Anna Maria Musti
- Molecular Biology top 5%
- Genetics top 1%
- Cellular and Molecular Neuroscience top 2%
- Oncology top 5%
- Endocrinology, Diabetes and Metabolism top 2%
- Co-authors
- Marcello MaggioliniDirk BohmannMathias TreierAdele VivacquaSebastiano AndòGeorg NagelPeter HegemannDoris Ollig
- Topics
- Cell death mechanisms and regulation (7 papers)Cancer-related Molecular Pathways (7 papers)Protein Kinase Regulation and GTPase Signaling (6 papers)
- Partner nations
- ItalyGermanyUnited States
In The Last Decade
Anna Maria Musti
38 papers receiving 3.7k citations
Hit Papers
Peers
Comparison fields: 5 of 118
- Molecular Biology 2.2k
- Genetics 1.2k
- Cellular and Molecular Neuroscience 908
- Oncology 614
- Endocrinology, Diabetes and Metabolism 469
Countries citing papers authored by Anna Maria Musti
This map shows the geographic impact of Anna Maria Musti's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anna Maria Musti with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anna Maria Musti more than expected).
Fields of papers citing papers by Anna Maria Musti
This network shows the impact of papers produced by Anna Maria Musti. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anna Maria Musti. The network helps show where Anna Maria Musti may publish in the future.
Co-authorship network of co-authors of Anna Maria Musti
This figure shows the co-authorship network connecting the top 25 collaborators of Anna Maria Musti. A scholar is included among the top collaborators of Anna Maria Musti based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anna Maria Musti. Anna Maria Musti is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 112 | |
| 2 | 46 | |
| 3 | 36 | |
| 4 | 51 | |
| 5 | 10 | |
| 6 | 21 | |
| 7 | 64 | |
| 8 | 16 | |
| 9 | 381 | |
| 10 | Channelrhodopsin-1: A Light-Gated Proton Channel in Green Algaebreakdown → | 814 |
| 11 | 47 | |
| 12 | 80 | |
| 13 | 46 | |
| 14 | 36 | |
| 15 | 10 | |
| 16 | 36 | |
| 17 | 11 | |
| 18 | 34 | |
| 19 | 101 | |
| 20 | 38 |
About Anna Maria Musti
Anna Maria Musti is a scholar working on Molecular Biology, Genetics and Oncology, having authored 38 papers that have together received 3.8k indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (7 papers), Cancer-related Molecular Pathways (7 papers) and Protein Kinase Regulation and GTPase Signaling (6 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (908 citations), Genetics (1.2k citations) and Molecular Biology (2.2k citations). Anna Maria Musti has collaborated with scholars based in Italy, Germany and United States. Frequent co-authors include Marcello Maggiolini, Dirk Bohmann, Mathias Treier, Adele Vivacqua, Sebastiano Andò, Georg Nagel, Peter Hegemann, Doris Ollig, Markus Fuhrmann and Ernst Bamberg. Their work appears in journals such as Science, Cell and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.