Anna Kowalska

1.4k total citations
59 papers, 865 citations indexed

About

Anna Kowalska is a scholar working on Physiology, Molecular Biology and Periodontics. According to data from OpenAlex, Anna Kowalska has authored 59 papers receiving a total of 865 indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Physiology, 23 papers in Molecular Biology and 7 papers in Periodontics. Recurrent topics in Anna Kowalska's work include Alzheimer's disease research and treatments (18 papers), Prion Diseases and Protein Misfolding (7 papers) and Bioinformatics and Genomic Networks (7 papers). Anna Kowalska is often cited by papers focused on Alzheimer's disease research and treatments (18 papers), Prion Diseases and Protein Misfolding (7 papers) and Bioinformatics and Genomic Networks (7 papers). Anna Kowalska collaborates with scholars based in Poland, United States and Japan. Anna Kowalska's co-authors include Zuzanna Ślebioda, Elżbieta Szponar, Marina Kirkitadze, Takeshi Tabira, M Wender, Keikichi Takahashi, Masato Hasegawa, Wojciech Kozubski, Barbara Dorocka−Bobkowska and Nina Titenko‐Holland and has published in prestigious journals such as The Journal of Immunology, Neurology and Annals of Neurology.

In The Last Decade

Anna Kowalska

55 papers receiving 829 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Anna Kowalska Poland 16 400 274 225 144 109 59 865
Mor-Li Hartman United States 14 410 1.0× 506 1.8× 117 0.5× 13 0.1× 22 0.2× 20 1.2k
Idit Shirazi Israel 13 237 0.6× 113 0.4× 48 0.2× 19 0.1× 86 0.8× 26 739
Fei Lan China 19 133 0.3× 469 1.7× 44 0.2× 14 0.1× 25 0.2× 35 910
Byung-Yong Rhim South Korea 14 111 0.3× 241 0.9× 55 0.2× 24 0.2× 24 0.2× 29 588
Xue Zhang China 19 100 0.3× 548 2.0× 18 0.1× 60 0.4× 25 0.2× 86 1.2k
Congcong Sun China 15 171 0.4× 413 1.5× 13 0.1× 170 1.2× 18 0.2× 38 771
Hua Tong China 15 149 0.4× 309 1.1× 17 0.1× 25 0.2× 30 0.3× 35 607
M Sobaniec‐Lotowska Poland 15 48 0.1× 179 0.7× 41 0.2× 18 0.1× 25 0.2× 75 704
Ruo‐Lan Xiang China 16 249 0.6× 253 0.9× 28 0.1× 18 0.1× 10 0.1× 54 654

Countries citing papers authored by Anna Kowalska

Since Specialization
Citations

This map shows the geographic impact of Anna Kowalska's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anna Kowalska with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anna Kowalska more than expected).

Fields of papers citing papers by Anna Kowalska

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anna Kowalska. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anna Kowalska. The network helps show where Anna Kowalska may publish in the future.

