Amr Al‐Haidari

956 citations
7 papers · 580 indexed · 1 hit paper · h-index 7
Topics
MicroRNA in disease regulation (3 papers)Circular RNAs in diseases (2 papers)Cancer-related molecular mechanisms research (2 papers)
Partner nations
SwedenChina

In The Last Decade

Amr Al‐Haidari

7 papers receiving 578 citations

Hit Papers

T cell receptor (TCR) signaling in health and disease20212026202220242021100200300

Peers

Amr Al‐Haidari
Comparison fields: 5 of 81
  • Molecular Biology 255
  • Immunology 249
  • Oncology 171
  • Cancer Research 153
  • Pulmonary and Respiratory Medicine 40
Replace Nima Etemadi with:
Nima Etemadi Australia
Els Louagie Belgium
Samuel D. Saibil Canada
Meidi Gu United States
Yulan Qing United States
Karine Enesa United Kingdom
Lucia Minoli Italy
Jessica McCready United States
Polina Weitzenfeld Israel
Amr Al‐Haidari relative to Nima Etemadi Australia Nima Etemadi's profile →
Citations per field
00.5×4.5×
Nima Etemadi · 1×
Citations per year

Countries citing papers authored by Amr Al‐Haidari

Since Specialization
Citations

This map shows the geographic impact of Amr Al‐Haidari's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Amr Al‐Haidari with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Amr Al‐Haidari more than expected).

Fields of papers citing papers by Amr Al‐Haidari

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Amr Al‐Haidari. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Amr Al‐Haidari. The network helps show where Amr Al‐Haidari may publish in the future.

Co-authorship network of co-authors of Amr Al‐Haidari

This figure shows the co-authorship network connecting the top 25 collaborators of Amr Al‐Haidari. A scholar is included among the top collaborators of Amr Al‐Haidari based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Amr Al‐Haidari. Amr Al‐Haidari is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
#WorkIndexed citations
1
T cell receptor (TCR) signaling in health and diseasebreakdown →
332
2 30
3 14
4 64
5 66
6 45
7 29

About Amr Al‐Haidari

Amr Al‐Haidari is a scholar working on Cancer Research, Gastroenterology and Immunology and Allergy, having authored 7 papers that have together received 580 indexed citations. Recurring topics across this work include MicroRNA in disease regulation (3 papers), Circular RNAs in diseases (2 papers) and Cancer-related molecular mechanisms research (2 papers). The work is most often cited by research in Immunology (249 citations), Cancer Research (153 citations) and Oncology (171 citations). Amr Al‐Haidari has collaborated with scholars based in Sweden and China. Frequent co-authors include Julhash U. Kazi, Jianmin Sun, Kinjal Shah, Henrik Thorlacius, Ingvar Syk, Raed Madhi and Milladur Rahman. Their work appears in journals such as Scientific Reports, Biochemical and Biophysical Research Communications and Frontiers in Immunology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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