Amy L. Mize

610 total citations
8 papers, 541 citations indexed

About

Amy L. Mize is a scholar working on Genetics, Endocrinology, Diabetes and Metabolism and Cellular and Molecular Neuroscience. According to data from OpenAlex, Amy L. Mize has authored 8 papers receiving a total of 541 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Genetics, 5 papers in Endocrinology, Diabetes and Metabolism and 4 papers in Cellular and Molecular Neuroscience. Recurrent topics in Amy L. Mize's work include Estrogen and related hormone effects (7 papers), Menopause: Health Impacts and Treatments (5 papers) and Receptor Mechanisms and Signaling (3 papers). Amy L. Mize is often cited by papers focused on Estrogen and related hormone effects (7 papers), Menopause: Health Impacts and Treatments (5 papers) and Receptor Mechanisms and Signaling (3 papers). Amy L. Mize collaborates with scholars based in United States and Bulgaria. Amy L. Mize's co-authors include Richard H. Alper, Robert A. Shapiro, Daniel M. Dorsa, Julie A. Harris, Jennifer Fitzpatrick, Christian Wade, Alan Poisner, Elena V. Zoubina, Peter G. Smith and Larry J. Young and has published in prestigious journals such as Brain Research, Endocrinology and Journal of Neurochemistry.

In The Last Decade

Amy L. Mize

8 papers receiving 533 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Amy L. Mize United States 8 265 239 130 113 104 8 541
Angela S. Pechenino United States 8 219 0.8× 190 0.8× 60 0.5× 151 1.3× 97 0.9× 8 460
Sarah Gingerich Canada 8 159 0.6× 116 0.5× 79 0.6× 63 0.6× 107 1.0× 8 417
Guadalberto Hernández Spain 14 104 0.4× 141 0.6× 116 0.9× 78 0.7× 96 0.9× 30 455
V. Lenoir France 13 61 0.2× 98 0.4× 144 1.1× 71 0.6× 90 0.9× 24 409
Brice J. Williams United States 7 150 0.6× 152 0.6× 76 0.6× 94 0.8× 48 0.5× 10 348
Aili Guo Italy 12 69 0.3× 126 0.5× 107 0.8× 188 1.7× 161 1.5× 26 562
M Motta Italy 9 106 0.4× 140 0.6× 84 0.6× 50 0.4× 108 1.0× 21 456
N Laflamme Canada 12 68 0.3× 216 0.9× 59 0.5× 198 1.8× 180 1.7× 16 608
Teruko Takeo Japan 15 67 0.3× 79 0.3× 130 1.0× 63 0.6× 238 2.3× 28 621
Marilyn M. Miller United States 10 101 0.4× 81 0.3× 80 0.6× 58 0.5× 93 0.9× 12 331

Countries citing papers authored by Amy L. Mize

Since Specialization
Citations

This map shows the geographic impact of Amy L. Mize's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Amy L. Mize with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Amy L. Mize more than expected).

Fields of papers citing papers by Amy L. Mize

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Amy L. Mize. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Amy L. Mize. The network helps show where Amy L. Mize may publish in the future.

Co-authorship network of co-authors of Amy L. Mize

This figure shows the co-authorship network connecting the top 25 collaborators of Amy L. Mize. A scholar is included among the top collaborators of Amy L. Mize based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Amy L. Mize. Amy L. Mize is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Mize, Amy L., Larry J. Young, & Richard H. Alper. (2003). Uncoupling of 5-HT1A receptors in the brain by estrogens: regional variations in antagonism by ICI 182,780. Neuropharmacology. 44(5). 584–591. 41 indexed citations
2.
Mize, Amy L. & Richard H. Alper. (2002). Rapid Uncoupling of Serotonin-1A Receptors in Rat Hippocampus by 17β-Estradiol in vitro Requires Protein Kinases A and C. Neuroendocrinology. 76(6). 339–347. 31 indexed citations
3.
Fitzpatrick, Jennifer, Amy L. Mize, Christian Wade, et al.. (2002). Estrogen‐mediated neuroprotection against β‐amyloid toxicity requires expression of estrogen receptor α or β and activation of the MAPK pathway. Journal of Neurochemistry. 82(3). 674–682. 139 indexed citations
4.
Mize, Amy L., Robert A. Shapiro, & Daniel M. Dorsa. (2002). Estrogen Receptor-Mediated Neuroprotection from Oxidative Stress Requires Activation of the Mitogen-Activated Protein Kinase Pathway. Endocrinology. 144(1). 306–312. 104 indexed citations
5.
Mize, Amy L., Alan Poisner, & Richard H. Alper. (2001). Estrogens Act in Rat Hippocampus and Frontal Cortex to Produce Rapid, Receptor-Mediated Decreases in Serotonin 5-HT<sub>1A</sub> Receptor Function. Neuroendocrinology. 73(3). 166–174. 61 indexed citations
6.
Zoubina, Elena V., Amy L. Mize, Richard H. Alper, & Peter G. Smith. (2001). Acute and chronic estrogen supplementation decreases uterine sympathetic innervation in ovariectomized adult virgin rats.. PubMed. 16(4). 989–96. 75 indexed citations
7.
8.
Mize, Amy L. & Richard H. Alper. (1999). Serotonin 5-HT1B/1D agonist-stimulated [35S]GTPγS binding in rat and guinea pig striatal membranes1Published on the World Wide Web on 16 June 1999.1. Brain Research. 836(1-2). 229–236. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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