Annalisa Facchini
About
Annalisa Facchini is a scholar working on Molecular Biology, Pharmacology and Biochemistry.
According to data from OpenAlex, Annalisa Facchini has authored 23 papers receiving a total of 720 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 6 papers in Pharmacology and 6 papers in Biochemistry. Recurrent topics in Annalisa Facchini's work include Polyamine Metabolism and Applications (13 papers), Amino Acid Enzymes and Metabolism (4 papers) and Osteoarthritis Treatment and Mechanisms (4 papers). Annalisa Facchini is often cited by papers focused on Polyamine Metabolism and Applications (13 papers), Amino Acid Enzymes and Metabolism (4 papers) and Osteoarthritis Treatment and Mechanisms (4 papers). Annalisa Facchini collaborates with scholars based in Italy, United States and India. Annalisa Facchini's co-authors include Flavio Flamigni, Claudio Stefanelli, Rosa Maria Borzı̀, Claudio Marcello Caldarera, Andrea Facchini, Ivana Stanić, Benedetta Tantini, Francesca Bonavita, Eleonora Olivotto and Silvia Cetrullo and has published in prestigious journals such as PLoS ONE, Biochemical Journal and FEBS Letters.
In The Last Decade
Annalisa Facchini
23 papers receiving 714 citations
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late) cites · hero ref
| Name | h | Career | Trend | Papers | Cites | |||||
|---|---|---|---|---|---|---|---|---|---|---|
| Annalisa Facchini Italy | 17 | 443 | 205 | 137 | 109 | 82 | 23 | 720 | ||
| Jesalyn Bolduc United States | 7 | 340 0.8× | 291 1.4× | 107 0.8× | 82 0.8× | 49 0.6× | 8 | 676 | ||
| Ivana Stanić Italy | 18 | 689 1.6× | 40 0.2× | 158 1.2× | 64 0.6× | 129 1.6× | 34 | 880 | ||
| M A Alvarez-Soria Spain | 9 | 181 0.4× | 354 1.7× | 288 2.1× | 93 0.9× | 17 0.2× | 11 | 631 | ||
| Toshinori Bito Japan | 22 | 302 0.7× | 146 0.7× | 64 0.5× | 59 0.5× | 14 0.2× | 44 | 1.1k | ||
| Fanny Robbesyn France | 12 | 292 0.7× | 58 0.3× | 40 0.3× | 67 0.6× | 41 0.5× | 15 | 707 | ||
| Chunshu Yang China | 22 | 749 1.7× | 144 0.7× | 60 0.4× | 31 0.3× | 56 0.7× | 46 | 1.3k | ||
| N. Patrick McCabe United States | 13 | 318 0.7× | 35 0.2× | 129 0.9× | 110 1.0× | 19 0.2× | 17 | 753 | ||
| Kye‐Im Jeon United States | 13 | 345 0.8× | 29 0.1× | 64 0.5× | 136 1.2× | 19 0.2× | 19 | 743 | ||
| Shan Zhao China | 14 | 368 0.8× | 26 0.1× | 41 0.3× | 113 1.0× | 36 0.4× | 43 | 705 |
Countries citing papers authored by Annalisa Facchini
Since
Specialization
Citations
This map shows the geographic impact of Annalisa Facchini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Annalisa Facchini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Annalisa Facchini more than expected).
Fields of papers citing papers by Annalisa Facchini
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by Annalisa Facchini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Annalisa Facchini. The network helps show where Annalisa Facchini may publish in the future.
Co-authorship network of co-authors of Annalisa Facchini
This figure shows the co-authorship network connecting the top 25 collaborators of Annalisa Facchini. A scholar is included among the top collaborators of Annalisa Facchini based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Annalisa Facchini. Annalisa Facchini is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Facchini, Annalisa, Silvia Cetrullo, Stefania D’Adamo, et al.. (2014). Hydroxytyrosol Prevents Increase of Osteoarthritis Markers in Human Chondrocytes Treated with Hydrogen Peroxide or Growth-Related Oncogene α. PLoS ONE. 9(10). e109724–e109724.
2.
Facchini, Annalisa, Daniela Platano, Eleonora Olivotto, et al.. (2013). Enhanced Osteoblastogenesis of Adipose-Derived Stem Cells on Spermine Delivery via β-Catenin Activation. Stem Cells and Development. 22(10). 1588–1601.
3.
Olivotto, Eleonora, Miguel Otero, Annalisa Astolfi, et al.. (2013). IKKα/CHUK Regulates Extracellular Matrix Remodeling Independent of Its Kinase Activity to Facilitate Articular Chondrocyte Differentiation. PLoS ONE. 8(9). e73024–e73024.
4.
Borzı̀, Rosa Maria, Manuela Minguzzi, Annalisa Facchini, et al.. (2013). Polyamine delivery as a tool to modulate stem cell differentiation in skeletal tissue engineering. Amino Acids. 46(3). 717–728.
5.
Cetrullo, Silvia, Benedetta Tantini, Annalisa Facchini, et al.. (2010). A pro-survival effect of polyamine depletion on norepinephrine-mediated apoptosis in cardiac cells: role of signaling enzymes. Amino Acids. 40(4). 1127–1137.
6.
