Alida Spalloni

1.7k total citations
34 papers, 1.4k citations indexed

About

Alida Spalloni is a scholar working on Neurology, Genetics and Cellular and Molecular Neuroscience. According to data from OpenAlex, Alida Spalloni has authored 34 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Neurology, 9 papers in Genetics and 7 papers in Cellular and Molecular Neuroscience. Recurrent topics in Alida Spalloni's work include Amyotrophic Lateral Sclerosis Research (26 papers), Parkinson's Disease Mechanisms and Treatments (10 papers) and Neurogenetic and Muscular Disorders Research (9 papers). Alida Spalloni is often cited by papers focused on Amyotrophic Lateral Sclerosis Research (26 papers), Parkinson's Disease Mechanisms and Treatments (10 papers) and Neurogenetic and Muscular Disorders Research (9 papers). Alida Spalloni collaborates with scholars based in Italy, United States and France. Alida Spalloni's co-authors include Patrizia Longone, Francesco Fornai, Michela Ferrucci, Antonio Paparelli, Cristina Zona, Nicola Biagio Mercuri, Nicola Modugno, Luigi Murri, Gloria Lazzeri and Olga Kastsiuchenka and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Annals of Neurology and Scientific Reports.

In The Last Decade

Alida Spalloni

33 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Alida Spalloni Italy 19 800 404 371 298 204 34 1.4k
Carol Milligan United States 19 652 0.8× 517 1.3× 388 1.0× 248 0.8× 182 0.9× 33 1.2k
P. Anthony Akkari Australia 18 658 0.8× 1.1k 2.8× 527 1.4× 228 0.8× 183 0.9× 46 1.8k
Jo A. Oostveen United States 18 737 0.9× 378 0.9× 227 0.6× 505 1.7× 451 2.2× 31 1.6k
Peter P. De Deyn Belgium 21 952 1.2× 480 1.2× 305 0.8× 163 0.5× 618 3.0× 36 1.7k
Masakuni Amari Japan 15 431 0.5× 275 0.7× 145 0.4× 123 0.4× 503 2.5× 45 1.0k
Autumn Klein United States 13 757 0.9× 925 2.3× 203 0.5× 813 2.7× 316 1.5× 22 1.9k
Christine Sato Canada 28 1.0k 1.3× 936 2.3× 304 0.8× 417 1.4× 943 4.6× 66 2.3k
Pol Andrés‐Benito Spain 22 345 0.4× 467 1.2× 111 0.3× 271 0.9× 520 2.5× 62 1.2k
Sara Sáez-Atiénzar United States 16 503 0.6× 482 1.2× 93 0.3× 174 0.6× 356 1.7× 24 1.2k
Tomokazu Obi Japan 16 740 0.9× 538 1.3× 159 0.4× 267 0.9× 325 1.6× 38 1.2k

Countries citing papers authored by Alida Spalloni

Since Specialization
Citations

This map shows the geographic impact of Alida Spalloni's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alida Spalloni with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alida Spalloni more than expected).

Fields of papers citing papers by Alida Spalloni

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alida Spalloni. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alida Spalloni. The network helps show where Alida Spalloni may publish in the future.

