Amel Ben Chehida

416 total citations
42 papers, 274 citations indexed

About

Amel Ben Chehida is a scholar working on Molecular Biology, Physiology and Clinical Biochemistry. According to data from OpenAlex, Amel Ben Chehida has authored 42 papers receiving a total of 274 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 14 papers in Physiology and 11 papers in Clinical Biochemistry. Recurrent topics in Amel Ben Chehida's work include Metabolism and Genetic Disorders (11 papers), Lysosomal Storage Disorders Research (11 papers) and Glycogen Storage Diseases and Myoclonus (8 papers). Amel Ben Chehida is often cited by papers focused on Metabolism and Genetic Disorders (11 papers), Lysosomal Storage Disorders Research (11 papers) and Glycogen Storage Diseases and Myoclonus (8 papers). Amel Ben Chehida collaborates with scholars based in Tunisia, France and Hong Kong. Amel Ben Chehida's co-authors include Néji Tebib, Hatem Azzouz, Hadhami Ben Turkia, S. Barsaoui, Khadija Boussetta, Azza Sammoud, Jalel Chemli, Kamel Monastiri, Sonia Abdelhak and M Chaâbouni and has published in prestigious journals such as Frontiers in Immunology, Archives of Disease in Childhood and Neuroscience Research.

In The Last Decade

Amel Ben Chehida

37 papers receiving 273 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Amel Ben Chehida Tunisia 9 84 70 53 51 47 42 274
Hatem Azzouz Tunisia 11 102 1.2× 97 1.4× 53 1.0× 61 1.2× 50 1.1× 39 392
Ivana Capuano Italy 11 142 1.7× 73 1.0× 10 0.2× 46 0.9× 41 0.9× 29 297
Satoko Takei Japan 10 147 1.8× 172 2.5× 48 0.9× 23 0.5× 15 0.3× 21 430
Liting Zhang China 12 83 1.0× 81 1.2× 19 0.4× 11 0.2× 17 0.4× 47 326
Mirella Collura Italy 12 85 1.0× 89 1.3× 16 0.3× 5 0.1× 132 2.8× 19 603
Ian N. Scobie United Kingdom 13 100 1.2× 97 1.4× 4 0.1× 37 0.7× 43 0.9× 28 437
Stefanie Reiermann Germany 8 178 2.1× 39 0.6× 4 0.1× 63 1.2× 11 0.2× 11 343
S. Quartuccio Italy 8 46 0.5× 62 0.9× 12 0.2× 60 1.2× 10 0.2× 12 282
Ildikó Ungvári Hungary 10 86 1.0× 87 1.2× 9 0.2× 8 0.2× 12 0.3× 11 301
M Cembrzýńska-Nowak Poland 7 205 2.4× 53 0.8× 25 0.5× 20 0.4× 17 0.4× 25 362

Countries citing papers authored by Amel Ben Chehida

Since Specialization
Citations

This map shows the geographic impact of Amel Ben Chehida's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Amel Ben Chehida with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Amel Ben Chehida more than expected).

Fields of papers citing papers by Amel Ben Chehida

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Amel Ben Chehida. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Amel Ben Chehida. The network helps show where Amel Ben Chehida may publish in the future.

