Alessandra Gnani

1.2k total citations
16 papers, 788 citations indexed

About

Alessandra Gnani is a scholar working on Hematology, Genetics and Rheumatology. According to data from OpenAlex, Alessandra Gnani has authored 16 papers receiving a total of 788 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Hematology, 14 papers in Genetics and 7 papers in Rheumatology. Recurrent topics in Alessandra Gnani's work include Chronic Myeloid Leukemia Treatments (15 papers), Chronic Lymphocytic Leukemia Research (14 papers) and Eosinophilic Disorders and Syndromes (7 papers). Alessandra Gnani is often cited by papers focused on Chronic Myeloid Leukemia Treatments (15 papers), Chronic Lymphocytic Leukemia Research (14 papers) and Eosinophilic Disorders and Syndromes (7 papers). Alessandra Gnani collaborates with scholars based in Italy and United Kingdom. Alessandra Gnani's co-authors include Giovanni Martinelli, Simona Soverini, Sabrina Colarossi, Fausto Castagnetti, Ilaria Iacobucci, Michele Baccarani, Gianantonio Rosti, Marilina Amabile, Angela Poerio and Elisabetta Abruzzese and has published in prestigious journals such as Journal of Clinical Oncology, Blood and The Lancet Oncology.

In The Last Decade

Alessandra Gnani

16 papers receiving 757 citations

Peers

Alessandra Gnani
Comparison fields: 5 of 32
  • Hematology 727
  • Genetics 597
  • Rheumatology 405
  • Public Health, Environmental and Occupational Health 157
  • Pathology and Forensic Medicine 72
Angela Poerio Italy
Nadine Besson United States
Simona Luatti Italy
Lan Beppu United States
Jarrad M. Goyne Australia
Angela Volkert United States
Dushyant Verma United States
Sara Dellasala United States
DK Hossfeld Germany
Wendy T Parker Australia
Angela Poerio Italy View profile →
Citations per field, relative to Alessandra Gnani
Alessandra Gnani · 1×
Citations per year, relative to Alessandra Gnani
Alessandra Gnani · 1×

Countries citing papers authored by Alessandra Gnani

Since Specialization
Citations

This map shows the geographic impact of Alessandra Gnani's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alessandra Gnani with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alessandra Gnani more than expected).

Fields of papers citing papers by Alessandra Gnani

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alessandra Gnani. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alessandra Gnani. The network helps show where Alessandra Gnani may publish in the future.

