Alicia S. Chung

2.9k total citations · 2 hit papers
16 papers, 2.3k citations indexed

About

Alicia S. Chung is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Alicia S. Chung has authored 16 papers receiving a total of 2.3k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 7 papers in Oncology and 5 papers in Immunology. Recurrent topics in Alicia S. Chung's work include Angiogenesis and VEGF in Cancer (6 papers), Cytokine Signaling Pathways and Interactions (3 papers) and interferon and immune responses (2 papers). Alicia S. Chung is often cited by papers focused on Angiogenesis and VEGF in Cancer (6 papers), Cytokine Signaling Pathways and Interactions (3 papers) and interferon and immune responses (2 papers). Alicia S. Chung collaborates with scholars based in United States and China. Alicia S. Chung's co-authors include Napoleone Ferrara, J. Jack Lee, Guanglei Zhuang, Y. Eugene Chin, Xiumin Wu, Franklin Peale, Hai Ngu, Y. Gloria Meng, Zhaoshi Jiang and Wenjun Ouyang and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Medicine.

In The Last Decade

Alicia S. Chung

16 papers receiving 2.3k citations

Hit Papers

Targeting the tumour vasculature: insights from physiolog... 2010 2026 2015 2020 2010 2011 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Alicia S. Chung United States 11 1.4k 798 521 517 213 16 2.3k
Janelle Simon United States 11 993 0.7× 727 0.9× 504 1.0× 379 0.7× 145 0.7× 13 2.0k
Naoharu Takano Japan 23 1.3k 0.9× 595 0.7× 348 0.7× 978 1.9× 164 0.8× 45 2.5k
Frédéric Mazurier France 29 1.6k 1.1× 498 0.6× 437 0.8× 457 0.9× 299 1.4× 67 2.8k
Xiaochun Xu United States 19 1.3k 0.9× 505 0.6× 366 0.7× 507 1.0× 341 1.6× 36 2.0k
Tim Hon Man Chan United States 31 1.8k 1.2× 647 0.8× 648 1.2× 489 0.9× 197 0.9× 66 2.7k
Simona Ronzoni Italy 17 2.2k 1.5× 1.3k 1.7× 328 0.6× 659 1.3× 168 0.8× 29 3.2k
Joseph Kwong Hong Kong 32 1.6k 1.2× 803 1.0× 476 0.9× 757 1.5× 170 0.8× 56 2.8k
Gianluca Bossi Italy 30 1.4k 1.0× 988 1.2× 254 0.5× 547 1.1× 144 0.7× 53 2.2k
Dayana B. Rivadeneira United States 17 1.1k 0.7× 957 1.2× 802 1.5× 714 1.4× 141 0.7× 26 2.3k
Xinhui Wang United States 21 828 0.6× 917 1.1× 536 1.0× 459 0.9× 101 0.5× 55 1.9k

Countries citing papers authored by Alicia S. Chung

Since Specialization
Citations

This map shows the geographic impact of Alicia S. Chung's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alicia S. Chung with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alicia S. Chung more than expected).

Fields of papers citing papers by Alicia S. Chung

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alicia S. Chung. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alicia S. Chung. The network helps show where Alicia S. Chung may publish in the future.

