Arthur L. Haas
- Molecular Biology top 1%
- Oncology top 1%
- Immunology top 1%
- Cell Biology top 1%
- Epidemiology top 2%
- Co-authors
- Avram HershkoI A RoseKeith R. LoebAaron CiechanoverThomas J. SiepmannDaniel FinleyKeith D. WilkinsonSeth Sadis
- Topics
- Ubiquitin and proteasome pathways (59 papers)Glycosylation and Glycoproteins Research (15 papers)Peptidase Inhibition and Analysis (15 papers)
- Cited by
- Molecular BiologyImmunologyOncology
- Journals
- Proceedings of the National Academy of SciencesJournal of Biological ChemistryJournal of Clinical Investigation
- Partner nations
- United StatesUnited KingdomIsrael
In The Last Decade
Arthur L. Haas
98 papers receiving 7.3k citations
Hit Papers
Peers
Comparison fields: 5 of 121
- Molecular Biology 5.8k
- Oncology 2.0k
- Immunology 1.7k
- Cell Biology 1.2k
- Epidemiology 1.1k
Countries citing papers authored by Arthur L. Haas
This map shows the geographic impact of Arthur L. Haas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Arthur L. Haas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Arthur L. Haas more than expected).
Fields of papers citing papers by Arthur L. Haas
This network shows the impact of papers produced by Arthur L. Haas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Arthur L. Haas. The network helps show where Arthur L. Haas may publish in the future.
Co-authorship network of co-authors of Arthur L. Haas
This figure shows the co-authorship network connecting the top 25 collaborators of Arthur L. Haas. A scholar is included among the top collaborators of Arthur L. Haas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Arthur L. Haas. Arthur L. Haas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 12 | |
| 2 | 18 | |
| 3 | 36 | |
| 4 | 16 | |
| 5 | 3 | |
| 6 | 48 | |
| 7 | 11 | |
| 8 | 23 | |
| 9 | 14 | |
| 10 | 14 | |
| 11 | 7 | |
| 12 | 49 | |
| 13 | 15 | |
| 14 | 91 | |
| 15 | 95 | |
| 16 | 24 | |
| 17 | 442 | |
| 18 | 26 | |
| 19 | 17 | |
| 20 | 17 |
About Arthur L. Haas
Arthur L. Haas is a scholar working on Oncology, Molecular Biology and Immunology, having authored 98 papers that have together received 7.5k indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (59 papers), Glycosylation and Glycoproteins Research (15 papers) and Peptidase Inhibition and Analysis (15 papers). The work is most often cited by research in Molecular Biology (5.8k citations), Immunology (1.7k citations) and Oncology (2.0k citations). Arthur L. Haas has collaborated with scholars based in United States, United Kingdom and Israel. Frequent co-authors include Avram Hershko, I A Rose, Keith R. Loeb, Aaron Ciechanover, Thomas J. Siepmann, Daniel Finley, Keith D. Wilkinson, Seth Sadis, Jean Spence and Michael K. Urban. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.