Alexander Hermani

1.0k total citations
8 papers, 847 citations indexed

About

Alexander Hermani is a scholar working on Molecular Biology, Genetics and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Alexander Hermani has authored 8 papers receiving a total of 847 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 4 papers in Genetics and 3 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Alexander Hermani's work include Estrogen and related hormone effects (4 papers), Metabolism, Diabetes, and Cancer (3 papers) and Growth Hormone and Insulin-like Growth Factors (3 papers). Alexander Hermani is often cited by papers focused on Estrogen and related hormone effects (4 papers), Metabolism, Diabetes, and Cancer (3 papers) and Growth Hormone and Insulin-like Growth Factors (3 papers). Alexander Hermani collaborates with scholars based in Germany, Israel and Canada. Alexander Hermani's co-authors include Doris Mayer, Senad Medunjanin, Barbara De Servi, Jean Grisouard, Philippe A. Tessier, Rainer Grobholz, Lutz Trojan, Peter Angel, Ashish Shukla and Jochen Heß and has published in prestigious journals such as Journal of Biological Chemistry, Oncogene and Clinical Cancer Research.

In The Last Decade

Alexander Hermani

8 papers receiving 830 citations

Peers

Alexander Hermani

MB

EDM

CR

IMMUN

ONCOL

Senad Medunjanin Germany

Angelo Boccia Italy

Dianne J. Beveridge Australia

Prince George United States

Debi K. Swertfeger United States

Essa Y. Baitei Saudi Arabia

Markus Haak Germany

Kotaro Azuma Japan

Qing Shao China

Tatjana Albrektsen Denmark

Senad Medunjanin Germany

Alexander Hermani

675 ×1.1

MB

98 ×0.5

EDM

195 ×1.0

CR

236 ×1.3

IMMUN

171 ×1.1

ONCOL

Citations per year, relative to Alexander Hermani Alexander Hermani (= 1×) peers Senad Medunjanin

Countries citing papers authored by Alexander Hermani

Since Specialization

Citations

This map shows the geographic impact of Alexander Hermani's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alexander Hermani with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alexander Hermani more than expected).

Fields of papers citing papers by Alexander Hermani

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alexander Hermani. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alexander Hermani. The network helps show where Alexander Hermani may publish in the future.

Co-authorship network of co-authors of Alexander Hermani

This figure shows the co-authorship network connecting the top 25 collaborators of Alexander Hermani. A scholar is included among the top collaborators of Alexander Hermani based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alexander Hermani. Alexander Hermani is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Hermani, Alexander, Ashish Shukla, Senad Medunjanin, Haim Werner, & Doris Mayer. (2013). Insulin-like growth factor binding protein-4 and -5 modulate ligand-dependent estrogen receptor-α activation in breast cancer cells in an IGF-independent manner. Cellular Signalling. 25(6). 1395–1402. 28 indexed citations

2.

Shukla, Ashish, Jean Grisouard, Volker Ehemann, et al.. (2009). Analysis of signaling pathways related to cell proliferation stimulated by insulin analogs in human mammary epithelial cell lines. Endocrine Related Cancer. 16(2). 429–441. 101 indexed citations

3.

Grisouard, Jean, et al.. (2007). Glycogen Synthase Kinase-3 Protects Estrogen Receptor α from Proteasomal Degradation and Is Required for Full Transcriptional Activity of the Receptor. Molecular Endocrinology. 21(10). 2427–2439. 54 indexed citations

4.

Hermani, Alexander, Jochen Heß, Barbara De Servi, et al.. (2005). Calcium-Binding Proteins S100A8 and S100A9 as Novel Diagnostic Markers in Human Prostate Cancer. Clinical Cancer Research. 11(14). 5146–5152. 210 indexed citations

5.

Hermani, Alexander, et al.. (2005). S100A8 and S100A9 activate MAP kinase and NF-κB signaling pathways and trigger translocation of RAGE in human prostate cancer cells. Experimental Cell Research. 312(2). 184–197. 218 indexed citations

6.

Medunjanin, Senad, Alexander Hermani, Barbara De Servi, et al.. (2005). Glycogen Synthase Kinase-3 Interacts with and Phosphorylates Estrogen Receptor α and Is Involved in the Regulation of Receptor Activity. Journal of Biological Chemistry. 280(38). 33006–33014. 122 indexed citations

7.

Servi, Barbara De, Alexander Hermani, Senad Medunjanin, & Doris Mayer. (2005). Impact of PKCδ on estrogen receptor localization and activity in breast cancer cells. Oncogene. 24(31). 4946–4955. 34 indexed citations

8.

Liao, Yongde, Ulrich Abel, Rainer Grobholz, et al.. (2005). Up-regulation of insulin-like growth factor axis components in human primary prostate cancer correlates with tumor grade. Human Pathology. 36(11). 1186–1196. 80 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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