Co-authorship network of co-authors of Anna Kowalska

This figure shows the co-authorship network connecting the top 25 collaborators of Anna Kowalska. A scholar is included among the top collaborators of Anna Kowalska based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anna Kowalska. Anna Kowalska is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Ślebioda, Zuzanna, et al.. (2017). The absence of an association between Interleukin 1β gene polymorphisms and recurrent aphthous stomatitis (RAS). Archives of Oral Biology. 84. 45–49. 9 indexed citations
4.
Flouris, Andreas D., Yulii V. Shidlovskii, Levon Yepiskoposyan, et al.. (2017). Role of UCP1 Gene Variants in Interethnic Differences in the Development of Cardio-Metabolic Diseases. Frontiers in Genetics. 8. 7–7. 8 indexed citations
5.
Ślebioda, Zuzanna, et al.. (2017). Vitamin D status in recurrent aphthous stomatitis. Advances in Dermatology and Allergology. 34(6). 612–617. 22 indexed citations
6.
Bednarek, Michał, et al.. (2014). Methotrexate (MTX) as a single agent for treating chronic, progressive sarcoidosis. European Respiratory Journal. 44(Suppl 58). P473–P473. 1 indexed citations
7.
Puścińska, Elżbieta, Adam Nowiński, Michał Bednarek, et al.. (2014). Mortality in sarcoidosis, results from one centre, long term observational study. European Respiratory Journal. 44(Suppl 58). P468–P468. 1 indexed citations
8.
Ślebioda, Zuzanna, Elżbieta Szponar, & Anna Kowalska. (2013). Defensins and their role in the maintenance of the oral cavity homeostasis – a literature review. Central European Journal of Immunology. 38(1). 111–117. 6 indexed citations
9.
Ślebioda, Zuzanna, Elżbieta Szponar, & Anna Kowalska. (2013). Recurrent aphthous stomatitis: genetic aspects of etiology. Advances in Dermatology and Allergology. 2(2). 96–102. 63 indexed citations
10.
Bykowska, K., Sylwia Chocholska, Katarzyna Wejksza, et al.. (2011). JAK2 mutation status, hemostatic risk factors and thrombophilic factors in essential thrombocythemia (ET) patients. Folia Histochemica et Cytobiologica. 49(2). 267–271. 6 indexed citations
11.
Miyamoto, Katsuichi, Anna Kowalska, Masato Hasegawa, et al.. (2001). Familial frontotemporal dementia and parkinsonism with a novel mutation at an intron 10+11‐splice site in the tau gene. Annals of Neurology. 50(1). 117–120. 47 indexed citations
12.
Kowalska, Anna, Takashi Asada, Kunimasa Arima, et al.. (2001). Genetic Analysis in Patients with Familial and Sporadic Frontotemporal Dementia: Two Tau Mutations in Only Familial Cases and No Association with Apolipoprotein ε4. Dementia and Geriatric Cognitive Disorders. 12(6). 387–392. 26 indexed citations
13.
Kodama, Kazuhiro, Shinichi Okada, Eizo Iseki, et al.. (2000). Familial frontotemporal dementia with a P301L tau mutation in Japan. Journal of the Neurological Sciences. 176(1). 57–64. 19 indexed citations
14.
Kowalska, Anna, et al.. (1999). A Polish pedigree with Alzheimer's disease determined by a novel mutation in exon 12 of the presenilin 1 gene: clinical and molecular characterization.. PubMed. 37(1). 57–61. 13 indexed citations
15.
Kowalska, Anna, M Wender, & Lars Lannfelt. (1998). Lack of Association between an Intronic Polymorphism in the Presenilin-1 Gene and Sporadic Late-Onset Alzheimer Disease in Polish Patients. Dementia and Geriatric Cognitive Disorders. 9(3). 137–139. 11 indexed citations
16.
Kowalska, Anna, et al.. (1998). Screening for presenilin-1 gene mutations by PCR-SSCP analysis in patients with early-onset Alzheimer's disease.. PubMed. 36(1). 32–7. 3 indexed citations
17.
Kowalska, Anna, et al.. (1998). Apolipoprotein E genotypes in sporadic early and late-onset Alzheimer's disease.. PubMed. 46(3). 177–81. 3 indexed citations
18.
Kowalska, Anna, et al.. (1996). Association between the PI*M3 allele of α1-antitrypsin and Alzheimer's disease? A preliminary report. Human Genetics. 98(6). 744–746. 8 indexed citations
19.
Kowalska, Anna, et al.. (1995). Alpha 1-Antitrypsin Subtypes in Polish Newborns. Human Heredity. 45(6). 351–354. 12 indexed citations
20.
Kowalska, Anna, et al.. (1991). Genetic polymorphism of alpha-1-antitrypsin (Locus Pi) in Polish population determined by isoelectric focusing. I. Studies of Poznan and its region. Bulletin of the Polish Academy of Sciences. Biological Sciences. 39(3). 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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