Co-authorship network of co-authors of Amel Ben Chehida

This figure shows the co-authorship network connecting the top 25 collaborators of Amel Ben Chehida. A scholar is included among the top collaborators of Amel Ben Chehida based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Amel Ben Chehida. Amel Ben Chehida is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Mekki, Najla, Amel Ben Chehida, Daniel Leung, et al.. (2025). Case Report: Dual molecular diagnosis of gain-of-function STAT1 mutation and regulatory STAT3 variant in a patient with a hyper-IgE-like phenotype. Frontiers in Immunology. 16. 1646761–1646761.
2.
3.
Chehida, Amel Ben, et al.. (2023). A Case of Infantile Cataract and Neonatal Hypoglycemia. Journal of Current Ophthalmology. 35(3). 291–293. 1 indexed citations
4.
Chehida, Amel Ben, Ichraf Kraoua, Haïfa Sanhaji, et al.. (2020). Non-ketotic hyperglycinaemia: a frequent, but poorly diagnosed and managed genetic disorder in Tunisia. Archives of Disease in Childhood. 106(3). 311–311. 1 indexed citations
5.
Chehida, Amel Ben, Lilia Kraoua, Heather Etchevers, et al.. (2019). A severe clinical phenotype of Noonan syndrome with neonatal hypertrophic cardiomyopathy in the second case worldwide withRAF1S259Y neomutation. Genetics Research. 101. e6–e6. 3 indexed citations
6.
Chehida, Amel Ben, et al.. (2019). Self-Reported Anxiety, Depression and Coping in Parents of Children with Phenylketonuria. Journal of Developmental and Physical Disabilities. 31(6). 753–776. 2 indexed citations
7.
Chabchoub, I., Amel Ben Chehida, Lobna Ayadi, et al.. (2019). Hemophagocytic Lymphohistiocytosis: A Rare Complication of an Ultrarare Lysosomal Storage Disease. Journal of Pediatric Hematology/Oncology. 42(4). 310–312. 4 indexed citations
8.
Chehida, Amel Ben, et al.. (2018). Patient-management Problem (PMP) for paediatrics learning: Value and students perceptions.. PubMed. 96(1). 1–5.
9.
Chehida, Amel Ben, Khadija Boussetta, S. Barsaoui, et al.. (2017). Full title: peripheral venous catheter complications in children: predisposing factors in a multicenter prospective cohort study. BMC Pediatrics. 17(1). 208–208. 66 indexed citations
10.
Ben‐Mustapha, Imen, Meriem Ben‐Ali, Karen Rouault, et al.. (2016). A Founder Effect of c.257 + 2T > C Mutation in NCF2 Gene Underlies Severe Chronic Granulomatous Disease in Eleven Patients. Journal of Clinical Immunology. 36(6). 547–554. 13 indexed citations
11.
Aloulou, Hajer, Syrine Gallas, I. Chabchoub, et al.. (2016). Clinical and Genetic Characterization of 26 Tunisian Patients with Allgrove Syndrome. Archives of Medical Research. 47(2). 105–110. 19 indexed citations
12.
13.
Chehida, Amel Ben, et al.. (2015). A novel homozygous missense mutation in the insulin receptor gene results in an atypical presentation of Rabson-Mendenhall syndrome. European Journal of Medical Genetics. 59(1). 16–19. 10 indexed citations
14.
Chehida, Amel Ben, Hatem Azzouz, Sonia Abdelhak, et al.. (2015). Complications rénales dans la glycogénose de type 1 : quelles implications pratiques ?. Néphrologie & Thérapeutique. 11(4). 240–245. 1 indexed citations
15.
Turkia, Hadhami Ben, et al.. (2015). Les maladies héréditaires du métabolisme en Tunisie: défis, acquis, espoirs. Archives de Pédiatrie. 22(5). 3–4. 3 indexed citations
16.
Azzouz, Hatem, François Petit, Mariem Ben Khelifa, et al.. (2013). Molecular and biochemical characterization of a novel intronic single point mutation in a Tunisian family with glycogen storage disease type III. Molecular Biology Reports. 40(7). 4197–4202. 7 indexed citations
17.
Turkia, Hadhami Ben, et al.. (2013). Molecular Characterization of X-Linked Adrenoleukodystrophy in a Tunisian Family: Identification of a Novel Missense Mutation in the <b><i>ABCD1</i></b> Gene. Neurodegenerative Diseases. 12(4). 207–211. 4 indexed citations
18.
Turkia, Hadhami Ben, et al.. (2009). Phenotypic continuum of type 2 Gaucher's disease: an intermediate phenotype between perinatal-lethal and classic type 2 Gaucher's disease. Journal of Perinatology. 29(2). 170–172. 12 indexed citations
19.
Turkia, Hadhami Ben, et al.. (2009). Cholestase néonatale révélatrice d’un phénotype intermédiaire d’une maladie de Gaucher type 2. Archives de Pédiatrie. 16(3). 255–257. 6 indexed citations
20.
Turkia, Hadhami Ben, et al.. (2008). Lipodystrophie congénitale généralisée de type 1 avec atteinte neurologique. Archives de Pédiatrie. 16(1). 27–31. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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