Andrew E. Leitch

1.1k total citations
8 papers, 843 citations indexed

About

Andrew E. Leitch is a scholar working on Immunology, Molecular Biology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Andrew E. Leitch has authored 8 papers receiving a total of 843 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Immunology, 5 papers in Molecular Biology and 1 paper in Pulmonary and Respiratory Medicine. Recurrent topics in Andrew E. Leitch's work include Phagocytosis and Immune Regulation (5 papers), Cell death mechanisms and regulation (3 papers) and Neutrophil, Myeloperoxidase and Oxidative Mechanisms (3 papers). Andrew E. Leitch is often cited by papers focused on Phagocytosis and Immune Regulation (5 papers), Cell death mechanisms and regulation (3 papers) and Neutrophil, Myeloperoxidase and Oxidative Mechanisms (3 papers). Andrew E. Leitch collaborates with scholars based in United Kingdom and Brazil. Andrew E. Leitch's co-authors include Rodger Duffin, Adriano G. Rossi, Christopher Haslett, Sarah Fox, Chris Haslett, John M. Hallett, Christopher D. Lucas, John A. Marwick, Tara A. Sheldrake and Michela Festa and has published in prestigious journals such as PLoS ONE, FEBS Letters and Immunological Reviews.

In The Last Decade

Andrew E. Leitch

8 papers receiving 831 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Andrew E. Leitch United Kingdom 8 546 325 92 85 84 8 843
Lihuang Zhang China 16 388 0.7× 331 1.0× 143 1.6× 50 0.6× 125 1.5× 40 873
Kenji Daigo Japan 15 519 1.0× 311 1.0× 65 0.7× 67 0.8× 67 0.8× 19 949
Shauna Dauphinee Canada 11 374 0.7× 353 1.1× 60 0.7× 57 0.7× 63 0.8× 18 813
Rami Bechara France 12 353 0.6× 271 0.8× 81 0.9× 53 0.6× 89 1.1× 29 837
Constance Whitney United States 10 482 0.9× 248 0.8× 88 1.0× 40 0.5× 43 0.5× 12 808
Luigi Tortola Switzerland 14 612 1.1× 328 1.0× 109 1.2× 39 0.5× 141 1.7× 30 966
Lars‐Peter Erwig United Kingdom 14 571 1.0× 245 0.8× 110 1.2× 52 0.6× 52 0.6× 17 1.0k
Chrysothemis C. Brown United States 13 648 1.2× 373 1.1× 60 0.7× 30 0.4× 149 1.8× 28 1.1k
Peter Helding Kvist Denmark 20 251 0.5× 266 0.8× 77 0.8× 63 0.7× 72 0.9× 42 965
Jie Wan China 17 312 0.6× 310 1.0× 80 0.9× 73 0.9× 118 1.4× 30 846

Countries citing papers authored by Andrew E. Leitch

Since Specialization
Citations

This map shows the geographic impact of Andrew E. Leitch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Andrew E. Leitch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Andrew E. Leitch more than expected).

Fields of papers citing papers by Andrew E. Leitch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Andrew E. Leitch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Andrew E. Leitch. The network helps show where Andrew E. Leitch may publish in the future.

Co-authorship network of co-authors of Andrew E. Leitch

This figure shows the co-authorship network connecting the top 25 collaborators of Andrew E. Leitch. A scholar is included among the top collaborators of Andrew E. Leitch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Andrew E. Leitch. Andrew E. Leitch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Leitch, Andrew E., Christopher D. Lucas, John A. Marwick, et al.. (2012). Cyclin-dependent kinases 7 and 9 specifically regulate neutrophil transcription and their inhibition drives apoptosis to promote resolution of inflammation. Cell Death and Differentiation. 19(12). 1950–1961. 87 indexed citations
2.
Alessandri, Ana L., Rodger Duffin, Andrew E. Leitch, et al.. (2011). Induction of Eosinophil Apoptosis by the Cyclin-Dependent Kinase Inhibitor AT7519 Promotes the Resolution of Eosinophil-Dominant Allergic Inflammation. PLoS ONE. 6(9). e25683–e25683. 29 indexed citations
3.
Duffin, Rodger, Andrew E. Leitch, Sarah Fox, Chris Haslett, & Adriano G. Rossi. (2010). Targeting granulocyte apoptosis: mechanisms, models, and therapies. Immunological Reviews. 236(1). 28–40. 108 indexed citations
4.
Fox, Sarah, Andrew E. Leitch, Rodger Duffin, Christopher Haslett, & Adriano G. Rossi. (2010). Neutrophil Apoptosis: Relevance to the Innate Immune Response and Inflammatory Disease. Journal of Innate Immunity. 2(3). 216–227. 327 indexed citations
5.
Leitch, Andrew E., Tara A. Sheldrake, Michela Festa, et al.. (2010). The cyclin‐dependent kinase inhibitor R‐roscovitine down‐regulates Mcl‐1 to override pro‐inflammatory signalling and drive neutrophil apoptosis. European Journal of Immunology. 40(4). 1127–1138. 73 indexed citations
6.
Duffin, Rodger, Andrew E. Leitch, Tara A. Sheldrake, et al.. (2009). The CDK inhibitor, R‐roscovitine, promotes eosinophil apoptosis by down‐regulation of Mcl‐1. FEBS Letters. 583(15). 2540–2546. 45 indexed citations
7.
Leitch, Andrew E., et al.. (2009). Cyclin‐dependent kinase inhibitor drugs as potential novel anti‐inflammatory and pro‐resolution agents. British Journal of Pharmacology. 158(4). 1004–1016. 64 indexed citations
8.
Hallett, John M., et al.. (2008). Novel pharmacological strategies for driving inflammatory cell apoptosis and enhancing the resolution of inflammation. Trends in Pharmacological Sciences. 29(5). 250–257. 110 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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