Alo Ray

1.0k total citations
28 papers, 829 citations indexed

About

Alo Ray is a scholar working on Molecular Biology, Physiology and Oncology. According to data from OpenAlex, Alo Ray has authored 28 papers receiving a total of 829 indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 7 papers in Physiology and 6 papers in Oncology. Recurrent topics in Alo Ray's work include DNA Repair Mechanisms (15 papers), Genomics and Chromatin Dynamics (7 papers) and Telomeres, Telomerase, and Senescence (6 papers). Alo Ray is often cited by papers focused on DNA Repair Mechanisms (15 papers), Genomics and Chromatin Dynamics (7 papers) and Telomeres, Telomerase, and Senescence (6 papers). Alo Ray collaborates with scholars based in United States, India and Saudi Arabia. Alo Ray's co-authors include Kurt W. Runge, Altaf A. Wani, Gulzar Wani, Aruna Battu, Ronald E. Hector, Kathleen L. Berkner, Keisha Milum, Qun Zhao, Qianzheng Zhu and Qi‐En Wang and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Alo Ray

28 papers receiving 818 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Alo Ray United States 15 720 235 124 91 80 28 829
Serge Gravel Canada 10 1.0k 1.4× 331 1.4× 176 1.4× 87 1.0× 162 2.0× 14 1.1k
Danna K. Morris United States 11 829 1.2× 577 2.5× 191 1.5× 189 2.1× 45 0.6× 15 1.0k
Sascha Feuerhahn Switzerland 9 770 1.1× 411 1.7× 92 0.7× 66 0.7× 155 1.9× 9 921
F Brad Johnson United States 9 674 0.9× 339 1.4× 51 0.4× 79 0.9× 120 1.5× 12 785
İlgen Mender United States 12 508 0.7× 512 2.2× 105 0.8× 81 0.9× 62 0.8× 15 796
Shaheen Kabir United States 9 555 0.8× 460 2.0× 49 0.4× 78 0.9× 38 0.5× 10 710
Fermı́n A. Goytisolo Spain 8 769 1.1× 671 2.9× 116 0.9× 138 1.5× 62 0.8× 8 994
Angela Chan United States 13 626 0.9× 185 0.8× 83 0.7× 67 0.7× 68 0.8× 16 709
Paul S. Bradshaw Canada 7 321 0.4× 178 0.8× 63 0.5× 35 0.4× 50 0.6× 9 414
Jaewon Min United States 11 626 0.9× 343 1.5× 80 0.6× 60 0.7× 30 0.4× 17 760

Countries citing papers authored by Alo Ray

Since Specialization
Citations

This map shows the geographic impact of Alo Ray's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alo Ray with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alo Ray more than expected).

Fields of papers citing papers by Alo Ray

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alo Ray. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alo Ray. The network helps show where Alo Ray may publish in the future.

