (2017). Intensity-based volumetric registration of magnetic resonance images and whole-mount sections of the prostate. Computerized Medical Imaging and Graphics. 63. 24–30. 15 indexed citations
7.
Castilho, Áurea, António Francisco Ambrósio, Espen Hartveit, & Margaret Lin Veruki. (2015). Disruption of a Neural Microcircuit in the Rod Pathway of the Mammalian Retina by Diabetes Mellitus. Journal of Neuroscience. 35(13). 5422–5433. 38 indexed citations
8.
Baptista, Filipa I., Áurea Castilho, Joana M. Gaspar, et al.. (2015). Long-term exposure to high glucose increases the content of several exocytotic proteins and of vesicular GABA transporter in cultured retinal neural cells. Neuroscience Letters. 602. 56–61. 14 indexed citations
9.
Castilho, Áurea, et al.. (2015). Diabetic hyperglycemia reduces Ca2+ permeability of extrasynaptic AMPA receptors in AII amacrine cells. Journal of Neurophysiology. 114(3). 1545–1553. 17 indexed citations
10.
Castilho, Áurea, Joana Liberal, Filipa I. Baptista, et al.. (2014). Diabetes causes transient changes in the composition and phosphorylation of α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptors and interaction with auxiliary proteins in the rat retina.. PubMed. 20. 894–907. 5 indexed citations
11.
Castilho, Áurea, et al.. (2012). Heme Oxygenase-1 Protects Retinal Endothelial Cells against High Glucose- and Oxidative/Nitrosative Stress-Induced Toxicity. PLoS ONE. 7(8). e42428–e42428. 81 indexed citations
12.
Castilho, Áurea, Joana Liberal, Filipa I. Baptista, et al.. (2012). Elevated glucose concentration changes the content and cellular localization of AMPA receptors in the retina but not in the hippocampus. Neuroscience. 219. 23–32. 16 indexed citations
13.
Dinis, Pedro & Áurea Castilho. (2012). Integrating Sieving and Laser Data To Obtain Bulk Grain-Size Distributions. Journal of Sedimentary Research. 82(9). 747–754. 12 indexed citations
14.
Gaspar, Joana M., Filipa I. Baptista, Joana Galvão, et al.. (2010). Diabetes differentially affects the content of exocytotic proteins in hippocampal and retinal nerve terminals. Neuroscience. 169(4). 1589–1600. 45 indexed citations
15.
16.
Gaspar, Joana M., Áurea Castilho, Filipa I. Baptista, Joana Liberal, & António Francisco Ambrósio. (2010). Long-term exposure to high glucose induces changes in the content and distribution of some exocytotic proteins in cultured hippocampal neurons. Neuroscience. 171(4). 981–992. 35 indexed citations
17.
Leal, Ermelindo C., Célia A. Aveleira, Áurea Castilho, et al.. (2009). High glucose and oxidative/nitrosative stress conditions induce apoptosis in retinal endothelial cells by a caspase-independent pathway. Experimental Eye Research. 88(5). 983–991. 47 indexed citations
18.
Leal, Ermelindo C., Célia A. Aveleira, Áurea Castilho, Filipa I. Baptista, & António Francisco Ambrósio. (2008). Müller Cells Do Not Influence Leukocyte Adhesion to Retinal Endothelial Cells. Ocular Immunology and Inflammation. 16(4). 173–179. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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