Andrew H. Schulick

1.6k total citations
14 papers, 1.3k citations indexed

About

Andrew H. Schulick is a scholar working on Molecular Biology, Immunology and Allergy and Genetics. According to data from OpenAlex, Andrew H. Schulick has authored 14 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 5 papers in Immunology and Allergy and 4 papers in Genetics. Recurrent topics in Andrew H. Schulick's work include Angiogenesis and VEGF in Cancer (6 papers), Cell Adhesion Molecules Research (5 papers) and Virus-based gene therapy research (4 papers). Andrew H. Schulick is often cited by papers focused on Angiogenesis and VEGF in Cancer (6 papers), Cell Adhesion Molecules Research (5 papers) and Virus-based gene therapy research (4 papers). Andrew H. Schulick collaborates with scholars based in United States, China and Japan. Andrew H. Schulick's co-authors include David A. Dichek, Renu Virmani, Kurt D. Newman, Jeffrey J. Rade, Patrick F. Dunn, Galina K. Sukhova, Peter Libby, Harry L. Bush, Gang Dong and Ross T. Lyon and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Circulation.

In The Last Decade

Andrew H. Schulick

14 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Andrew H. Schulick United States 12 648 390 297 287 241 14 1.3k
Guy Leclerc Canada 19 612 0.9× 219 0.6× 620 2.1× 280 1.0× 326 1.4× 29 1.4k
Jacob M. Waugh United States 19 658 1.0× 142 0.4× 560 1.9× 165 0.6× 164 0.7× 33 1.6k
Elizabeth M. Leaf United States 13 442 0.7× 465 1.2× 243 0.8× 138 0.5× 260 1.1× 25 1.7k
M Almeida United States 5 501 0.8× 253 0.6× 122 0.4× 79 0.3× 110 0.5× 5 1.3k
LV Parise United States 10 410 0.6× 200 0.5× 404 1.4× 381 1.3× 398 1.7× 11 2.3k
Dimitra Micha Netherlands 22 521 0.8× 633 1.6× 151 0.5× 134 0.5× 555 2.3× 73 1.5k
Sybil B. Williams United States 22 207 0.3× 165 0.4× 239 0.8× 156 0.5× 203 0.8× 36 1.2k
Shafie Fazel Canada 22 730 1.1× 108 0.3× 944 3.2× 711 2.5× 288 1.2× 49 2.0k
Andres Ferbér United States 17 901 1.4× 221 0.6× 173 0.6× 127 0.4× 96 0.4× 38 1.5k
Michael Maysky United States 7 908 1.4× 251 0.6× 572 1.9× 370 1.3× 56 0.2× 18 1.4k

Countries citing papers authored by Andrew H. Schulick

Since Specialization
Citations

This map shows the geographic impact of Andrew H. Schulick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Andrew H. Schulick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Andrew H. Schulick more than expected).

Fields of papers citing papers by Andrew H. Schulick

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Andrew H. Schulick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Andrew H. Schulick. The network helps show where Andrew H. Schulick may publish in the future.

Co-authorship network of co-authors of Andrew H. Schulick

This figure shows the co-authorship network connecting the top 25 collaborators of Andrew H. Schulick. A scholar is included among the top collaborators of Andrew H. Schulick based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Andrew H. Schulick. Andrew H. Schulick is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
1.
Shao, Hongwei, Yaohong Tan, Darwin Eton, et al.. (2008). Statin and Stromal Cell-Derived Factor-1 Additively Promote Angiogenesis by Enhancement of Progenitor Cells Incorporation into New Vessels. Stem Cells. 26(5). 1376–1384. 107 indexed citations
2.
Tan, Yaohong, Hongwei Shao, Darwin Eton, et al.. (2006). Stromal cell-derived factor-1 enhances pro-angiogenic effect of granulocyte-colony stimulating factor. Cardiovascular Research. 73(4). 823–832. 39 indexed citations
3.
Heller, Jennifer A., Alan Weinberg, Kambhampaty Krishnasastry, et al.. (2000). Two decades of abdominal aortic aneurysm repair: Have we made any progress?. Journal of Vascular Surgery. 32(6). 1091–1100. 172 indexed citations
4.
Du, Baoheng, Chenzhong Fu, K. Craig Kent, et al.. (2000). Elevated Egr-1 in Human Atherosclerotic Cells Transcriptionally Represses the Transforming Growth Factor-β Type II Receptor. Journal of Biological Chemistry. 275(50). 39039–39047. 54 indexed citations
5.
Rao, Patrick M., Colleen M. Feltmate, James T. Rhea, Andrew H. Schulick, & Robert A. Novelline. (1999). Helical Computed Tomography in Differentiating Appendicitis and Acute Gynecologic Conditions. Obstetrics and Gynecology. 93(3). 417–421. 9 indexed citations
6.
Schulick, Andrew H., Allen J. Taylor, Gang Dong, et al.. (1998). Overexpression of transforming growth factor β1 in arterial endothelium causes hyperplasia, apoptosis, and cartilaginous metaplasia. Proceedings of the National Academy of Sciences. 95(12). 6983–6988. 156 indexed citations
7.
Schulick, Andrew H., Giuseppe Vassalli, Patrick F. Dunn, et al.. (1997). Established immunity precludes adenovirus-mediated gene transfer in rat carotid arteries. Potential for immunosuppression and vector engineering to overcome barriers of immunity.. Journal of Clinical Investigation. 99(2). 209–219. 119 indexed citations
8.
Taylor, Allen J., David A. Dichek, Gang Dong, et al.. (1997). Regional Variability in the Time Course of TGF-β1 Expression, Cellular Proliferation and Extracellular Matrix Expansion following Arterial Injury. Growth Factors. 14(4). 297–306. 15 indexed citations
9.
Rade, Jeffrey J., Andrew H. Schulick, Renu Virmani, & David A. Dichek. (1996). Local adenoviral–mediated expression of recombinant hirudin reduces neointima formation after arterial injury. Nature Medicine. 2(3). 293–298. 128 indexed citations
10.
Dong, Gang, Andrew H. Schulick, Mary Beth DeYoung, & David A. Dichek. (1996). Identification of a cis-Acting Sequence in the Human Plasminogen Activator Inhibitor Type-1 Gene That Mediates Transforming Growth Factor-β1 Responsiveness in Endothelium in Vivo. Journal of Biological Chemistry. 271(47). 29969–29977. 38 indexed citations
11.
Newman, Kurt D., Patrick F. Dunn, Andrew H. Schulick, et al.. (1995). Adenovirus-mediated gene transfer into normal rabbit arteries results in prolonged vascular cell activation, inflammation, and neointimal hyperplasia.. Journal of Clinical Investigation. 96(6). 2955–2965. 259 indexed citations
12.
Schulick, Andrew H., Gang Dong, Kurt D. Newman, Renu Virmani, & David A. Dichek. (1995). Endothelium-Specific In Vivo Gene Transfer. Circulation Research. 77(3). 475–485. 106 indexed citations
13.
Schulick, Andrew H., Kurt D. Newman, Renu Virmani, & David A. Dichek. (1995). In Vivo Gene Transfer Into Injured Carotid Arteries. Circulation. 91(9). 2407–2414. 100 indexed citations
14.
Schulick, Andrew H., et al.. (1990). Effects of maitotoxin on atrial natriuretic factor-mediated accumulation of cyclic GMP in PC12 cells. Life Sciences. 46(10). 671–678. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026