Apichaya Puangpetch
- Pharmacology top 5%
- Molecular Biology
- Pharmacology top 5%
- Pathology and Forensic Medicine
- Rheumatology top 10%
- Co-authors
- Chonlaphat SukasemThawinee JantararoungtongNapatrupron KoomdeeMonpat ChamnanphonYaowaluck HongkaewTicha RerkpattanapipatJettanong KlaewsongkramNatchaya Vanwong
- Topics
- Drug-Induced Adverse Reactions (8 papers)Autoimmune Bullous Skin Diseases (5 papers)HIV/AIDS drug development and treatment (5 papers)
- Cited by
- PharmacologyToxicology
- Partner nations
- ThailandUnited StatesBangladesh
In The Last Decade
Apichaya Puangpetch
37 papers receiving 581 citations
Peers
Comparison fields: 5 of 84
- Pharmacology 255
- Molecular Biology 111
- Pharmacology 107
- Pathology and Forensic Medicine 97
- Rheumatology 90
Countries citing papers authored by Apichaya Puangpetch
This map shows the geographic impact of Apichaya Puangpetch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Apichaya Puangpetch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Apichaya Puangpetch more than expected).
Fields of papers citing papers by Apichaya Puangpetch
This network shows the impact of papers produced by Apichaya Puangpetch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Apichaya Puangpetch. The network helps show where Apichaya Puangpetch may publish in the future.
Co-authorship network of co-authors of Apichaya Puangpetch
This figure shows the co-authorship network connecting the top 25 collaborators of Apichaya Puangpetch. A scholar is included among the top collaborators of Apichaya Puangpetch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Apichaya Puangpetch. Apichaya Puangpetch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 3 | |
| 2 | 1 | |
| 3 | 20 | |
| 4 | 23 | |
| 5 | 28 | |
| 6 | 8 | |
| 7 | 11 | |
| 8 | 56 | |
| 9 | 7 | |
| 10 | 14 | |
| 11 | 20 | |
| 12 | 9 | |
| 13 | 44 | |
| 14 | 15 | |
| 15 | 26 | |
| 16 | 9 | |
| 17 | 44 | |
| 18 | 33 | |
| 19 | 26 | |
| 20 | 18 |
About Apichaya Puangpetch
Apichaya Puangpetch is a scholar working on Pharmacology, Virology and Rheumatology, having authored 37 papers that have together received 596 indexed citations. Recurring topics across this work include Drug-Induced Adverse Reactions (8 papers), Autoimmune Bullous Skin Diseases (5 papers) and HIV/AIDS drug development and treatment (5 papers). The work is most often cited by research in Pharmacology (255 citations), Pharmacology (107 citations) and Toxicology (37 citations). Apichaya Puangpetch has collaborated with scholars based in Thailand, United States and Bangladesh. Frequent co-authors include Chonlaphat Sukasem, Thawinee Jantararoungtong, Napatrupron Koomdee, Monpat Chamnanphon, Yaowaluck Hongkaew, Ticha Rerkpattanapipat, Jettanong Klaewsongkram, Natchaya Vanwong, Patompong Satapornpong and Phichai Chansriwong. Their work appears in journals such as PLoS ONE, Scientific Reports and Frontiers in Pharmacology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.