Annick Causse

443 total citations
11 papers, 346 citations indexed

About

Annick Causse is a scholar working on Molecular Biology, Oncology and Genetics. According to data from OpenAlex, Annick Causse has authored 11 papers receiving a total of 346 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 6 papers in Oncology and 4 papers in Genetics. Recurrent topics in Annick Causse's work include Cancer therapeutics and mechanisms (3 papers), DNA Repair Mechanisms (3 papers) and Cancer-related Molecular Pathways (3 papers). Annick Causse is often cited by papers focused on Cancer therapeutics and mechanisms (3 papers), DNA Repair Mechanisms (3 papers) and Cancer-related Molecular Pathways (3 papers). Annick Causse collaborates with scholars based in France and Israel. Annick Causse's co-authors include Pierre Martineau, Céline Gongora, Maguy Del Rio, Nadia Vezzio-Vié, Vincent Denis, Charles Theillet, Carmen Rodríguez, Laetitia Marzi, Salomé Paillas and Arnaud Coquelle and has published in prestigious journals such as Cancer Research, Molecular Cancer and Autophagy.

In The Last Decade

Annick Causse

11 papers receiving 338 citations

Peers

Annick Causse

MB

ONCOL

CR

PFM

EPIDE

Moses Adeyanju United States

Carla Kantara United States

Pamela Rutherford United States

Yinyin Li United States

M.J. Tilby United Kingdom

Jiawen Tian China

Chu Myong Seong South Korea

Nadia Vezzio-Vié France

Monika Lamparska‐Przybysz Poland

Andreas Ziesch Germany

Moses Adeyanju United States

Annick Causse

294 ×1.2

MB

143 ×0.9

ONCOL

89 ×1.6

CR

68 ×1.3

PFM

17 ×0.3

EPIDE

Citations per year, relative to Annick Causse Annick Causse (= 1×) peers Moses Adeyanju

Countries citing papers authored by Annick Causse

Since Specialization

Citations

This map shows the geographic impact of Annick Causse's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Annick Causse with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Annick Causse more than expected).

Fields of papers citing papers by Annick Causse

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Annick Causse. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Annick Causse. The network helps show where Annick Causse may publish in the future.

Co-authorship network of co-authors of Annick Causse

This figure shows the co-authorship network connecting the top 25 collaborators of Annick Causse. A scholar is included among the top collaborators of Annick Causse based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Annick Causse. Annick Causse is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

1.

Mazard, Thibault, Annick Causse, J Simony, et al.. (2013). Sorafenib Overcomes Irinotecan Resistance in Colorectal Cancer by Inhibiting the ABCG2 Drug-Efflux Pump. Molecular Cancer Therapeutics. 12(10). 2121–2134. 43 indexed citations

2.

Paillas, Salomé, Annick Causse, Laetitia Marzi, et al.. (2012). MAPK14/p38α confers irinotecan resistance to TP53-defective cells by inducing survival autophagy. Autophagy. 8(7). 1098–1112. 73 indexed citations

3.

Gongora, Céline, Nadia Vezzio-Vié, Sandie Tuduri, et al.. (2011). New Topoisomerase I mutations are associated with resistance to camptothecin. Molecular Cancer. 10(1). 64–64. 56 indexed citations

4.

Paillas, Salomé, Florence Boissière‐Michot, Frédéric Bibeau, et al.. (2010). Targeting the p38 MAPK Pathway Inhibits Irinotecan Resistance in Colon Adenocarcinoma. Cancer Research. 71(3). 1041–1049. 76 indexed citations

5.

Rodríguez, Carmen, et al.. (2001). Involvement of ATM missense variants and mutations in a series of unselected breast cancer cases. Genes Chromosomes and Cancer. 33(2). 141–149. 13 indexed citations

6.

Rodríguez, Carmen, et al.. (2000). At least five regions of imbalance on 6q in breast tumors, combining losses and gains. Genes Chromosomes and Cancer. 27(1). 76–84. 49 indexed citations

7.

Rodrı́guez, Carmen, et al.. (2000). At least five regions of imbalance on 6q in breast tumors, combining losses and gains. Genes Chromosomes and Cancer. 27(1). 76–84. 3 indexed citations

8.

Cuny, Marguerite, J Simony-Lafontaine, Julien Grenier, et al.. (1994). Quantification of ERBB2 protein expression in breast cancer: three levels of expression defined by their clinico-pathological correlations.. PubMed. 6(4-5). 169–76. 7 indexed citations

9.

Causse, Annick, et al.. (1986). Enzyme immunoassay for human chorionic gonadotropin using monoclonal antibodies elicited with a synthetic peptide. Journal of Immunological Methods. 92(1). 89–95. 14 indexed citations

10.

Caraux, Jean, et al.. (1981). Human T lymphocyte colonies. I. Surface markers and cytotoxic potential of colony cells.. PubMed. 34(1). 34–9. 2 indexed citations

11.

Klein, Bernard, et al.. (1981). Human T-lymphocyte colonies: generation of colonies in different lymphocyte subpopulations.. PubMed. 43(1). 39–45. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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