Anna Pellattiero

412 total citations
7 papers, 160 citations indexed

About

Anna Pellattiero is a scholar working on Molecular Biology, Hematology and Cancer Research. According to data from OpenAlex, Anna Pellattiero has authored 7 papers receiving a total of 160 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 1 paper in Hematology and 1 paper in Cancer Research. Recurrent topics in Anna Pellattiero's work include Mitochondrial Function and Pathology (7 papers), ATP Synthase and ATPases Research (7 papers) and Acute Myeloid Leukemia Research (1 paper). Anna Pellattiero is often cited by papers focused on Mitochondrial Function and Pathology (7 papers), ATP Synthase and ATPases Research (7 papers) and Acute Myeloid Leukemia Research (1 paper). Anna Pellattiero collaborates with scholars based in Italy, United States and Japan. Anna Pellattiero's co-authors include Luca Scorrano, María Eugenia Soriano, José Antonio Enrı́quez, Rubén Quintana–Cabrera, Paolo Bernardi, Christina Glytsou, Enrique Calvo, Andrea Urbani, Mauro Corrado and Jesús Vázquez and has published in prestigious journals such as Nature Communications, Science Advances and Cell Death and Disease.

In The Last Decade

Anna Pellattiero

7 papers receiving 159 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Anna Pellattiero Italy	5	144	43	22	16	14	7	160
Abhishek Aich Germany	9	251 1.7×	66 1.5×	29 1.3×	17 1.1×	18 1.3×	12	305
Vendula Havlíčková Czechia	4	186 1.3×	80 1.9×	15 0.7×	16 1.0×	11 0.8×	6	213
Claire Churchhouse United States	6	95 0.7×	10 0.2×	32 1.5×	20 1.3×	14 1.0×	7	184
Rocío Rius Australia	8	109 0.8×	49 1.1×	9 0.4×	7 0.4×	9 0.6×	10	162
Rose McGlauflin United States	3	161 1.1×	18 0.4×	35 1.6×	77 4.8×	19 1.4×	3	193
Renaud Vatrinet Italy	5	112 0.8×	15 0.3×	79 3.6×	35 2.2×	7 0.5×	6	166
Jennifer Trombley United States	4	93 0.6×	8 0.2×	8 0.4×	14 0.9×	13 0.9×	5	159
Zhongfu Ying China	5	190 1.3×	24 0.6×	89 4.0×	8 0.5×	19 1.4×	7	236
Caroline Biagosch Germany	2	71 0.5×	27 0.6×	7 0.3×	22 1.4×	12 0.9×	2	102
Navratan Bagwan Spain	7	145 1.0×	8 0.2×	13 0.6×	14 0.9×	10 0.7×	10	232

Countries citing papers authored by Anna Pellattiero

Since Specialization

Citations

This map shows the geographic impact of Anna Pellattiero's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anna Pellattiero with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anna Pellattiero more than expected).

Fields of papers citing papers by Anna Pellattiero

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anna Pellattiero. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anna Pellattiero. The network helps show where Anna Pellattiero may publish in the future.

Co-authorship network of co-authors of Anna Pellattiero

This figure shows the co-authorship network connecting the top 25 collaborators of Anna Pellattiero. A scholar is included among the top collaborators of Anna Pellattiero based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anna Pellattiero. Anna Pellattiero is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Witkowski, Matthew T., Anna Pellattiero, Kristina Ames, et al.. (2025). Small-molecule OPA1 inhibitors reverse mitochondrial adaptations to overcome therapy resistance in acute myeloid leukemia. Science Advances. 11(42). eadx8662–eadx8662. 1 indexed citations

2.

Pellattiero, Anna, et al.. (2025). Metastatic breast cancer cells are selectively dependent on the mitochondrial cristae-shaping protein OPA1. Cell Death and Disease. 16(1). 539–539. 5 indexed citations

3.

Pellattiero, Anna, Mattia Sturlese, Nikolaos Biris, et al.. (2025). Small molecule OPA1 inhibitors amplify cytochrome c release and reverse cancer cells resistance to Bcl-2 inhibitors. Science Advances. 11(27). eadx4562–eadx4562. 5 indexed citations

4.

Kohno, Susumu, Anna Pellattiero, Keitaro Shibata, et al.. (2023). Inhibition of the mitochondria-shaping protein Opa1 restores sensitivity to Gefitinib in a lung adenocarcinomaresistant cell line. Cell Death and Disease. 14(4). 241–241. 24 indexed citations

5.

Semenzato, Martina, Mark J. Kohr, Roberta Menabò, et al.. (2023). Oxidization of optic atrophy 1 cysteines occurs during heart ischemia-reperfusion and amplifies cell death by oxidative stress. Redox Biology. 63. 102755–102755. 10 indexed citations

6.

Pellattiero, Anna. (2019). Pharmacological modulation of mitochondrial dynamics: identification of a specific OPA1 inhibitor to enhance apoptotic release of cytochrome c.. Padua@research (University of Padova). 1 indexed citations

7.

Quintana–Cabrera, Rubén, Christina Glytsou, Mauro Corrado, et al.. (2018). The cristae modulator Optic atrophy 1 requires mitochondrial ATP synthase oligomers to safeguard mitochondrial function. Nature Communications. 9(1). 3399–3399. 114 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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