Andreas Bühler

7.2k total citations
47 papers, 2.7k citations indexed

About

Andreas Bühler is a scholar working on Genetics, Pathology and Forensic Medicine and Immunology. According to data from OpenAlex, Andreas Bühler has authored 47 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 40 papers in Genetics, 22 papers in Pathology and Forensic Medicine and 19 papers in Immunology. Recurrent topics in Andreas Bühler's work include Chronic Lymphocytic Leukemia Research (40 papers), Lymphoma Diagnosis and Treatment (22 papers) and Immunodeficiency and Autoimmune Disorders (17 papers). Andreas Bühler is often cited by papers focused on Chronic Lymphocytic Leukemia Research (40 papers), Lymphoma Diagnosis and Treatment (22 papers) and Immunodeficiency and Autoimmune Disorders (17 papers). Andreas Bühler collaborates with scholars based in Germany, Austria and United States. Andreas Bühler's co-authors include Stephan Stilgenbauer, Hartmut Döhner, Dirk Winkler, Thorsten Zenz, Michael Hallek, Raymonde Busch, Sonja Häbe, Barbara Eichhorst, Jennifer Edelmann and Daniel Mertens and has published in prestigious journals such as Journal of Clinical Oncology, Blood and Oecologia.

In The Last Decade

Andreas Bühler

44 papers receiving 2.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Andreas Bühler Germany 21 2.2k 1.6k 920 599 520 47 2.7k
Elke Leupolt Germany 11 2.5k 1.2× 1.9k 1.2× 1.2k 1.3× 666 1.1× 573 1.1× 11 2.9k
Marta Crespo Spain 19 1.5k 0.7× 1.3k 0.8× 925 1.0× 870 1.5× 619 1.2× 52 2.7k
Anne Gardiner United Kingdom 15 3.1k 1.4× 2.2k 1.4× 1.6k 1.8× 801 1.3× 455 0.9× 27 3.4k
Alexander Kröber Germany 20 3.9k 1.8× 2.9k 1.8× 2.0k 2.1× 852 1.4× 683 1.3× 28 4.4k
Monica Else United Kingdom 22 1.6k 0.7× 1.2k 0.7× 712 0.8× 428 0.7× 403 0.8× 42 2.0k
H. Döhner Germany 19 1.1k 0.5× 850 0.5× 507 0.6× 546 0.9× 441 0.8× 27 1.8k
Francesco Forconi United Kingdom 35 3.1k 1.4× 2.1k 1.3× 1.8k 1.9× 1.1k 1.9× 860 1.7× 156 4.2k
Susana Ben‐Neriah Canada 22 990 0.5× 1.9k 1.1× 430 0.5× 504 0.8× 1.3k 2.6× 60 2.6k
Mariela Sivina United States 18 1.5k 0.7× 947 0.6× 778 0.8× 649 1.1× 367 0.7× 39 2.0k
Sharon Barrans United Kingdom 21 962 0.4× 1.7k 1.1× 402 0.4× 506 0.8× 1.0k 2.0× 54 2.3k

Countries citing papers authored by Andreas Bühler

Since Specialization
Citations

This map shows the geographic impact of Andreas Bühler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Andreas Bühler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Andreas Bühler more than expected).

Fields of papers citing papers by Andreas Bühler

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Andreas Bühler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Andreas Bühler. The network helps show where Andreas Bühler may publish in the future.