Co-authorship network of co-authors of Alessandra Gnani

This figure shows the co-authorship network connecting the top 25 collaborators of Alessandra Gnani. A scholar is included among the top collaborators of Alessandra Gnani based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alessandra Gnani. Alessandra Gnani is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
# Work Indexed citations
1
Low-level Bcr–Abl mutations are very rare in chronic myeloid leukemia patients who are in major molecular response on first-line nilotinib
2011 ·Leukemia Research ·Simona Soverini, Alessandra Gnani, Caterina De Benedittis, Fausto Castagnetti, Gabriele Gugliotta, Ilaria Iacobucci, Francesca Palandri, Gianantonio Rosti, Nicoletta Testoni, Simona Luatti, Giulia Marzocchi, Michele Baccarani, Giovanni Martinelli
4
2
Ultra-Deep Amplicon Sequencing Using Roche 454 Technology Allows High Sensitivity Bcr-Abl Kinase Domain Mutation Screening and Anticipates Emerging Mutations Leading to Resistance to Tyrosine Kinase Inhibitors in Philadelphia-Positive Leukemia Patients,
2011 ·Blood ·Simona Soverini, Caterina De Benedittis, Alessandra Gnani, Ilaria Iacobucci, Domenico Russo, Federica Cattina, Fausto Castagnetti, Gabriele Gugliotta, Cristina Papayannidis, Antonella Vitale, Loredana Elia, Robin Foà, Francesca Palandri, Annalisa Lonetti, Maria Teresa Bochicchio, Sandra Durante, Gianantonio Rosti, Michele Baccarani, Giovanni Martinelli
1
3
Philadelphia-positive acute lymphoblastic leukemia patients already harbor BCR-ABL kinase domain mutations at low levels at the time of diagnosis
2010 ·Haematologica ·Simona Soverini, Alessandro Vitale, Antonio Poerio, Alessandra Gnani, Sabrina Colarossi, Ilaria Iacobucci, Giuseppe Cimino, L Elia, Annalisa Lonetti, Marco Vignetti, Stefania Paolini, Giovanna Meloni, (unknown), Cristina Papayannidis, Marilina Amabile, Anna Guarini, Michele Baccarani, Giovanni Martinelli, R. Foà
64
4
EML4-ALK fusion gene in lung adenocarcinoma: A retrospective analysis of the outcome of cisplatin plus pemetrexed treated patients.
2010 ·Journal of Clinical Oncology ·Giuseppe Altavilla, (unknown), (unknown), Maria Teresa Rizzo, Giuseppe Galletti, (unknown), Alessandra Gnani, Vincenzo Pitini
12
5
Whole-Transcriptome Sequencing In Chronic Myeloid Leukemia Reveals Novel Gene Mutations That May Be Associated with Disease Pathogenesis and Progression
2010 ·Blood ·Simona Soverini, Angela Poerio, Alberto Ferrarini, Ilaria Iacobucci, Marco Sazzini, Joannah Score, (unknown), Luciano Xumerle, Sabrina Colarossi, Alessandra Gnani, Fausto Castagnetti, Francesca Palandri, Gabriele Gugliotta, Marilina Amabile, Gianantonio Rosti, Michele Baccarani, Nicholas C.P. Cross, Massimo Delledonne, Giovanni Martinelli
3
6
GENOME-WIDE SCREENING OF CHRONIC MYELOID LEUKEMIA PATIENTS BY SNP ARRAYS: ALTERATIONS ASSOCIATED WITH DISEASE PROGRESSION
2009 ·Haematologica ·Sabrina Colarossi, Alessandra Gnani, Simona Soverini, Fabio Castagnetti, G Marzocchi, Simona Luatti, Annalisa Astolfi, Serena Formica, Ilaria Iacobucci, Annalisa Lonetti, Francesca Palandri, Antonio Poerio, Marilina Amabile, (unknown), Andrea Pession, Nicoletta Testoni, Michele Baccarani, Giovanni Martinelli
2
7
High-Resolution Genome Wide Copy Number Alteration (CNA) and Loss of Heterozigosity (LOH) Analysis in Chronic Myeloid Leukemia (CML) Shows That High and Intermediate Sokal Risk Pts (Pts) Have Multiple Losses Targeting Genes Involved in DNA Repair.
2009 ·Blood ·Simona Soverini, Sabrina Colarossi, Alessandra Gnani, Annalisa Astolfi, Serena Formica, Fausto Castagnetti, Francesca Palandri, Gabriele Gugliotta, Angela Poerio, Ilaria Iacobucci, Marilina Amabile, Giulia Marzocchi, Simona Luatti, Carolina Terragna, Sandra Durante, Nicoletta Testoni, Gianantonio Rosti, Michele Baccarani, Giovanni Martinelli
2
8
Philadelphia-positive patients who already harbor imatinib-resistant Bcr-Abl kinase domain mutations have a higher likelihood of developing additional mutations associated with resistance to second- or third-line tyrosine kinase inhibitors
