Amjad Naami
Impact in
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- Cancer, Hypoxia, and Metabolism
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- Cell Adhesion Molecules Research
Papers in
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- Wnt/β-catenin signaling in development and cancer 2
- TGF-β signaling in diseases 1
- Protein Tyrosine Phosphatases 1
- Metabolism, Diabetes, and Cancer 1
- Surgery 3
- Co-authors
- Ruth Knüchel (5 shared papers)Edgar Dahl (4 shared papers)Arndt Hartmann (4 shared papers)Jürgen Veeck (3 shared papers)Nuran Bektas (3 shared papers)Glen Kristiansen (2 shared papers)Erik Noetzel (2 shared papers)Harald P. Kühl (1 shared paper)
- Journals
- Oncogene (2 papers)Urology (1 paper)Coronary Artery Disease (1 paper)Investigative Radiology (1 paper)European Journal of Pediatrics (1 paper)
- Partner nations
- GermanySwitzerlandUnited States
In The Last Decade
Amjad Naami
12 papers receiving 387 citations
Peers
Comparison fields: 5 of 65
- Cancer Research 61
- Immunology and Allergy 23
- Immunology 79
- Oncology 78
- Pulmonary and Respiratory Medicine 83
Countries citing papers authored by Amjad Naami
This map shows the geographic impact of Amjad Naami's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Amjad Naami with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Amjad Naami more than expected).
Fields of papers citing papers by Amjad Naami
This network shows the impact of papers produced by Amjad Naami. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Amjad Naami. The network helps show where Amjad Naami may publish in the future.
Co-authors
The 25 scholars most cited alongside Amjad Naami, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2008 | 94 | |
| 2 | 2007 | 77 | |
| 3 | 2008 | 47 | |
| 4 | 2010 | 46 | |
| 5 | 2006 | 37 | |
| 6 | 2001 | 24 | |
| 7 | 2013 | 23 | |
| 8 | 2006 | 17 | |
| 9 | 2013 | 13 | |
| 10 | 2008 | 7 | |
| 11 | 2008 | 6 | |
| 12 | 2001 | 1 |
About Amjad Naami
Amjad Naami is a scholar working on Molecular Biology, Surgery, Epidemiology, Radiology, Nuclear Medicine and Imaging and Immunology and Allergy, having authored 12 papers that have together received 392 indexed citations. Recurring topics across this work include Wnt/β-catenin signaling in development and cancer (2 papers), Cell Adhesion Molecules Research (2 papers), Cytomegalovirus and herpesvirus research (2 papers), Cytokine Signaling Pathways and Interactions (1 paper), TGF-β signaling in diseases (1 paper), Protein Tyrosine Phosphatases (1 paper), Galectins and Cancer Biology (1 paper) and Metabolism, Diabetes, and Cancer (1 paper). The work is most often cited by research in Cancer Research (61 citations), Immunology and Allergy (23 citations), Immunology (79 citations), Oncology (78 citations) and Pulmonary and Respiratory Medicine (83 citations). Amjad Naami has collaborated with scholars based in Germany, Switzerland and United States. Frequent co-authors include Ruth Knüchel, Edgar Dahl, Arndt Hartmann, Jürgen Veeck, Nuran Bektas, Glen Kristiansen, Erik Noetzel, Harald P. Kühl, Elmar Spuentrup and Thorsten Reffelmann. Their work appears in journals such as Oncogene, Urology, Coronary Artery Disease, Investigative Radiology and European Journal of Pediatrics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.