Anne Korwitz

1.3k total citations
9 papers, 1.0k citations indexed

About

Anne Korwitz is a scholar working on Molecular Biology, Clinical Biochemistry and Cellular and Molecular Neuroscience. According to data from OpenAlex, Anne Korwitz has authored 9 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 4 papers in Clinical Biochemistry and 2 papers in Cellular and Molecular Neuroscience. Recurrent topics in Anne Korwitz's work include Mitochondrial Function and Pathology (8 papers), ATP Synthase and ATPases Research (7 papers) and Metabolism and Genetic Disorders (4 papers). Anne Korwitz is often cited by papers focused on Mitochondrial Function and Pathology (8 papers), ATP Synthase and ATPases Research (7 papers) and Metabolism and Genetic Disorders (4 papers). Anne Korwitz collaborates with scholars based in Germany, Spain and Italy. Anne Korwitz's co-authors include Thomas Langer, Elena I. Rugarli, Michael J. Baker, Takashi Tatsuta, Philipp Lampe, Ricarda Richter‐Dennerlein, Ruchika Anand, Diana Stojanovski, Carsten Merkwirth and Paola Martinelli and has published in prestigious journals such as Journal of Clinical Investigation, The Journal of Experimental Medicine and The Journal of Cell Biology.

In The Last Decade

Anne Korwitz

9 papers receiving 1.0k citations

Peers

Anne Korwitz

MB

CB

PHYSI

EPIDE

CB

Shuxia Meng United States

Valentina Debattisti United States

Veronica Granatiero Italy

Marta Zaninello Italy

Michele D. Allen United States

Johanna H.K. Kauppila Germany

Marlène Daloyau France

Davor Ivankovic United Kingdom

Sofía García United States

Esther Barth Germany

Shuxia Meng United States

Anne Korwitz

796 ×0.9

MB

199 ×0.8

CB

98 ×0.5

PHYSI

121 ×0.8

EPIDE

120 ×1.0

CB

Citations per year, relative to Anne Korwitz Anne Korwitz (= 1×) peers Shuxia Meng

Countries citing papers authored by Anne Korwitz

Since Specialization

Citations

This map shows the geographic impact of Anne Korwitz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anne Korwitz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anne Korwitz more than expected).

Fields of papers citing papers by Anne Korwitz

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anne Korwitz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anne Korwitz. The network helps show where Anne Korwitz may publish in the future.

Co-authorship network of co-authors of Anne Korwitz

This figure shows the co-authorship network connecting the top 25 collaborators of Anne Korwitz. A scholar is included among the top collaborators of Anne Korwitz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anne Korwitz. Anne Korwitz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Korwitz, Anne, Carsten Merkwirth, Ricarda Richter‐Dennerlein, et al.. (2016). Loss of OMA1 delays neurodegeneration by preventing stress-induced OPA1 processing in mitochondria. The Journal of Experimental Medicine. 213(2). 2132OIA1–2132OIA1. 1 indexed citations

2.

König, Tim, Simon E. Tröder, Kavya Bakka, et al.. (2016). The m -AAA Protease Associated with Neurodegeneration Limits MCU Activity in Mitochondria. Molecular Cell. 64(1). 148–162. 152 indexed citations

3.

Korwitz, Anne, Carsten Merkwirth, Ricarda Richter‐Dennerlein, et al.. (2016). Loss of OMA1 delays neurodegeneration by preventing stress-induced OPA1 processing in mitochondria. The Journal of Cell Biology. 212(2). 157–166. 114 indexed citations

4.

Cosialls, Ana M., Diana M. González‐Gironès, Helena Pomares, et al.. (2015). A novel prohibitin-binding compound induces the mitochondrial apoptotic pathway through NOXA and BIM upregulation. Oncotarget. 6(39). 41750–41765. 29 indexed citations

5.

Kondadi, Arun Kumar, Shaomeng Wang, Sara Montagner, et al.. (2014). Loss of the m-AAA protease subunit AFG3L2 causes mitochondrial transport defects and tau hyperphosphorylation. The EMBO Journal. 33(9). 1011–1026. 64 indexed citations

6.

Richter‐Dennerlein, Ricarda, Anne Korwitz, Mathias Haag, et al.. (2014). DNAJC19, a Mitochondrial Cochaperone Associated with Cardiomyopathy, Forms a Complex with Prohibitins to Regulate Cardiolipin Remodeling. Cell Metabolism. 20(1). 158–171. 160 indexed citations

7.

Baker, Michael J., Philipp Lampe, Diana Stojanovski, et al.. (2014). Stress-induced OMA1 activation and autocatalytic turnover regulate OPA1-dependent mitochondrial dynamics. The EMBO Journal. 33(6). 578–593. 262 indexed citations

8.

Sepúlveda‐Falla, Diego, Álvaro Barrera-Ocampo, Christian Hagel, et al.. (2014). Familial Alzheimer’s disease–associated presenilin-1 alters cerebellar activity and calcium homeostasis. Journal of Clinical Investigation. 124(4). 1552–1567. 104 indexed citations

9.

Merkwirth, Carsten, Paola Martinelli, Anne Korwitz, et al.. (2012). Loss of Prohibitin Membrane Scaffolds Impairs Mitochondrial Architecture and Leads to Tau Hyperphosphorylation and Neurodegeneration. PLoS Genetics. 8(11). e1003021–e1003021. 136 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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