Alison G. Compton

4.7k total citations
42 papers, 2.5k citations indexed

About

Alison G. Compton is a scholar working on Molecular Biology, Clinical Biochemistry and Genetics. According to data from OpenAlex, Alison G. Compton has authored 42 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 41 papers in Molecular Biology, 25 papers in Clinical Biochemistry and 10 papers in Genetics. Recurrent topics in Alison G. Compton's work include Mitochondrial Function and Pathology (33 papers), Metabolism and Genetic Disorders (25 papers) and ATP Synthase and ATPases Research (13 papers). Alison G. Compton is often cited by papers focused on Mitochondrial Function and Pathology (33 papers), Metabolism and Genetic Disorders (25 papers) and ATP Synthase and ATPases Research (13 papers). Alison G. Compton collaborates with scholars based in Australia, United States and United Kingdom. Alison G. Compton's co-authors include David R. Thorburn, Elena J. Tucker, Nicole J. Lake, Shamima Rahman, John Christodoulou, Ann E. Frazier, Sarah E. Calvo, Michael T. Ryan, Vamsi K. Mootha and Damien L. Bruno and has published in prestigious journals such as Nature, Journal of Biological Chemistry and Nature Genetics.

In The Last Decade

Alison G. Compton

41 papers receiving 2.5k citations

Peers

Alison G. Compton
Eleonora Lamantea Italy
Véronique Paquis‐Flucklinger France
Henna Tyynismaa Finland
Elena J. Tucker Australia
John P. Grady United Kingdom
Caterina Garone Italy
Sandra R. Bacman United States
Nadine Gigarel France
Emmanuelle Sarzi France
Helen Griffin United Kingdom
Eleonora Lamantea Italy
Alison G. Compton
Citations per year, relative to Alison G. Compton Alison G. Compton (= 1×) peers Eleonora Lamantea

Countries citing papers authored by Alison G. Compton

Since Specialization
Citations

This map shows the geographic impact of Alison G. Compton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alison G. Compton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alison G. Compton more than expected).

Fields of papers citing papers by Alison G. Compton

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alison G. Compton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alison G. Compton. The network helps show where Alison G. Compton may publish in the future.

