Ariel Stanhill

824 total citations
18 papers, 678 citations indexed

About

Ariel Stanhill is a scholar working on Molecular Biology, Cell Biology and Epidemiology. According to data from OpenAlex, Ariel Stanhill has authored 18 papers receiving a total of 678 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 11 papers in Cell Biology and 5 papers in Epidemiology. Recurrent topics in Ariel Stanhill's work include Ubiquitin and proteasome pathways (11 papers), Endoplasmic Reticulum Stress and Disease (11 papers) and Autophagy in Disease and Therapy (5 papers). Ariel Stanhill is often cited by papers focused on Ubiquitin and proteasome pathways (11 papers), Endoplasmic Reticulum Stress and Disease (11 papers) and Autophagy in Disease and Therapy (5 papers). Ariel Stanhill collaborates with scholars based in Israel, United States and Germany. Ariel Stanhill's co-authors include Aaron Ciechanover, David Engelberg, Cole M. Haynes, David Ron, Ilana Braunstein, Yuhong Zhang, Thomas A. Neubert, Chong‐Feng Xu, Enid E. Martinez and Juliya Kalinina and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Molecular Cell.

In The Last Decade

Ariel Stanhill

17 papers receiving 673 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ariel Stanhill Israel 14 585 241 151 77 66 18 678
Joy L. Nishikawa Canada 6 1.1k 1.9× 203 0.8× 99 0.7× 86 1.1× 48 0.7× 8 1.3k
Patricia G. Tu United States 8 399 0.7× 348 1.4× 157 1.0× 23 0.3× 41 0.6× 8 669
Seweryn Mroczek Poland 15 871 1.5× 127 0.5× 108 0.7× 30 0.4× 28 0.4× 27 985
Kojiro Takeda Japan 12 452 0.8× 151 0.6× 67 0.4× 24 0.3× 38 0.6× 19 526
Przemyslaw A. Filipek Austria 7 392 0.7× 208 0.9× 154 1.0× 39 0.5× 15 0.2× 10 696
Kentaro Kajiwara Japan 14 555 0.9× 308 1.3× 79 0.5× 40 0.5× 12 0.2× 26 731
M. Daniel Ricketts United States 13 640 1.1× 102 0.4× 236 1.6× 33 0.4× 33 0.5× 14 836
Scott K. Wooden United States 13 613 1.0× 427 1.8× 70 0.5× 64 0.8× 28 0.4× 15 844
Seung Jae Jeong South Korea 8 670 1.1× 178 0.7× 103 0.7× 37 0.5× 16 0.2× 11 883
Régis Courbeyrette France 13 764 1.3× 241 1.0× 79 0.5× 64 0.8× 10 0.2× 14 870

Countries citing papers authored by Ariel Stanhill

Since Specialization
Citations

This map shows the geographic impact of Ariel Stanhill's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ariel Stanhill with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ariel Stanhill more than expected).

Fields of papers citing papers by Ariel Stanhill

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ariel Stanhill. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ariel Stanhill. The network helps show where Ariel Stanhill may publish in the future.

