Amado A. Quintar

731 total citations
30 papers, 571 citations indexed

About

Amado A. Quintar is a scholar working on Immunology, Pulmonary and Respiratory Medicine and Molecular Biology. According to data from OpenAlex, Amado A. Quintar has authored 30 papers receiving a total of 571 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Immunology, 14 papers in Pulmonary and Respiratory Medicine and 7 papers in Molecular Biology. Recurrent topics in Amado A. Quintar's work include Immune Response and Inflammation (9 papers), Urinary Bladder and Prostate Research (6 papers) and Prostate Cancer Treatment and Research (6 papers). Amado A. Quintar is often cited by papers focused on Immune Response and Inflammation (9 papers), Urinary Bladder and Prostate Research (6 papers) and Prostate Cancer Treatment and Research (6 papers). Amado A. Quintar collaborates with scholars based in Argentina, United States and Brazil. Amado A. Quintar's co-authors include Cristina A. Maldonado, Juan P. Nicola, Felix Roth, Agustı́n Aoki, Ana Lucía De Paul, Klaus Ley, Mariana Maccioni, Ana M. Masini‐Repiso, Alex Márki and Zbigniew Mikulski and has published in prestigious journals such as The Journal of Experimental Medicine, PLoS ONE and Circulation Research.

In The Last Decade

Amado A. Quintar

29 papers receiving 562 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Amado A. Quintar Argentina 14 263 148 146 68 66 30 571
S. Goralnick United States 9 164 0.6× 71 0.5× 83 0.6× 42 0.6× 129 2.0× 13 563
Colm Costigan Ireland 17 161 0.6× 93 0.6× 266 1.8× 331 4.9× 25 0.4× 34 800
Erna Sziksz Hungary 14 167 0.6× 65 0.4× 207 1.4× 48 0.7× 6 0.1× 29 665
Janet Cornelius United States 10 173 0.7× 158 1.1× 147 1.0× 165 2.4× 16 0.2× 15 762
Hyun J. Bang United States 12 54 0.2× 148 1.0× 261 1.8× 62 0.9× 17 0.3× 15 924
Şeyma Özkanlı Türkiye 13 55 0.2× 61 0.4× 90 0.6× 18 0.3× 24 0.4× 72 453
Yong‐Wei Fu China 8 79 0.3× 39 0.3× 122 0.8× 30 0.4× 12 0.2× 12 386
Johannes Eberle Austria 8 36 0.1× 125 0.8× 155 1.1× 105 1.5× 187 2.8× 16 726
Marìa Albertina Romaniuk Argentina 11 215 0.8× 35 0.2× 248 1.7× 10 0.1× 16 0.2× 17 601
J. N. Bulmer United Kingdom 13 175 0.7× 30 0.2× 102 0.7× 239 3.5× 24 0.4× 20 787

Countries citing papers authored by Amado A. Quintar

Since Specialization
Citations

This map shows the geographic impact of Amado A. Quintar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Amado A. Quintar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Amado A. Quintar more than expected).

Fields of papers citing papers by Amado A. Quintar

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Amado A. Quintar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Amado A. Quintar. The network helps show where Amado A. Quintar may publish in the future.

