Anne V. Powles

967 total citations
18 papers, 721 citations indexed

About

Anne V. Powles is a scholar working on Immunology, Dermatology and Physiology. According to data from OpenAlex, Anne V. Powles has authored 18 papers receiving a total of 721 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Immunology, 8 papers in Dermatology and 4 papers in Physiology. Recurrent topics in Anne V. Powles's work include Psoriasis: Treatment and Pathogenesis (12 papers), Dermatology and Skin Diseases (8 papers) and Asthma and respiratory diseases (4 papers). Anne V. Powles is often cited by papers focused on Psoriasis: Treatment and Pathogenesis (12 papers), Dermatology and Skin Diseases (8 papers) and Asthma and respiratory diseases (4 papers). Anne V. Powles collaborates with scholars based in United Kingdom, United States and Sweden. Anne V. Powles's co-authors include Lionel Fry, Barbara S. Baker, Lars Engstrand, Kay E. Davies, Mark I. McCarthy, Robert Williamson, James L. Weber, Elizabeth Fisher, J. Garioch and Catherine Hardman and has published in prestigious journals such as Nature Genetics, Journal of Investigative Dermatology and Trends in Immunology.

In The Last Decade

Anne V. Powles

18 papers receiving 699 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Anne V. Powles United Kingdom 12 513 360 150 129 82 18 721
Michael Buslau Germany 9 353 0.7× 248 0.7× 62 0.4× 70 0.5× 66 0.8× 19 596
Magali de Heusch Belgium 11 559 1.1× 140 0.4× 85 0.6× 69 0.5× 86 1.0× 15 674
Danielle D. Kish United States 15 564 1.1× 135 0.4× 151 1.0× 51 0.4× 130 1.6× 28 848
Trevor R. F. Smith United States 9 630 1.2× 181 0.5× 75 0.5× 436 3.4× 68 0.8× 13 987
Erika Rickel United States 8 334 0.7× 122 0.3× 143 1.0× 198 1.5× 41 0.5× 13 683
Astrid van Leeuwen Netherlands 12 116 0.2× 407 1.1× 97 0.6× 121 0.9× 33 0.4× 19 983
Hanne Jansen Netherlands 5 428 0.8× 313 0.9× 44 0.3× 446 3.5× 38 0.5× 6 916
Susan Harvey United Kingdom 14 152 0.3× 163 0.5× 160 1.1× 37 0.3× 87 1.1× 27 638
Martin E. Dahl United States 8 615 1.2× 316 0.9× 86 0.6× 619 4.8× 50 0.6× 9 1.1k
Judith Zumkehr Switzerland 8 297 0.6× 235 0.7× 47 0.3× 333 2.6× 43 0.5× 8 750

Countries citing papers authored by Anne V. Powles

Since Specialization
Citations

This map shows the geographic impact of Anne V. Powles's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anne V. Powles with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anne V. Powles more than expected).

Fields of papers citing papers by Anne V. Powles

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anne V. Powles. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anne V. Powles. The network helps show where Anne V. Powles may publish in the future.