Facchini, Annalisa, Ivana Stanić, Silvia Cetrullo, et al.. (2010). Sulforaphane protects human chondrocytes against cell death induced by various stimuli. Journal of Cellular Physiology. 226(7). 1771–1779.
7.
Borzı̀, Rosa Maria, Eleonora Olivotto, Stefania Pagani, et al.. (2010). Matrix metalloproteinase 13 loss associated with impaired extracellular matrix remodeling disrupts chondrocyte differentiation by concerted effects on multiple regulatory factors. Arthritis & Rheumatism. 62(8). 2370–2381.
8.
Stanić, Ivana, Annalisa Facchini, Rosa Maria Borzı̀, Claudio Stefanelli, & Flavio Flamigni. (2008). The polyamine analogue N1,N11‐diethylnorspermine can induce chondrocyte apoptosis independently of its ability to alter metabolism and levels of natural polyamines. Journal of Cellular Physiology. 219(1). 109–116.
9.
Stanić, Ivana, Silvia Cetrullo, Annalisa Facchini, et al.. (2008). Effect of the polyamine analogue N1,N11‐diethylnorspermine on cell survival and susceptibility to apoptosis of human chondrocytes. Journal of Cellular Physiology. 216(1). 153–161.
10.
Olivotto, Eleonora, Rosa Maria Borzı̀, Stefania Pagani, et al.. (2007). Differential requirements for IKKα and IKKβ in the differentiation of primary human osteoarthritic chondrocytes. Arthritis & Rheumatism. 58(1). 227–239.
11.
Olivotto, Eleonora, Patricia Fernández, Elisabetta Falcieri, et al.. (2006). Chondrocyte hypertrophy and apoptosis induced by GROα require three‐dimensional interaction with the extracellular matrix and a co‐receptor role of chondroitin sulfate and are associated with the mitochondrial splicing variant of cathepsin B. Journal of Cellular Physiology. 210(2). 417–427.
12.
Facchini, Annalisa, Rosa Maria Borzı̀, Kenneth B. Marcu, et al.. (2005). Polyamine depletion inhibits NF‐κB binding to DNA and interleukin‐8 production in human chondrocytes stimulated by tumor necrosis factor‐α. Journal of Cellular Physiology. 204(3). 956–963.
13.
Stanić, Ivana, Annalisa Facchini, Rosa Maria Borzı̀, et al.. (2005). Polyamine depletion inhibits apoptosis following blocking of survival pathways in human chondrocytes stimulated by tumor necrosis factor‐α. Journal of Cellular Physiology. 206(1). 138–146.
14.
Facchini, Annalisa, Rosa Maria Borzı̀, & Flavio Flamigni. (2005). Induction of ornithine decarboxylase in T/C‐28a2 chondrocytes by lysophosphatidic acid: Signaling pathway and inhibition of cell proliferation. FEBS Letters. 579(13). 2919–2925.
15.
Bonavita, Francesca, Claudio Stefanelli, Emanuele� Giordano, et al.. (2003). H9c2 cardiac myoblasts undergo apoptosis in a model of ischemia consisting of serum deprivation and hypoxia: inhibition by PMA. FEBS Letters. 536(1-3). 85–91.
16.
Facchini, Annalisa, et al.. (2003). Effect of Green Tea Extract on the Induction of Ornithine Decarboxylase and the Activation of Extracellular Signal-Regulated Kinase in Bladder Carcinoma ECV304 Cells. Nutrition and Cancer. 47(1). 104–111.
17.
Ghelli, Anna, Anna Maria Porcelli, Annalisa Facchini, et al.. (2002). Phospholipase D1 is threonine-phosphorylated in human-airway epithelial cells stimulated by sphingosine-1-phosphate by a mechanism involving Src tyrosine kinase and protein kinase Cδ. Biochemical Journal. 366(1). 187–193.
18.
Flamigni, Flavio, Annalisa Facchini, Ivana Stanić, et al.. (2001). Control of survival of proliferating L1210 cells by soluble guanylate cyclase and p44/42 mitogen-activated protein kinase modulators11Abbreviations: L-NMMA, NG-monomethyl-l-arginine; MAPK, mitogen-activated protein kinase; NO, nitric oxide; NOS, nitric oxide synthase; ODQ, 1H-[1,2,4]oxadiazole [4,3-a]quinoxalin-1-one; PI3K, phosphoinositide 3-kinase; PKA, cyclic AMP-dependent protein kinase; PMA, phorbol myristate acetate; sGC, soluble guanylate cyclase; and SNAP, S-nitroso-N-acetyl-d,l-penicillamine.. Biochemical Pharmacology. 62(3). 319–328.
19.
Flamigni, Flavio, Annalisa Facchini, Emanuele� Giordano, Benedetta Tantini, & Claudio Stefanelli. (2001). Signaling pathways leading to the induction of ornithine decarboxylase: Opposite effects of p44/42 mitogen-activated protein kinase (MAPK) and p38 MAPK inhibitors. Biochemical Pharmacology. 61(1). 25–32.
20.
Stefanelli, Claudio, Francesca Bonavita, Ivana Stanić, et al.. (1998). Spermine causes caspase activation in leukaemia cells. FEBS Letters. 437(3). 233–236.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
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