Co-authorship network of co-authors of Andreas Bühler

This figure shows the co-authorship network connecting the top 25 collaborators of Andreas Bühler. A scholar is included among the top collaborators of Andreas Bühler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Andreas Bühler. Andreas Bühler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Bühler, Andreas & Rabea Schweiger. (2024). Previous infestation by conspecifics leads to a transient increase of the performance of Sitobion avenae aphids on wheat leaves. Ecological Entomology. 49(4). 476–488. 2 indexed citations
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Bühler, Andreas & Rabea Schweiger. (2024). Niche construction and niche choice by aphids infesting wheat ears. Oecologia. 206(1-2). 47–59. 1 indexed citations
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Bühler, Andreas, Thomas J. Kipps, Laura Z. Rassenti, et al.. (2016). Lenalidomide treatment and prognostic markers in relapsed or refractory chronic lymphocytic leukemia: data from the prospective, multicenter phase-II CLL-009 trial. Blood Cancer Journal. 6(3). e404–e404. 11 indexed citations
6.
Bloehdorn, Johannes, Martin Sill, Christian Langer, et al.. (2013). High CD52 mRNA Expression Is An Adverse Prognostic Factor In Fludarabine-Refractory CLL Treated With Alemtuzumab On The Gcllsg CLL2H Trial. Blood. 122(21). 1618–1618. 1 indexed citations
7.
Jebaraj, Billy Michael Chelliah, Dirk Kienle, Andreas Bühler, et al.. (2012). BRAFmutations in chronic lymphocytic leukemia. Leukemia & lymphoma. 54(6). 1177–1182. 41 indexed citations
8.
Dreger, Peter, Hartmut Döhner, Fabienne McClanahan, et al.. (2012). Early autologous stem cell transplantation for chronic lymphocytic leukemia: long-term follow-up of the German CLL Study Group CLL3 trial. Blood. 119(21). 4851–4859. 17 indexed citations
9.
Böttcher, Sebastian, Matthias Ritgen, Kirsten Fischer, et al.. (2012). Minimal Residual Disease Quantification Is an Independent Predictor of Progression-Free and Overall Survival in Chronic Lymphocytic Leukemia: A Multivariate Analysis From the Randomized GCLLSG CLL8 Trial. Journal of Clinical Oncology. 30(9). 980–988. 305 indexed citations
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Edelmann, Jennifer, et al.. (2010). Mutation analysis of the TNFAIP3 (A20) tumor suppressor gene in CLL. International Journal of Cancer. 128(7). 1747–1750. 16 indexed citations
12.
Winkler, Dirk, Manuela Zucknick, K. Schulze, et al.. (2010). Protein expression analysis of chronic lymphocytic leukemia defines the effect of genetic aberrations and uncovers a correlation of CDK4, P27 and P53 with hierarchical risk. Haematologica. 95(11). 1880–1888. 3 indexed citations
13.
Zenz, Thorsten, Barbara Eichhorst, Raymonde Busch, et al.. (2010). TP53 Mutation and Survival in Chronic Lymphocytic Leukemia. Journal of Clinical Oncology. 28(29). 4473–4479. 413 indexed citations
14.
Zenz, Thorsten, Raymonde Busch, Anna Maria Fink, et al.. (2010). Genetics of Patients with F-Refractory CLL or Early Relapse After FC or FCR: Results From the CLL8 Trial of the GCLLSG. Blood. 116(21). 2427–2427. 14 indexed citations
15.
Kienle, Dirk, Axel Benner, Dirk Winkler, et al.. (2009). Gene expression factors as predictors of genetic risk and survival in chronic lymphocytic leukemia. Haematologica. 95(1). 102–109. 24 indexed citations
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Zenz, Thorsten, Alexander Kröber, Katrin Scherer, et al.. (2008). Monoallelic TP53 inactivation is associated with poor prognosis in chronic lymphocytic leukemia: results from a detailed genetic characterization with long-term follow-up. Blood. 112(8). 3322–3329. 269 indexed citations
19.
Kröber, Alexander, Johannes Bloehdorn, Sebastian Häfner, et al.. (2006). Additional Genetic High-Risk Features Such As 11q Deletion, 17p Deletion, and V3-21 Usage Characterize Discordance of ZAP-70 and VH Mutation Status in Chronic Lymphocytic Leukemia. Journal of Clinical Oncology. 24(6). 969–975. 134 indexed citations
20.
Kröber, Alexander, Dirk Kienle, Dirk Winkler, et al.. (2004). ZAP-70 Expression, VH-Mutation Status, Genomic Aberrations and Prognosis in Chronic Lymphocytic Leukemia.. Blood. 104(11). 1920–1920. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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