Co-authorship network of co-authors of Alida Spalloni

This figure shows the co-authorship network connecting the top 25 collaborators of Alida Spalloni. A scholar is included among the top collaborators of Alida Spalloni based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alida Spalloni. Alida Spalloni is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Salvatori, Illari, Valentina Nesci, Alida Spalloni, et al.. (2024). Trimetazidine Improves Mitochondrial Dysfunction in SOD1G93A Cellular Models of Amyotrophic Lateral Sclerosis through Autophagy Activation. International Journal of Molecular Sciences. 25(6). 3251–3251. 5 indexed citations
2.
Salvatori, I., Marco De Bardi, Juliette Gimenez, et al.. (2024). Hydrogen Sulfide Modulates Astrocytic Toxicity in Mouse Spinal Cord Cultures: Implications for Amyotrophic Lateral Sclerosis. Antioxidants. 13(10). 1241–1241.
3.
Gimenez, Juliette, et al.. (2023). TDP-43 Epigenetic Facets and Their Neurodegenerative Implications. International Journal of Molecular Sciences. 24(18). 13807–13807. 5 indexed citations
4.
Spalloni, Alida, Silvia Caioli, Elisa Bonomi, et al.. (2021). Cerebrospinal fluid from frontotemporal dementia patients is toxic to neurons. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1867(6). 166122–166122. 3 indexed citations
5.
Spalloni, Alida, Viviana Greco, Victor Corasolla Carregari, et al.. (2019). Impact of Pharmacological Inhibition of Hydrogen Sulphide Production in the SOD1G93A-ALS Mouse Model. International Journal of Molecular Sciences. 20(10). 2550–2550. 15 indexed citations
6.
Greco, Viviana, Patrizia Longone, Alida Spalloni, Luisa Pieroni, & Andrea Urbani. (2019). Crosstalk Between Oxidative Stress and Mitochondrial Damage: Focus on Amyotrophic Lateral Sclerosis. Advances in experimental medicine and biology. 1158. 71–82. 26 indexed citations
7.
Picardi, Gennaro, Alida Spalloni, Amanda Generosi, et al.. (2018). Tissue degeneration in ALS affected spinal cord evaluated by Raman spectroscopy. Scientific Reports. 8(1). 13110–13110. 10 indexed citations
8.
Spalloni, Alida & Patrizia Longone. (2015). Cognitive impairment in amyotrophic lateral sclerosis, clues from the SOD1 mouse. Neuroscience & Biobehavioral Reviews. 60. 12–25. 15 indexed citations
9.
D’Antoni, Simona, Michela Spatuzza, Antonio Berretta, et al.. (2014). Endothelin-1 is over-expressed in amyotrophic lateral sclerosis and induces motor neuron cell death. Neurobiology of Disease. 65. 160–171. 25 indexed citations
10.
Spalloni, Alida, et al.. (2012). Role of the N-methyl-d-aspartate receptors complex in amyotrophic lateral sclerosis. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1832(2). 312–322. 56 indexed citations
11.
D’Aguanno, Simona, Maria Teresa Cencioni, Luisa Pieroni, et al.. (2011). Protein repertoire impact of Ubiquitin–Proteasome System impairment: Insight into the protective role of beta-estradiol. Journal of Proteomics. 75(4). 1440–1453. 13 indexed citations
12.
Spalloni, Alida, Fulvio Florenzano, Ruth E. Westenbroek, et al.. (2011). Increased expression of the beta3 subunit of voltage-gated Na+ channels in the spinal cord of the SOD1G93A mouse. Molecular and Cellular Neuroscience. 47(2). 108–118. 19 indexed citations
13.
14.
Frazzini, Valério, et al.. (2010). Zinc pre-treatment enhances NMDAR-mediated excitotoxicity in cultured cortical neurons from SOD1G93A mouse, a model of amyotrophic lateral sclerosis. Neuropharmacology. 60(7-8). 1200–1208. 20 indexed citations
15.
Ferrucci, Michela, Alida Spalloni, Federica Fulceri, et al.. (2009). A systematic study of brainstem motor nuclei in a mouse model of ALS, the effects of lithium. Neurobiology of Disease. 37(2). 370–383. 73 indexed citations
16.
Fornai, Francesco, Patrizia Longone, Olga Kastsiuchenka, et al.. (2008). Lithium delays progression of amyotrophic lateral sclerosis. Proceedings of the National Academy of Sciences. 105(6). 2052–2057. 427 indexed citations
17.
Sgobio, Carmelo, Antonio Trabalza, Alida Spalloni, et al.. (2007). Abnormal medial prefrontal cortex connectivity and defective fear extinction in the presymptomatic G93A SOD1 mouse model of ALS. Genes Brain & Behavior. 7(4). 427–434. 33 indexed citations
18.
Spalloni, Alida, Tiziana Pascucci, Federica Albo, et al.. (2004). Altered vulnerability to kainate excitotoxicity of transgenic-Cu/Zn SOD1 neurones. Neuroreport. 15(16). 2477–2480. 12 indexed citations
19.
Spalloni, Alida, Federica Albo, Francesca Ferrari, et al.. (2004). Cu/Zn-superoxide dismutase (GLY93→ALA) mutation alters AMPA receptor subunit expression and function and potentiates kainate-mediated toxicity in motor neurons in culture. Neurobiology of Disease. 15(2). 340–350. 67 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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