Aria Baniahmad

7.3k total citations
151 papers, 5.6k citations indexed

About

Aria Baniahmad is a scholar working on Molecular Biology, Genetics and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Aria Baniahmad has authored 151 papers receiving a total of 5.6k indexed citations (citations by other indexed papers that have themselves been cited), including 109 papers in Molecular Biology, 54 papers in Genetics and 51 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Aria Baniahmad's work include Prostate Cancer Treatment and Research (49 papers), Estrogen and related hormone effects (43 papers) and Genomics and Chromatin Dynamics (24 papers). Aria Baniahmad is often cited by papers focused on Prostate Cancer Treatment and Research (49 papers), Estrogen and related hormone effects (43 papers) and Genomics and Chromatin Dynamics (24 papers). Aria Baniahmad collaborates with scholars based in Germany, Iran and Iraq. Aria Baniahmad's co-authors include Rainer Renkawitz, Christof Steiner, Ming‐Jer Tsai, Leslie Burke, Marc Müller, Stephan P. Tenbaum, Sophia Y. Tsai, Thanakorn Pungsrinont, Mohammad Taheri and Soudeh Ghafouri‐Fard and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Aria Baniahmad

149 papers receiving 5.5k citations

Peers

Aria Baniahmad
David M. Heery United Kingdom
Sergio A. Oñate United States
David M. Lonard United States
Steven K. Nordeen United States
Ferdinando Auricchio Italy
John M. Kokontis United States
Monica M. Montano United States
Diane M. Robins United States
Valentina Perissi United States
A. E. Wakeling United Kingdom
David M. Heery United Kingdom
Aria Baniahmad
Citations per year, relative to Aria Baniahmad Aria Baniahmad (= 1×) peers David M. Heery

Countries citing papers authored by Aria Baniahmad

Since Specialization
Citations

This map shows the geographic impact of Aria Baniahmad's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Aria Baniahmad with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Aria Baniahmad more than expected).

Fields of papers citing papers by Aria Baniahmad

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Aria Baniahmad. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Aria Baniahmad. The network helps show where Aria Baniahmad may publish in the future.

Co-authorship network of co-authors of Aria Baniahmad

This figure shows the co-authorship network connecting the top 25 collaborators of Aria Baniahmad. A scholar is included among the top collaborators of Aria Baniahmad based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Aria Baniahmad. Aria Baniahmad is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kallenbach, Julia, et al.. (2024). The oncogenic lncRNA MIR503HG suppresses cellular senescence counteracting supraphysiological androgen treatment in prostate cancer. Journal of Experimental & Clinical Cancer Research. 43(1). 321–321. 3 indexed citations
2.
Kallenbach, Julia, et al.. (2024). The Novel Direct AR Target Gene Annexin A2 Mediates Androgen-Induced Cellular Senescence in Prostate Cancer Cells. Biochemical Genetics. 63(6). 5013–5028. 2 indexed citations
3.
Pungsrinont, Thanakorn, et al.. (2023). Androgen receptor agonist and antagonist reduce response of cytokine‐induced killer cells on prostate cancer cells. Journal of Cellular and Molecular Medicine. 27(19). 2970–2982. 2 indexed citations
4.
Ghafouri‐Fard, Soudeh, Bashdar Mahmud Hussen, Snur Rasool Abdullah, et al.. (2023). A review on the role of long non-coding RNA prostate androgen-regulated transcript 1 (PART1) in the etiology of different disorders. Frontiers in Cell and Developmental Biology. 11. 1124615–1124615. 4 indexed citations
5.
Ghafouri‐Fard, Soudeh, Tayyebeh Khoshbakht, Bashdar Mahmud Hussen, et al.. (2022). A Concise Review on Dysregulation of LINC00665 in Cancers. Cells. 11(22). 3575–3575. 6 indexed citations
6.
7.
Taheri, Mohammad, et al.. (2022). LncRNA/miRNA/mRNA Network Introduces Novel Biomarkers in Prostate Cancer. Cells. 11(23). 3776–3776. 14 indexed citations
8.
Ghafouri‐Fard, Soudeh, Mohammad Taheri, & Aria Baniahmad. (2022). Inhibitor of Growth Factors Regulate Cellular Senescence. Cancers. 14(13). 3107–3107. 3 indexed citations
9.
Bartsch, Sophie, Mohsen Esmaeili, Thanakorn Pungsrinont, et al.. (2021). Antithetic hTERT Regulation by Androgens in Prostate Cancer Cells: hTERT Inhibition Is Mediated by the ING1 and ING2 Tumor Suppressors. Cancers. 13(16). 4025–4025. 15 indexed citations
10.
Taheri, Mohammad, Tayyebeh Khoshbakht, Elena Jamali, et al.. (2021). Interaction between Non-Coding RNAs and Androgen Receptor with an Especial Focus on Prostate Cancer. Cells. 10(11). 3198–3198. 10 indexed citations
11.
Kallenbach, Julia, Seyed Mohammad Mahdi Rasa, Martin Ungelenk, et al.. (2021). The androgen receptor—lncRNASAT1-AKT-p15 axis mediates androgen-induced cellular senescence in prostate cancer cells. Oncogene. 41(7). 943–959. 30 indexed citations
12.
Kraft, Florian, et al.. (2020). Thyroid hormone induces cellular senescence in prostate cancer cells through induction of DEC1. The Journal of Steroid Biochemistry and Molecular Biology. 201. 105689–105689. 13 indexed citations
13.
Baniahmad, Aria. (2013). Why do we need to age?. Hormone Molecular Biology and Clinical Investigation. 16(1). 3–5. 1 indexed citations
14.
Lorenz, Viola, et al.. (2011). Sodium butyrate induces cellular senescence in neuroblastoma and prostate cancer cells. Hormone Molecular Biology and Clinical Investigation. 7(1). 265–272. 15 indexed citations
15.
Kob, Robert, et al.. (2009). Regulation of the anaphase-promoting complex by the COP9 signalosome. Cell Cycle. 8(13). 2041–2049. 12 indexed citations
16.
Papaioannou, Maria, Undine Schubert, Tamzin Tanner, et al.. (2008). The natural compound atraric acid is an antagonist of the human androgen receptor inhibiting cellular invasiveness and prostate cancer cell growth. Journal of Cellular and Molecular Medicine. 13(8b). 2210–2223. 45 indexed citations
17.
Eckey, Maren, Hong Wei, Maria Papaioannou, & Aria Baniahmad. (2007). The Nucleosome Assembly Activity of NAP1 Is Enhanced by Alien. Molecular and Cellular Biology. 27(10). 3557–3568. 25 indexed citations
18.
Tenbaum, Stephan P., Thomas Schlitt, Juan Bernal, et al.. (2003). Alien/CSN2 gene expression is regulated by thyroid hormone in rat brain. Developmental Biology. 254(1). 149–160. 26 indexed citations
19.
Baniahmad, Aria, Dorit Thormeyer, & Rainer Renkawitz. (1997). τ4/τc/AF-2 of the Thyroid Hormone Receptor Relieves Silencing of the Retinoic Acid Receptor Silencer Core Independent of Both τ4 Activation Function and Full Dissociation of Corepressors. Molecular and Cellular Biology. 17(8). 4259–4271. 22 indexed citations
20.
Baniahmad, Aria, et al.. (1993). 転写因子TF II Bとヒト甲状腺ホルモン受容体βとの結合は標的遺伝子の発現と甲状腺ホルモンによる活性化を仲介する. Proc Natl Acad Sci USA. 90(19). 8832–8836. 119 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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