Co-authorship network of co-authors of Amado A. Quintar

This figure shows the co-authorship network connecting the top 25 collaborators of Amado A. Quintar. A scholar is included among the top collaborators of Amado A. Quintar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Amado A. Quintar. Amado A. Quintar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Oliveira‐Paula, Gustavo H., Sophia Liu, Graziele C. Ferreira, et al.. (2024). The β-catenin C terminus links Wnt and sphingosine-1-phosphate signaling pathways to promote vascular remodeling and atherosclerosis. Science Advances. 10(11). eadg9278–eadg9278. 10 indexed citations
3.
Berg, Gabriela, et al.. (2024). Extracellular Vesicles Contribute to Oxidized LDL-Induced Stromal Cell Proliferation in Benign Prostatic Hyperplasia. Biology. 13(10). 827–827. 2 indexed citations
4.
Quintar, Amado A., et al.. (2021). The pathological growth of the prostate gland in atherogenic contexts. Experimental Gerontology. 148. 111304–111304. 10 indexed citations
5.
Silva, Juliete A.F., et al.. (2020). Prostate immunology: A challenging puzzle. Journal of Reproductive Immunology. 142. 103190–103190. 7 indexed citations
6.
Sosa, Liliana del Valle, et al.. (2020). Testosterone-loaded GM1 micelles targeted to the intracellular androgen receptor for the specific induction of genomic androgen signaling. International Journal of Pharmaceutics. 591. 119985–119985. 4 indexed citations
7.
Quintar, Amado A., Elmer A. Fernández, César Seigi Fuziwara, et al.. (2018). Functional Toll-like Receptor 4 Overexpression in Papillary Thyroid Cancer by MAPK/ERK–Induced ETS1 Transcriptional Activity. Molecular Cancer Research. 16(5). 833–845. 29 indexed citations
8.
Nicola, Juan P., et al.. (2018). Inefficient N2-Like Neutrophils Are Promoted by Androgens During Infection. Frontiers in Immunology. 9. 1980–1980. 50 indexed citations
9.
Sundblad, Victoria, Amado A. Quintar, Luciano Gastón Morosi, et al.. (2018). Galectins in Intestinal Inflammation: Galectin-1 Expression Delineates Response to Treatment in Celiac Disease Patients. Frontiers in Immunology. 9. 379–379. 32 indexed citations
10.
Quintar, Amado A., et al.. (2017). The mongolian gerbil (Meriones unguiculatus) as a model for inflammation‐promoted prostate carcinogenesis. Cell Biology International. 41(11). 1234–1238. 11 indexed citations
11.
García, Jorge, et al.. (2017). Evidence of eosinophil extracellular trap cell death in COPD: does it represent the trigger that switches on the disease?. International Journal of COPD. Volume 12. 885–896. 40 indexed citations
12.
Quintar, Amado A., et al.. (2016). Testosterone Rescues the De‐Differentiation of Smooth Muscle Cells Through Serum Response Factor/Myocardin. Journal of Cellular Physiology. 232(10). 2806–2817. 9 indexed citations
13.
Acosta, Patricio L., et al.. (2014). Protective phenotypes of club cells and alveolar macrophages are favored as part of endotoxin-mediated prevention of asthma. Experimental Biology and Medicine. 240(7). 904–916. 8 indexed citations
14.
Quintar, Amado A., et al.. (2013). Testosterone abrogates TLR4 activation in prostate smooth muscle cells contributing to the preservation of a differentiated phenotype. Journal of Cellular Physiology. 228(7). 1551–1560. 20 indexed citations
15.
Quintar, Amado A., et al.. (2012). Androgen depletion augments antibacterial prostate host defences in rats. International Journal of Andrology. 35(6). 845–859. 17 indexed citations
16.
Quintar, Amado A., et al.. (2010). Dedifferentiation of prostate smooth muscle cells in response to bacterial LPS. The Prostate. 71(10). 1097–1107. 27 indexed citations
17.
Quintar, Amado A., et al.. (2010). Acute inflammation promotes early cellular stimulation of the epithelial and stromal compartments of the rat prostate. The Prostate. 70(11). 1153–1165. 21 indexed citations
18.
Gatti, Gerardo, Amado A. Quintar, Virginia Andreani, et al.. (2009). Expression of Toll‐like receptor 4 in the prostate gland and its association with the severity of prostate cancer. The Prostate. 69(13). 1387–1397. 54 indexed citations
19.
Quintar, Amado A., et al.. (2007). Increased expression of uteroglobin associated with tubal inflammation and ectopic pregnancy. Fertility and Sterility. 89(6). 1613–1617. 7 indexed citations
20.
Roth, Felix, et al.. (2006). Budesonide effects on Clara cell under normal and allergic inflammatory condition. Histochemistry and Cell Biology. 127(1). 55–68. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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