Co-authorship network of co-authors of Alo Ray

This figure shows the co-authorship network connecting the top 25 collaborators of Alo Ray. A scholar is included among the top collaborators of Alo Ray based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alo Ray. Alo Ray is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ray, Alo & Mateusz Opyrchal. (2025). Targeting PARP1: A Promising Approach for Next-Generation Poly (ADP-ribose) Polymerase Inhibitors. Current Breast Cancer Reports. 17(1). 22–22. 2 indexed citations
2.
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Zhang, Haitao, et al.. (2018). Rif1 phosphorylation site analysis in telomere length regulation and the response to damaged telomeres. DNA repair. 65. 26–33. 8 indexed citations
5.
Ray, Alo, et al.. (2016). ATR- and ATM-Mediated DNA Damage Response Is Dependent on Excision Repair Assembly during G1 but Not in S Phase of Cell Cycle. PLoS ONE. 11(7). e0159344–e0159344. 35 indexed citations
6.
Lu, Jingwei, Manjusri Das, Suman Kanji, et al.. (2014). Induction of ATM/ATR pathway combined with Vγ2Vδ2 T cells enhances cytotoxicity of ovarian cancer cells. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1842(7). 1071–1079. 8 indexed citations
7.
Ray, Alo, Keisha Milum, Aruna Battu, Gulzar Wani, & Altaf A. Wani. (2013). NER initiation factors, DDB2 and XPC, regulate UV radiation response by recruiting ATR and ATM kinases to DNA damage sites. DNA repair. 12(4). 273–283. 60 indexed citations
8.
Hector, Ronald E., et al.. (2012). Mec1p associates with functionally compromised telomeres. Chromosoma. 121(3). 277–290. 11 indexed citations
9.
Battu, Aruna, Alo Ray, & Altaf A. Wani. (2011). ASF1A and ATM regulate H3K56-mediated cell-cycle checkpoint recovery in response to UV irradiation. Nucleic Acids Research. 39(18). 7931–7945. 61 indexed citations
10.
Arab, Hany H., Gulzar Wani, Alo Ray, et al.. (2010). Dissociation of CAK from Core TFIIH Reveals a Functional Link between XP-G/CS and the TFIIH Disassembly State. PLoS ONE. 5(6). e11007–e11007. 26 indexed citations
11.
Zhao, Qun, Qi-En Wang, Alo Ray, et al.. (2009). Modulation of Nucleotide Excision Repair by Mammalian SWI/SNF Chromatin-remodeling Complex. Journal of Biological Chemistry. 284(44). 30424–30432. 78 indexed citations
12.
Ray, Alo, Gulzar Wani, Qun Zhao, et al.. (2009). Human SNF5/INI1, a Component of the Human SWI/SNF Chromatin Remodeling Complex, Promotes Nucleotide Excision Repair by Influencing ATM Recruitment and Downstream H2AX Phosphorylation. Molecular and Cellular Biology. 29(23). 6206–6219. 57 indexed citations
13.
Elmahdy, Mohamed, Qianzheng Zhu, Bassant M. Barakat, et al.. (2008). Thymoquinone upregulates PTEN expression and induces apoptosis in doxorubicin-resistant human breast cancer cells. Cancer Research. 68. 630–630. 2 indexed citations
14.
Zhao, Qun, Bassant M. Barakat, Song Qin, et al.. (2008). The p38 Mitogen-activated Protein Kinase Augments Nucleotide Excision Repair by Mediating DDB2 Degradation and Chromatin Relaxation. Journal of Biological Chemistry. 283(47). 32553–32561. 43 indexed citations
15.
Zhu, Qianzheng, Gulzar Wani, Hany H. Arab, et al.. (2008). Chromatin restoration following nucleotide excision repair involves the incorporation of ubiquitinated H2A at damaged genomic sites. DNA repair. 8(2). 262–273. 47 indexed citations
16.
Hector, Ronald E., et al.. (2007). Tel1p Preferentially Associates with Short Telomeres to Stimulate Their Elongation. Molecular Cell. 27(5). 851–858. 105 indexed citations
17.
Ray, Alo, et al.. (2003). Sir3p phosphorylation by the Slt2p pathway effects redistribution of silencing function and shortened lifespan. Nature Genetics. 33(4). 522–526. 45 indexed citations
18.
Phan, Dillon, Geoffrey W. Birrell, Sophie Bonnal, et al.. (2001). Purification and Characterization of Human Cell-Cell Adhesion Molecule 1 (C-CAM1) Expressed in Insect Cells. Protein Expression and Purification. 21(2). 343–351. 5 indexed citations
19.
Ray, Alo & Pratima Sinha. (1995). The mcm2-1 mutation of yeast causes DNA damage with a RAD9 requirement for repair. Current Genetics. 27(2). 95–101. 4 indexed citations
20.
Ray, Alo, et al.. (1994). A 61-kb ring chromosome shows an ARS-dependent increase in its mitotic stability in the mcm2 mutant of yeast. Current Genetics. 26(5-6). 403–409. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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