Co-authorship network of co-authors of Ariel Stanhill

This figure shows the co-authorship network connecting the top 25 collaborators of Ariel Stanhill. A scholar is included among the top collaborators of Ariel Stanhill based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ariel Stanhill. Ariel Stanhill is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Stanhill, Ariel, et al.. (2024). A non-symmetrical p97 conformation initiates a multistep recruitment of Ufd1/Npl4. iScience. 27(6). 110061–110061.
2.
Kaushansky, Nathali, et al.. (2023). The role of RNF149 in the pre-emptive quality control substrate ubiquitination. Communications Biology. 6(1). 385–385. 4 indexed citations
3.
Braunstein, Ilana, et al.. (2021). An Arsenite Relay between PSMD14 and AIRAP Enables Revival of Proteasomal DUB Activity. Biomolecules. 11(9). 1317–1317. 2 indexed citations
4.
Rahighi, Simin, Ilana Braunstein, Nicola Ternette, et al.. (2016). Selective Binding of AIRAPL Tandem UIMs to Lys48-Linked Tri-Ubiquitin Chains. Structure. 24(3). 412–422. 18 indexed citations
5.
Braunstein, Ilana, et al.. (2015). Proteasomal degradation of preemptive quality control (pQC) substrates is mediated by an AIRAPL–p97 complex. Molecular Biology of the Cell. 26(21). 3719–3727. 25 indexed citations
6.
Braunstein, Ilana, et al.. (2014). VWA domain of S5a restricts the ability to bind ubiquitin and Ubl to the 26S proteasome. Molecular Biology of the Cell. 25(25). 3988–3998. 12 indexed citations
7.
Braunstein, Ilana, et al.. (2014). Inherent Asymmetry in the 26S Proteasome Is Defined by the Ubiquitin Receptor RPN13. Journal of Biological Chemistry. 289(9). 5609–5618. 19 indexed citations
8.
Braunstein, Ilana, et al.. (2014). Stress-induced Start Codon Fidelity Regulates Arsenite-inducible Regulatory Particle-associated Protein (AIRAP) Translation. Journal of Biological Chemistry. 289(30). 20706–20716. 14 indexed citations
9.
Ciechanover, Aaron & Ariel Stanhill. (2013). The complexity of recognition of ubiquitinated substrates by the 26S proteasome. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1843(1). 86–96. 120 indexed citations
10.
Alter, Joel, et al.. (2013). Signal-peptide-mediated translocation is regulated by a p97–AIRAPL complex. Biochemical Journal. 457(2). 253–261. 21 indexed citations
11.
Stanhill, Ariel, et al.. (2011). Stalled Proteasomes Are Directly Relieved by P97 Recruitment. Journal of Biological Chemistry. 286(35). 30274–30283. 33 indexed citations
12.
Wiseman, R. Luke, King‐Tung Chin, Cole M. Haynes, et al.. (2009). Thioredoxin-related Protein 32 Is an Arsenite-regulated Thiol Reductase of the Proteasome 19 S Particle. Journal of Biological Chemistry. 284(22). 15233–15245. 39 indexed citations
13.
Yun, Chi Young, Ariel Stanhill, Yun Yang, et al.. (2008). Proteasomal adaptation to environmental stress links resistance to proteotoxicity with longevity in Caenorhabditis elegans. Proceedings of the National Academy of Sciences. 105(19). 7094–7099. 85 indexed citations
14.
Stanhill, Ariel, Cole M. Haynes, Yuhong Zhang, et al.. (2006). An Arsenite-Inducible 19S Regulatory Particle-Associated Protein Adapts Proteasomes to Proteotoxicity. Molecular Cell. 23(6). 875–885. 98 indexed citations
15.
Stanhill, Ariel, Vladimir Levin, Ayal Hendel, et al.. (2005). Ha-rasval12 induces HSP70b transcription via the HSE/HSF1 system, but HSP70b expression is suppressed in Ha-rasval12-transformed cells. Oncogene. 25(10). 1485–1495. 21 indexed citations
16.
Stanhill, Ariel, et al.. (2002). HSF and Msn2/4p can exclusively or cooperatively activate the yeast HSP104 gene. Molecular Microbiology. 44(1). 21–35. 51 indexed citations
17.
Stanhill, Ariel, et al.. (1999). The Yeast Ras/Cyclic AMP Pathway Induces Invasive Growth by Suppressing the Cellular Stress Response. Molecular and Cellular Biology. 19(11). 7529–7538. 95 indexed citations
18.
Stanhill, Ariel, et al.. (1997). Translocation of NLS–BSA conjugates into nuclei of permeabilized mammalian cells can be supported by protoplast extract. FEBS Letters. 412(3). 535–539. 21 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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