Anetta Wronska

2.9k total citations
23 papers, 1.8k citations indexed

About

Anetta Wronska is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Physiology. According to data from OpenAlex, Anetta Wronska has authored 23 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 14 papers in Cardiology and Cardiovascular Medicine and 5 papers in Physiology. Recurrent topics in Anetta Wronska's work include Ion channel regulation and function (13 papers), Cardiac electrophysiology and arrhythmias (11 papers) and Cardiovascular Effects of Exercise (4 papers). Anetta Wronska is often cited by papers focused on Ion channel regulation and function (13 papers), Cardiac electrophysiology and arrhythmias (11 papers) and Cardiovascular Effects of Exercise (4 papers). Anetta Wronska collaborates with scholars based in United States, Germany and France. Anetta Wronska's co-authors include Andrew R. Marks, Steven Reiken, Bi-Xing Chen, Stephan E. Lehnart, Gaetano Santulli, Iwona Kurkowska‐Jastrzębska, Xander H.T. Wehrens, Joshua R. Vest, Matthew J. Betzenhauser and Miroslav Důra and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and Nature Communications.

In The Last Decade

Anetta Wronska

22 papers receiving 1.8k citations

Peers

Anetta Wronska
Khalid Chakir United States
Yoshinori Nishijima United States
Olha M. Koval United States
Ilona Bódi United States
Éric Morel France
Francesca Rochais France
Ernesto A. Aiello Argentina
Dong I. Lee United States
Madeline Nieves‐Cintrón United States
Yuejin Wu United States
Khalid Chakir United States
Anetta Wronska
Citations per year, relative to Anetta Wronska Anetta Wronska (= 1×) peers Khalid Chakir

Countries citing papers authored by Anetta Wronska

Since Specialization
Citations

This map shows the geographic impact of Anetta Wronska's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anetta Wronska with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anetta Wronska more than expected).

Fields of papers citing papers by Anetta Wronska

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anetta Wronska. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anetta Wronska. The network helps show where Anetta Wronska may publish in the future.