Co-authorship network of co-authors of Anne V. Powles

This figure shows the co-authorship network connecting the top 25 collaborators of Anne V. Powles. A scholar is included among the top collaborators of Anne V. Powles based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anne V. Powles. Anne V. Powles is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Fry, Lionel, Barbara S. Baker, Anne V. Powles, & Lars Engstrand. (2014). Psoriasis is not an autoimmune disease?. Experimental Dermatology. 24(4). 241–244. 41 indexed citations
2.
Engstrand, Lars, et al.. (2011). Comparison of bacterial microbiota in skin biopsies from normal and psoriatic skin. Archives of Dermatological Research. 304(1). 15–22. 211 indexed citations
3.
Sela, Shlomo, et al.. (2010). Evidence for the presence of bacteria in the blood of psoriasis patients. Archives of Dermatological Research. 302(7). 495–498. 41 indexed citations
4.
Nahary, Limor, Shlomo Sela, Lionel Fry, et al.. (2008). An investigation of antistreptococcal antibody responses in guttate psoriasis. Archives of Dermatological Research. 300(8). 441–449. 11 indexed citations
5.
Kainu, Kati, Katja Kivinen, Marco Zucchelli, et al.. (2008). Association of psoriasis to PGLYRP and SPRR genes at PSORS4 locus on 1q shows heterogeneity between Finnish, Swedish and Irish families. Experimental Dermatology. 18(2). 109–115. 34 indexed citations
6.
Fry, Lionel, Barbara S. Baker, & Anne V. Powles. (2006). Psoriasis—a Possible Candidate for Vaccination. Journal of Immunology Research. 13(2-4). 361–367. 3 indexed citations
7.
Baker, Barbara S., Anne V. Powles, & Lionel Fry. (2006). Peptidoglycan: a major aetiological factor for psoriasis?. Trends in Immunology. 27(12). 545–551. 33 indexed citations
8.
Fry, Lionel, Barbara S. Baker, & Anne V. Powles. (2006). Psoriasis — A possible candidate for vaccination. Autoimmunity Reviews. 6(5). 286–289. 4 indexed citations
9.
Parkinson, James R.C., Céline Charon, Barbara S. Baker, et al.. (2004). Variation at the IRF2 Gene and Susceptibility to Psoriasis in Chromosome 4q-Linked Families. Journal of Investigative Dermatology. 122(3). 640–643. 6 indexed citations
10.
Baker, Barbara S., Jean-Marc Ovigne, Vincent A. Fischetti, Anne V. Powles, & Lionel Fry. (2003). Reduced IFN-γ Responses Associated with HLA-DR15 Presentation of Streptococcal Cell Wall Proteins to Dermal Th-1 Cells in Psoriasis. Journal of Clinical Immunology. 23(5). 407–414. 8 indexed citations
11.
Baker, Barbara S., J.‐M. Ovigne, Vincent A. Fischetti, et al.. (2001). Non-M protein(s) on the cell wall and membrane of group A streptococci induce(s) IFN-γ production by dermal CD4 + T cells in psoriasis. Archives of Dermatological Research. 293(4). 165–170. 16 indexed citations
12.
Ovigne, Jean-Marc, et al.. (2000). Skin CD4+ T Cells Produce Interferon-γIn Vitro in Response to Streptococcal Antigens in Chronic Plaque Psoriasis. Journal of Investigative Dermatology. 114(3). 576–580. 37 indexed citations
13.
Baker, Barbara S., J. Garioch, Catherine Hardman, Anne V. Powles, & Lionel Fry. (1997). Induction of cutaneous lymphocyte-associated antigen expression by group A streptococcal antigens in psoriasis. Archives of Dermatological Research. 289(12). 671–676. 23 indexed citations
14.
Fry, Lionel, Anne V. Powles, James L. Weber, et al.. (1996). Evidence that a locus for familial psoriasis maps to chromosome 4q. Nature Genetics. 14(2). 231–233. 166 indexed citations
15.
Baker, Barbara S., et al.. (1996). Cytokine expression in psoriatic skin lesions during PUVA therapy. Archives of Dermatological Research. 288(8). 421–425. 43 indexed citations
16.
Baker, Barbara S., et al.. (1996). Cytokine expression in psoriatic skin lesions during PUVA therapy. Archives of Dermatological Research. 288(8). 421–425. 9 indexed citations
17.
Baker, Barbara S., et al.. (1995). A comparison of the stimulatory effects of cytokines on normal and psoriatic keratinocytes in vitro. Archives of Dermatological Research. 287(3-4). 231–236. 24 indexed citations
18.
Garioch, J., et al.. (1993). Normal response to tumor necrosis factor‐alpha and transforming growth factor‐beta by keratinocytes in psoriasis. Experimental Dermatology. 2(5). 224–230. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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