Allison J. Bartuski

676 total citations
7 papers, 581 citations indexed

About

Allison J. Bartuski is a scholar working on Cancer Research, Oncology and Genetics. According to data from OpenAlex, Allison J. Bartuski has authored 7 papers receiving a total of 581 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Cancer Research, 3 papers in Oncology and 3 papers in Genetics. Recurrent topics in Allison J. Bartuski's work include Protease and Inhibitor Mechanisms (4 papers), Peptidase Inhibition and Analysis (3 papers) and Animal Genetics and Reproduction (2 papers). Allison J. Bartuski is often cited by papers focused on Protease and Inhibitor Mechanisms (4 papers), Peptidase Inhibition and Analysis (3 papers) and Animal Genetics and Reproduction (2 papers). Allison J. Bartuski collaborates with scholars based in United States and Australia. Allison J. Bartuski's co-authors include Charles Schick, Gary A. Silverman, Yoshiro Kamachi, Sule Çataltepe, Philip A. Pemberton, Norman M. Schechter, Dieter Brömme, Guo‐Ping Shi, Harold A. Chapman and Yoshiki Uemura and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Biochemistry.

In The Last Decade

Allison J. Bartuski

7 papers receiving 573 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Allison J. Bartuski United States	6	265	244	111	107	98	7	581
Lucy East United Kingdom	7	172 0.6×	374 1.5×	54 0.5×	97 0.9×	310 3.2×	7	835
Michéle Brillard‐Bourdet France	16	172 0.6×	220 0.9×	23 0.2×	119 1.1×	138 1.4×	24	666
Ding Ma China	17	166 0.6×	557 2.3×	80 0.7×	198 1.9×	151 1.5×	33	915
Stéphanie Salesse France	16	118 0.4×	326 1.3×	47 0.4×	121 1.1×	88 0.9×	25	695
Phillip A. Morton United States	15	207 0.8×	276 1.1×	71 0.6×	142 1.3×	599 6.1×	19	999
Bruno Dumont France	15	103 0.4×	378 1.5×	113 1.0×	134 1.3×	91 0.9×	20	834
Teruaki Oku Japan	15	65 0.2×	215 0.9×	106 1.0×	80 0.7×	139 1.4×	40	556
Sofia Z. Raptis United States	8	286 1.1×	314 1.3×	54 0.5×	234 2.2×	504 5.1×	10	1.0k
Daniel Skup Canada	15	269 1.0×	405 1.7×	30 0.3×	259 2.4×	177 1.8×	33	708
Juha Hakulinen Finland	15	208 0.8×	432 1.8×	35 0.3×	161 1.5×	522 5.3×	17	1.0k

Countries citing papers authored by Allison J. Bartuski

Since Specialization

Citations

This map shows the geographic impact of Allison J. Bartuski's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Allison J. Bartuski with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Allison J. Bartuski more than expected).

Fields of papers citing papers by Allison J. Bartuski

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Allison J. Bartuski. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Allison J. Bartuski. The network helps show where Allison J. Bartuski may publish in the future.

Co-authorship network of co-authors of Allison J. Bartuski

This figure shows the co-authorship network connecting the top 25 collaborators of Allison J. Bartuski. A scholar is included among the top collaborators of Allison J. Bartuski based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Allison J. Bartuski. Allison J. Bartuski is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Schick, Charles, Philip A. Pemberton, Guo‐Ping Shi, et al.. (1998). Cross-Class Inhibition of the Cysteine Proteinases Cathepsins K, L, and S by the Serpin Squamous Cell Carcinoma Antigen 1: A Kinetic Analysis. Biochemistry. 37(15). 5258–5266. 234 indexed citations

2.

Bartuski, Allison J., Yoshiro Kamachi, Charles Schick, et al.. (1998). A Murine Ortholog of the Human Serpin SCCA2 Maps to Chromosome 1 and Inhibits Chymotrypsin-like Serine Proteinases. Genomics. 54(2). 297–306. 11 indexed citations

3.

Silverman, Gary A., et al.. (1998). SCCA1 and SCCA2 Are Proteinase Inhibitors That Map to the Serpin Cluster at 18q21.3. Tumor Biology. 19(6). 480–487. 33 indexed citations

4.

Schick, Charles, Dieter Brömme, Allison J. Bartuski, et al.. (1998). The reactive site loop of the serpin SCCA1 is essential for cysteine proteinase inhibition. Proceedings of the National Academy of Sciences. 95(23). 13465–13470. 68 indexed citations

5.

Bartuski, Allison J., et al.. (1997). Cytoplasmic Antiproteinase 2 (PI8) and Bomapin (PI10) Map to the Serpin Cluster at 18q21.3. Genomics. 43(3). 321–328. 44 indexed citations

6.

Schick, Charles, Yoshiro Kamachi, Allison J. Bartuski, et al.. (1997). Squamous Cell Carcinoma Antigen 2 Is a Novel Serpin That Inhibits the Chymotrypsin-like Proteinases Cathepsin G and Mast Cell Chymase. Journal of Biological Chemistry. 272(3). 1849–1855. 188 indexed citations

7.

Trask, Barbara J., Ger van den Engh, Allison J. Bartuski, et al.. (1995). Analysis of randomly amplified flow-sorted chromosomes using the polymerase chain reaction. Genomics. 26(2). 364–371. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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