A.S. Volmer

522 total citations
8 papers, 274 citations indexed

About

A.S. Volmer is a scholar working on Pulmonary and Respiratory Medicine, Molecular Biology and Epidemiology. According to data from OpenAlex, A.S. Volmer has authored 8 papers receiving a total of 274 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Pulmonary and Respiratory Medicine, 3 papers in Molecular Biology and 1 paper in Epidemiology. Recurrent topics in A.S. Volmer's work include Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (3 papers), Neonatal Respiratory Health Research (3 papers) and Cystic Fibrosis Research Advances (3 papers). A.S. Volmer is often cited by papers focused on Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (3 papers), Neonatal Respiratory Health Research (3 papers) and Cystic Fibrosis Research Advances (3 papers). A.S. Volmer collaborates with scholars based in United States, China and Germany. A.S. Volmer's co-authors include Wanda K. O’Neal, Richard C. Boucher, Barbara R. Grubb, Kim Burns, Christopher M. Evans, Alessandra Livraghi-Butrico, Rodney C. Gilmore, Scott H. Randell, Kenichi Okuda and Takafumi Kato and has published in prestigious journals such as Journal of Clinical Investigation, American Journal of Respiratory and Critical Care Medicine and Biochemical and Biophysical Research Communications.

In The Last Decade

A.S. Volmer

8 papers receiving 273 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
A.S. Volmer United States 6 190 84 52 34 27 8 274
Chandra L. Shrestha United States 13 307 1.6× 92 1.1× 28 0.5× 40 1.2× 69 2.6× 20 405
Xizi Du China 11 93 0.5× 118 1.4× 98 1.9× 65 1.9× 46 1.7× 27 290
Michael V. Rector United States 6 353 1.9× 93 1.1× 61 1.2× 18 0.5× 25 0.9× 7 436
Hiro Matsui United States 4 198 1.0× 292 3.5× 45 0.9× 15 0.4× 38 1.4× 5 615
Qi Cheng China 11 74 0.4× 137 1.6× 22 0.4× 39 1.1× 93 3.4× 20 403
Stephen J. Coles United States 9 172 0.9× 88 1.0× 158 3.0× 26 0.8× 13 0.5× 13 332
Mary Abigail S. Garcia United States 4 182 1.0× 118 1.4× 38 0.7× 17 0.5× 6 0.2× 6 308
Maximiliano Ruben Ferrero Argentina 11 55 0.3× 70 0.8× 33 0.6× 108 3.2× 68 2.5× 24 261
Andreas Claaß Germany 8 304 1.6× 71 0.8× 28 0.5× 13 0.4× 54 2.0× 13 372
Luigi Ratclif Italy 4 89 0.5× 88 1.0× 12 0.2× 39 1.1× 14 0.5× 5 168

Countries citing papers authored by A.S. Volmer

Since Specialization
Citations

This map shows the geographic impact of A.S. Volmer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A.S. Volmer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A.S. Volmer more than expected).

Fields of papers citing papers by A.S. Volmer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A.S. Volmer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A.S. Volmer. The network helps show where A.S. Volmer may publish in the future.

Co-authorship network of co-authors of A.S. Volmer

This figure shows the co-authorship network connecting the top 25 collaborators of A.S. Volmer. A scholar is included among the top collaborators of A.S. Volmer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with A.S. Volmer. A.S. Volmer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Boucher, R. C., Liou Y. Sun, A.S. Volmer, et al.. (2021). XBP1s regulates MUC5B in a promoter variant-dependent pathway in idiopathic pulmonary fibrosis airway epithelia. UNC Libraries. 1 indexed citations
2.
Boucher, R. C., A.S. Volmer, Kimberlie A. Burns, et al.. (2020). Lung disease phenotypes caused by overexpression of combinations of α-, β-, and γ-subunits of the epithelial sodium channel in mouse airways. UNC Libraries. 1 indexed citations
3.
Chen, Gang, Carla M. P. Ribeiro, Ling Sun, et al.. (2019). XBP1S Regulates MUC5B in a Promoter Variant–Dependent Pathway in Idiopathic Pulmonary Fibrosis Airway Epithelia. American Journal of Respiratory and Critical Care Medicine. 200(2). 220–234. 45 indexed citations
4.
Chen, Gang, Ling Sun, Takafumi Kato, et al.. (2019). IL-1β dominates the promucin secretory cytokine profile in cystic fibrosis. Journal of Clinical Investigation. 129(10). 4433–4450. 85 indexed citations
5.
Chen, Gang, A.S. Volmer, Yangmei Deng, et al.. (2018). Role of Spdef in the Regulation of Muc5b Expression in the Airways of Naive and Mucoobstructed Mice. American Journal of Respiratory Cell and Molecular Biology. 59(3). 383–396. 21 indexed citations
6.
Livraghi-Butrico, Alessandra, A.S. Volmer, Rodney C. Gilmore, et al.. (2017). Lung disease phenotypes caused by over-expression of combinations of alpha, beta, and gamma subunits of the epithelial sodium channel in mouse airways. American Journal of Physiology-Lung Cellular and Molecular Physiology. 314(2). ajplung.00382.2–ajplung.00382.2. 12 indexed citations
7.
Grubb, Barbara R., A.S. Volmer, Kim Burns, et al.. (2016). Contribution of mucus concentration and secreted mucins Muc5ac and Muc5b to the pathogenesis of muco-obstructive lung disease. Mucosal Immunology. 10(2). 395–407. 91 indexed citations
8.
Kim, Sally A., Andy Hudmon, A.S. Volmer, & M. Neal Waxham. (2001). CaM-Kinase II Dephosphorylates Thr286 by a Reversal of the Autophosphorylation Reaction. Biochemical and Biophysical Research Communications. 282(3). 773–780. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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