Co-authorship network of co-authors of Alicia S. Chung

This figure shows the co-authorship network connecting the top 25 collaborators of Alicia S. Chung. A scholar is included among the top collaborators of Alicia S. Chung based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alicia S. Chung. Alicia S. Chung is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Zhuang, Guanglei, Kebing Yu, Zhaoshi Jiang, et al.. (2013). Phosphoproteomic Analysis Implicates the mTORC2-FoxO1 Axis in VEGF Signaling and Feedback Activation of Receptor Tyrosine Kinases. Science Signaling. 6(271). ra25–ra25. 60 indexed citations
2.
Chung, Alicia S., Xiumin Wu, Guanglei Zhuang, et al.. (2013). An interleukin-17–mediated paracrine network promotes tumor resistance to anti-angiogenic therapy. Nature Medicine. 19(9). 1114–1123. 349 indexed citations
3.
Phan, Vernon T., Xiumin Wu, Jason Cheng, et al.. (2013). Oncogenic RAS pathway activation promotes resistance to anti-VEGF therapy through G-CSF–induced neutrophil recruitment. Proceedings of the National Academy of Sciences. 110(15). 6079–6084. 93 indexed citations
4.
Chung, Alicia S., Marcin Kowanetz, Xiumin Wu, et al.. (2012). Differential drug class‐specific metastatic effects following treatment with a panel of angiogenesis inhibitors. The Journal of Pathology. 227(4). 404–416. 71 indexed citations
5.
Chung, Alicia S. & Napoleone Ferrara. (2011). Developmental and Pathological Angiogenesis. Annual Review of Cell and Developmental Biology. 27(1). 563–584. 599 indexed citations breakdown →
6.
Guan, Yingjie, Zhe Zhang, Chen Yu, et al.. (2011). Phospho-SXXE/D Motif Mediated TNF Receptor 1–TRADD Death Domain Complex Formation for T Cell Activation and Migration. The Journal of Immunology. 187(3). 1289–1297. 9 indexed citations
7.
Chung, Alicia S., J. Jack Lee, & Napoleone Ferrara. (2010). Targeting the tumour vasculature: insights from physiological angiogenesis. Nature reviews. Cancer. 10(7). 505–514. 620 indexed citations breakdown →
8.
Chung, Alicia S.. (2009). Lipid sorting and clustering. Nature Reviews Molecular Cell Biology. 10(7). 442–443. 1 indexed citations
9.
Chung, Alicia S. & Y. Eugene Chin. (2009). Antibody Array Platform to Monitor Protein Tyrosine Phosphorylation in Mammalian Cells. Methods in molecular biology. 527. 247–255. 4 indexed citations
10.
Chung, Alicia S.. (2009). An 'IDOL' regulator of blood cholesterol levels. Nature Reviews Molecular Cell Biology. 10(8). 507–507. 1 indexed citations
11.
Chung, Alicia S.. (2009). How different progenitor cells are called from the bone marrow. 1 indexed citations
12.
Chung, Alicia S., Yingjie Guan, Zhenglong Yuan, Jorge E. Albina, & Y. Eugene Chin. (2005). Ankyrin Repeat and SOCS Box 3 (ASB3) Mediates Ubiquitination and Degradation of Tumor Necrosis Factor Receptor II. Molecular and Cellular Biology. 25(11). 4716–4726. 59 indexed citations
13.
Ivanov, Stanimir S., Alicia S. Chung, Zhenglong Yuan, et al.. (2004). Antibodies Immobilized as Arrays to Profile Protein Post-translational Modifications in Mammalian Cells. Molecular & Cellular Proteomics. 3(8). 788–795. 47 indexed citations
14.
Xia, Ling, Lijuan Wang, Alicia S. Chung, et al.. (2002). Identification of Both Positive and Negative Domains within the Epidermal Growth Factor Receptor COOH-terminal Region for Signal Transducer and Activator of Transcription (STAT) Activation. Journal of Biological Chemistry. 277(34). 30716–30723. 97 indexed citations
15.
Wu, Tong, Y. Kate Hong, Xudong Wang, et al.. (2002). SHP-2 Is a Dual-specificity Phosphatase Involved in Stat1 Dephosphorylation at Both Tyrosine and Serine Residues in Nuclei. Journal of Biological Chemistry. 277(49). 47572–47580. 125 indexed citations
16.
Ries, Stefan, Christian Brandts, Alicia S. Chung, et al.. (2000). Loss of p14ARF in tumor cells facilitates replication of the adenovirus mutant dl1520 (ONYX-015). Nature Medicine. 6(10). 1128–1133. 159 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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