Co-authorship network of co-authors of Anetta Wronska

This figure shows the co-authorship network connecting the top 25 collaborators of Anetta Wronska. A scholar is included among the top collaborators of Anetta Wronska based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anetta Wronska. Anetta Wronska is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Reiken, Steve, Joël Rousseau, Anetta Wronska, et al.. (2025). Universal Prime Editing Therapeutic Strategy for RyR1-Related Myopathies: A Protective Mutation Rescues Leaky RyR1 Channel. International Journal of Molecular Sciences. 26(7). 2835–2835.
2.
Miotto, Marco, Steve Reiken, Anetta Wronska, et al.. (2024). Structural basis for ryanodine receptor type 2 leak in heart failure and arrhythmogenic disorders. Nature Communications. 15(1). 8080–8080. 11 indexed citations
3.
Miotto, Marco, Steve Reiken, Haikel Dridi, et al.. (2024). Structural insights into the regulation of RyR1 by S100A1. Proceedings of the National Academy of Sciences. 121(27). e2400497121–e2400497121. 3 indexed citations
4.
Wronska, Anetta. (2022). The Role of microRNA in the Development, Diagnosis, and Treatment of Cardiovascular Disease: Recent Developments. Journal of Pharmacology and Experimental Therapeutics. 384(1). 123–132. 32 indexed citations
5.
Melville, Zephan, Haikel Dridi, Qi Yuan, et al.. (2022). A drug and ATP binding site in type 1 ryanodine receptor. Structure. 30(7). 1025–1034.e4. 31 indexed citations
6.
Miotto, Marco, Haikel Dridi, Qi Yuan, et al.. (2022). Structural analyses of human ryanodine receptor type 2 channels reveal the mechanisms for sudden cardiac death and treatment. Science Advances. 8(29). eabo1272–eabo1272. 37 indexed citations
7.
Yuan, Qi, Haikel Dridi, Oliver B. Clarke, et al.. (2021). RyR1-related myopathy mutations in ATP and calcium binding sites impair channel regulation. Acta Neuropathologica Communications. 9(1). 186–186. 11 indexed citations
8.
Yuan, Qi, Jingyi Yang, Gaetano Santulli, et al.. (2016). Maintenance of normal blood pressure is dependent on IP3R1-mediated regulation of eNOS. Proceedings of the National Academy of Sciences. 113(30). 8532–8537. 53 indexed citations
9.
Cheung, Jim W., Albano C. Méli, Wenjun Xie, et al.. (2014). Short-coupled polymorphic ventricular tachycardia at rest linked to a novel ryanodine receptor (RyR2) mutation: Leaky RyR2 channels under non-stress conditions. International Journal of Cardiology. 180. 228–236. 32 indexed citations
10.
Santulli, Gaetano, Anetta Wronska, Kunihiro Uryu, et al.. (2014). A selective microRNA-based strategy inhibits restenosis while preserving endothelial function. Journal of Clinical Investigation. 124(9). 4102–4114. 143 indexed citations
11.
Wronska, Anetta, Iwona Kurkowska‐Jastrzębska, & Gaetano Santulli. (2014). Application of microRNAs in diagnosis and treatment of cardiovascular disease. Acta Physiologica. 213(1). 60–83. 132 indexed citations
12.
Shan, Jian, Wenjun Xie, Matthew J. Betzenhauser, et al.. (2012). Calcium Leak Through Ryanodine Receptors Leads to Atrial Fibrillation in 3 Mouse Models of Catecholaminergic Polymorphic Ventricular Tachycardia. Circulation Research. 111(6). 708–717. 157 indexed citations
13.
Özcan, Lale, Catherine C. L. Wong, Gang Li, et al.. (2012). Calcium Signaling through CaMKII Regulates Hepatic Glucose Production in Fasting and Obesity. Cell Metabolism. 15(5). 739–751. 171 indexed citations
14.
Méli, Albano C., Marwan M. Refaat, Miroslav Důra, et al.. (2011). A Novel Ryanodine Receptor Mutation Linked to Sudden Death Increases Sensitivity to Cytosolic Calcium. Circulation Research. 109(3). 281–290. 48 indexed citations
15.
Shan, Jian, Matthew J. Betzenhauser, Alexander Kushnir, et al.. (2010). Role of chronic ryanodine receptor phosphorylation in heart failure and β-adrenergic receptor blockade in mice. Journal of Clinical Investigation. 120(12). 4375–4387. 182 indexed citations
16.
Marjamaa, Annukka, Päivi J. Laitinen-Forsblom, Anetta Wronska, et al.. (2009). Ryanodine receptor (RyR2) mutations in sudden cardiac death: Studies in extended pedigrees and phenotypic characterization in vitro. International Journal of Cardiology. 147(2). 246–252. 22 indexed citations
17.
Lehnart, Stephan E., Marco Mongillo, Andrew M. Bellinger, et al.. (2008). Leaky Ca2+ release channel/ryanodine receptor 2 causes seizures and sudden cardiac death in mice. Journal of Clinical Investigation. 118(6). 2230–45. 307 indexed citations
18.
Tester, David J., Miroslav Důra, Elisa Carturan, et al.. (2007). A mechanism for sudden infant death syndrome (SIDS): Stress-induced leak via ryanodine receptors. Heart Rhythm. 4(6). 733–739. 120 indexed citations
19.
Seigel, Gail M., et al.. (2004). Progression of early postnatal retinal pathology in a mouse model of neuronal ceroid lipofuscinosis. Eye. 19(12). 1306–1312. 18 indexed citations
20.
Węgiel, Jerzy, Humi Imaki, Kuo-Chiang Wang, et al.. (2003). Origin and turnover of microglial cells in fibrillar plaques of APPsw transgenic mice. Acta Neuropathologica. 105(4). 393–402. 45 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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