Amanda B. Parris

558 total citations
22 papers, 409 citations indexed

About

Amanda B. Parris is a scholar working on Oncology, Molecular Biology and Cancer Research. According to data from OpenAlex, Amanda B. Parris has authored 22 papers receiving a total of 409 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Oncology, 13 papers in Molecular Biology and 7 papers in Cancer Research. Recurrent topics in Amanda B. Parris's work include Cancer-related Molecular Pathways (9 papers), Metabolism, Diabetes, and Cancer (6 papers) and Cancer, Hypoxia, and Metabolism (4 papers). Amanda B. Parris is often cited by papers focused on Cancer-related Molecular Pathways (9 papers), Metabolism, Diabetes, and Cancer (6 papers) and Cancer, Hypoxia, and Metabolism (4 papers). Amanda B. Parris collaborates with scholars based in United States and China. Amanda B. Parris's co-authors include Xiaohe Yang, Qingxia Zhao, Ming Zhao, Zhikun Ma, Ying Xing, Xiaoshan Feng, Ming Zhao, Nipun Saini, Yujie Shi and Lingfei Kong and has published in prestigious journals such as PLoS ONE, Cancer Research and Scientific Reports.

In The Last Decade

Amanda B. Parris

20 papers receiving 407 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Amanda B. Parris United States 15 254 126 96 51 45 22 409
Monserrat Olea‐Flores Mexico 11 272 1.1× 148 1.2× 158 1.6× 47 0.9× 38 0.8× 21 555
Renhua Gai China 10 303 1.2× 122 1.0× 99 1.0× 71 1.4× 28 0.6× 14 490
Shotaro Yamano Japan 13 266 1.0× 117 0.9× 110 1.1× 86 1.7× 31 0.7× 44 539
Phillip M. Shelton United States 6 294 1.2× 91 0.7× 70 0.7× 24 0.5× 32 0.7× 8 411
Lokman Varışlı Türkiye 11 261 1.0× 94 0.7× 100 1.0× 88 1.7× 17 0.4× 28 410
Hongqiang Sheng China 8 266 1.0× 119 0.9× 146 1.5× 41 0.8× 62 1.4× 14 450
Nobuyuki Kuribayashi Japan 15 300 1.2× 163 1.3× 96 1.0× 33 0.6× 36 0.8× 37 545
Xiuling Li China 14 228 0.9× 108 0.9× 145 1.5× 82 1.6× 18 0.4× 29 468
Débora Kristina Alves-Fernandes Brazil 10 311 1.2× 113 0.9× 95 1.0× 25 0.5× 33 0.7× 14 544

Countries citing papers authored by Amanda B. Parris

Since Specialization
Citations

This map shows the geographic impact of Amanda B. Parris's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Amanda B. Parris with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Amanda B. Parris more than expected).

Fields of papers citing papers by Amanda B. Parris

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Amanda B. Parris. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Amanda B. Parris. The network helps show where Amanda B. Parris may publish in the future.

Co-authorship network of co-authors of Amanda B. Parris

This figure shows the co-authorship network connecting the top 25 collaborators of Amanda B. Parris. A scholar is included among the top collaborators of Amanda B. Parris based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Amanda B. Parris. Amanda B. Parris is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Parris, Amanda B., et al.. (2022). Metformin-induced downregulation of c-Met is a determinant of sensitivity in MDA-MB-468 breast cancer cells. Biochemical and Biophysical Research Communications. 613. 100–106. 3 indexed citations
2.
3.
Ma, Zhikun, Amanda B. Parris, Meghan B. Davis, et al.. (2020). In Utero Exposure to Bisphenol a Promotes Mammary Tumor Risk in MMTV-Erbb2 Transgenic Mice Through the Induction of ER-erbB2 Crosstalk. International Journal of Molecular Sciences. 21(9). 3095–3095. 11 indexed citations
4.
Shi, Yujie, Zhikun Ma, Qiong Cheng, et al.. (2020). FGFR1 overexpression renders breast cancer cells resistant to metformin through activation of IRS1/ERK signaling. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1868(1). 118877–118877. 15 indexed citations
5.
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Ma, Zhikun, Young Kim, Xiaoshan Feng, et al.. (2018). DMBA promotes ErbB2‑mediated carcinogenesis via ErbB2 and estrogen receptor pathway activation and genomic instability. Oncology Reports. 40(3). 1632–1640. 11 indexed citations
7.
8.
Saini, Nipun, Amanda B. Parris, Zhikun Ma, et al.. (2018). Ganetespib targets multiple levels of the receptor tyrosine kinase signaling cascade and preferentially inhibits ErbB2-overexpressing breast cancer cells. Scientific Reports. 8(1). 6829–6829. 20 indexed citations
9.
Parris, Amanda B.. (2018). The logic of imagination: a Spinozan critique of imaginative freedom. The Institutional Repository at DePaul University (DePaul University).
10.
Zhao, Ming, et al.. (2017). p53 pathway determines the cellular response to alcohol-induced DNA damage in MCF-7 breast cancer cells. PLoS ONE. 12(4). e0175121–e0175121. 20 indexed citations
11.
Zhao, Qingxia, Amanda B. Parris, Ming Zhao, et al.. (2017). FGFR inhibitor, AZD4547, impedes the stemness of mammary epithelial cells in the premalignant tissues of MMTV-ErbB2 transgenic mice. Scientific Reports. 7(1). 11306–11306. 14 indexed citations
12.
Ma, Zhikun, et al.. (2017). Short-term early exposure to lapatinib confers lifelong protection from mammary tumor development in MMTV-erbB-2 transgenic mice. Journal of Experimental & Clinical Cancer Research. 36(1). 6–6. 8 indexed citations
13.
Parris, Amanda B., et al.. (2017). Buformin inhibits the stemness of erbB-2-overexpressing breast cancer cells and premalignant mammary tissues of MMTV-erbB-2 transgenic mice. Journal of Experimental & Clinical Cancer Research. 36(1). 28–28. 17 indexed citations
14.
Zhao, Ming, Amanda B. Parris, Qingxia Zhao, et al.. (2017). Activation of cancerous inhibitor of PP2A (CIP2A) contributes to lapatinib resistance through induction of CIP2A-Akt feedback loop in ErbB2-positive breast cancer cells. Oncotarget. 8(35). 58847–58864. 15 indexed citations
15.
Saini, Nipun, et al.. (2017). Ganetespib induces G2/M cell cycle arrest and apoptosis in gastric cancer cells through targeting of receptor tyrosine kinase signaling. International Journal of Oncology. 51(3). 967–974. 29 indexed citations
16.
17.
Askew, Emily B., Suxia Bai, Amanda B. Parris, John T. Minges, & Elizabeth M. Wilson. (2016). Androgen receptor regulation by histone methyltransferase Suppressor of variegation 3-9 homolog 2 and Melanoma antigen-A11. Molecular and Cellular Endocrinology. 443. 42–51. 16 indexed citations
18.
Zhao, Qingxia, et al.. (2016). Genistein targets the cancerous inhibitor of PP2A to induce growth inhibition and apoptosis in breast cancer cells. International Journal of Oncology. 49(3). 1203–1210. 44 indexed citations
19.
Zhao, Ming, et al.. (2016). Alcohol promotes migration and invasion of triple‐negative breast cancer cells through activation of p38 MAPK and JNK. Molecular Carcinogenesis. 56(3). 849–862. 37 indexed citations
20.
Zhao, Ming, et al.. (2015). p53 is required for metformin-induced growth inhibition, senescence and apoptosis in breast cancer cells. Biochemical and Biophysical Research Communications. 464(4). 1267–1274. 55 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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