Co-authorship network of co-authors of Alison G. Compton

This figure shows the co-authorship network connecting the top 25 collaborators of Alison G. Compton. A scholar is included among the top collaborators of Alison G. Compton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alison G. Compton. Alison G. Compton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Bergen, Nicole J. Van, et al.. (2025). Therapies for Mitochondrial Disease: Past, Present, and Future. Journal of Inherited Metabolic Disease. 48(4). e70065–e70065. 1 indexed citations
2.
Barnett, Christopher F., Mary‐Louise Freckmann, Matthew F. Hunter, et al.. (2024). Critically unwell infants and children with mitochondrial disorders diagnosed by ultrarapid genomic sequencing. Genetics in Medicine. 27(1). 101293–101293. 1 indexed citations
3.
Lake, Nicole J., Kaiyue Ma, Wei Liu, et al.. (2024). Quantifying constraint in the human mitochondrial genome. Nature. 635(8038). 390–397. 18 indexed citations
4.
Hock, Daniella H., Nicole J. Lake, Sarah E. Calvo, et al.. (2023). Multi-omics identifies large mitoribosomal subunit instability caused by pathogenic MRPL39 variants as a cause of pediatric onset mitochondrial disease. Human Molecular Genetics. 32(15). 2441–2454. 13 indexed citations
5.
Akesson, Lauren, Rocío Rius, Natasha J. Brown, et al.. (2022). Distinct diagnostic trajectories in NBAS‐associated acute liver failure highlights the need for timely functional studies. JIMD Reports. 63(3). 240–249. 2 indexed citations
6.
Rius, Rocío, Neal K. Bennett, Kaustuv Bhattacharya, et al.. (2022). Biallelic pathogenic variants in COX11 are associated with an infantile‐onset mitochondrial encephalopathy. Human Mutation. 43(12). 1970–1978. 9 indexed citations
7.
Riley, Lisa G., Joëlle Rudinger‐Thirion, Magali Frugier, et al.. (2020). The expandingLARS2phenotypic spectrum: HLASA, Perrault syndrome with leukodystrophy, and mitochondrial myopathy. Human Mutation. 41(8). 1425–1434. 18 indexed citations
8.
Riley, Lisa G., Mark J. Cowley, Velimir Gayevskiy, et al.. (2020). The diagnostic utility of genome sequencing in a pediatric cohort with suspected mitochondrial disease. Genetics in Medicine. 22(7). 1254–1261. 45 indexed citations
9.
Lake, Nicole J., Luke E. Formosa, David A. Stroud, et al.. (2019). A patient with homozygous nonsense variants in two Leigh syndrome disease genes: Distinguishing a dual diagnosis from a hypomorphic protein‐truncating variant. Human Mutation. 40(7). 893–898. 7 indexed citations
10.
Frazier, Ann E., David R. Thorburn, & Alison G. Compton. (2017). Mitochondrial energy generation disorders: genes, mechanisms, and clues to pathology. Journal of Biological Chemistry. 294(14). 5386–5395. 176 indexed citations
11.
Lim, Sze Chern, Justine E. Marum, Elena J. Tucker, et al.. (2013). Mutations in LYRM4, encoding iron–sulfur cluster biogenesis factor ISD11, cause deficiency of multiple respiratory chain complexes. Human Molecular Genetics. 22(22). 4460–4473. 81 indexed citations
12.
Tucker, Elena J., Bas F.J. Wanschers, Radek Szklarczyk, et al.. (2013). Mutations in the UQCC1-Interacting Protein, UQCC2, Cause Human Complex III Deficiency Associated with Perturbed Cytochrome b Protein Expression. PLoS Genetics. 9(12). e1004034–e1004034. 85 indexed citations
13.
Tucker, Elena J., Steven G. Hershman, Caroline Köhrer, et al.. (2011). Mutations in MTFMT Underlie a Human Disorder of Formylation Causing Impaired Mitochondrial Translation. Cell Metabolism. 14(3). 428–434. 125 indexed citations
14.
Compton, Alison G., Christopher Troedson, Meredith Wilson, et al.. (2010). Application of oligonucleotide array CGH in the detection of a large intragenic deletion in POLG associated with Alpers Syndrome. Mitochondrion. 11(1). 104–107. 11 indexed citations
15.
Tucker, Elena J., Alison G. Compton, & David R. Thorburn. (2010). Recent Advances in the Genetics of Mitochondrial Encephalopathies. Current Neurology and Neuroscience Reports. 10(4). 277–285. 41 indexed citations
16.
Riley, Lisa G., Sandra T. Cooper, Peter F. Hickey, et al.. (2010). Mutation of the Mitochondrial Tyrosyl-tRNA Synthetase Gene, YARS2, Causes Myopathy, Lactic Acidosis, and Sideroblastic Anemia—MLASA Syndrome. The American Journal of Human Genetics. 87(1). 52–59. 173 indexed citations
17.
Calvo, Sarah E., Elena J. Tucker, Alison G. Compton, et al.. (2010). High-throughput, pooled sequencing identifies mutations in NUBPL and FOXRED1 in human complex I deficiency. Nature Genetics. 42(10). 851–858. 279 indexed citations
18.
Au, Carol G., Tanya L. Butler, Jonathan R. Egan, et al.. (2008). Changes in skeletal muscle expression of AQP1 and AQP4 in dystrophinopathy and dysferlinopathy patients. Acta Neuropathologica. 116(3). 235–246. 24 indexed citations
19.
Compton, Alison G., Douglas E. Albrecht, Jane T. Seto, et al.. (2008). Mutations in Contactin-1, a Neural Adhesion and Neuromuscular Junction Protein, Cause a Familial Form of Lethal Congenital Myopathy. The American Journal of Human Genetics. 83(6). 714–724. 61 indexed citations
20.
Compton, Alison G., et al.. (2005). The Syntrophin-Dystrobrevin Subcomplex in Human Neuromuscular Disorders. Journal of Neuropathology & Experimental Neurology. 64(4). 350–361. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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