2009 ·Blood ·Simona Soverini, Alessandra Gnani, Sabrina Colarossi, Fausto Castagnetti, Elisabetta Abruzzese, Stefania Paolini, Serena Merante, Ester Orlandi, Silvia De Matteis, Antonella Gozzini, Ilaria Iacobucci, Francesca Palandri, Gabriele Gugliotta, Cristina Papayannidis, Angela Poerio, Marilina Amabile, Daniela Cilloni, Gianantonio Rosti, Michele Baccarani, Giovanni Martinelli
136
9
High-Resolution Molecular Allelokaryotyping of Chronic Myeloid Leukemia Patients in Blast Crisis by 6.0 SNP-Arrays Shows a High-Frequency of Uniparental Disomy and Focal Copy Number Alterations Affecting the Whole Sequence or Specific Exons of Oncogenes and Tumor Suppressor Genes.
2009 ·Blood ·Simona Soverini, Sabrina Colarossi, Alessandra Gnani, Fausto Castagnetti, Annalisa Astolfi, Serena Formica, Francesca Palandri, Ilaria Iacobucci, Gabriele Gugliotta, Angela Poerio, Marilina Amabile, Giulia Marzocchi, Nicoletta Testoni, Elisabetta Abruzzese, Gianantonio Rosti, Michele Baccarani, Giovanni Martinelli
1
10
Second-line treatment with dasatinib in patients resistant to imatinib can select novel inhibitor-specific BCR-ABL mutants in Ph+ ALL
2007 ·The Lancet Oncology ·Simona Soverini, Giovanni Martinelli, Sabrina Colarossi, Alessandra Gnani, Michela Rondoni, Fausto Castagnetti, Stefania Paolini, Gianantonio Rosti, Michele Baccarani
30
11
Resistance to dasatinib in Philadelphia-positive leukemia patients and the presence or the selection of mutations at residues 315 and 317 in the BCR-ABL kinase domain
2007 ·Haematologica ·Simona Soverini, Sabrina Colarossi, Alessandra Gnani, Fausto Castagnetti, G. Rosti, Costanza Bosi, Stefania Paolini, Michela Rondoni, Pier Paolo Piccaluga, Francesca Palandri, (unknown), G Marzocchi, Simona Luatti, Nicoletta Testoni, Ilaria Iacobucci, Daniela Cilloni, Giuseppe Saglio, Michele Baccarani, Giovanni Martinelli
133
12
In Early-Chronic Phase Chronic Myeloid Leukemia Patients Treated with Imatinib, Resistance Is Rarely Mediated by Abl Kinase Domain Mutations.
2007 ·Blood ·Simona Soverini, Alessandra Gnani, Sabrina Colarossi, Fausto Castagnetti, Elisabetta Abruzzese, Serena Merante, Massimo Breccia, Adele Capucci, Giorgina Specchia, Franca Radaelli, Francesca Palandri, Angela Poerio, Ilaria Iacobucci, Marilina Amabile, Gianantonio Rosti, Michele Baccarani, Giovanni Martinelli
2
13
Philadelphia Chromosome-Positive Leukemia Patients Who Harbor Imatinib-Resistant Mutations Have a Higher Likelihood of Developing Additional Mutations Associated with Resistance to Novel Tyrosine Kinase Inhibitors.
2007 ·Blood ·Simona Soverini, Alessandra Gnani, Sabrina Colarossi, Fausto Castagnetti, Francesca Palandri, (unknown), Stefania Paolini, Elisabetta Abruzzese, Serena Merante, Michela Rondoni, Ilaria Iacobucci, Angela Poerio, Gianantonio Rosti, Michele Baccarani, Giovanni Martinelli
7
14
Mutations at Residues 315 and 317 in the ABL Kinase Domain Are the Main Cause of Resistance to Dasatinib in Philadelphia-Positive (Ph+) Leukemia Patients (pts).
2006 ·Blood ·Simona Soverini, Giovanni Martinelli, Sabrina Colarossi, Alessandra Gnani, Fausto Castagnetti, Gianantonio Rosti, Costanza Bosi, Stefania Paolini, Michela Rondoni, Pier Paolo Piccaluga, Francesca Palandri, (unknown), Giulia Marzocchi, Simona Luatti, Elisabetta Abruzzese, Ilaria Iacobucci, Nicoletta Testoni, Michele Baccarani
6
15
Contribution of ABL Kinase Domain Mutations to Imatinib Resistance in Different Subsets of Philadelphia-Positive Patients: By the GIMEMA Working Party on Chronic Myeloid Leukemia
2006 ·Clinical Cancer Research ·Simona Soverini, Sabrina Colarossi, Alessandra Gnani, Gianantonio Rosti, Fausto Castagnetti, Angela Poerio, Ilaria Iacobucci, Marilina Amabile, Elisabetta Abruzzese, Ester Orlandi, Franca Radaelli, Fabrizio Ciccone, Mario Tiribelli, Roberto Di Lorenzo, Clementina Caracciolo, Barbara Izzo, Fabrizio Pane, Giuseppe Saglio, Michele Baccarani, Giovanni Martinelli, (unknown)
380
16
Frequency, Distribution and Prognostic Value of ABL Kinase Domain (KD) Mutations in Different Subsets of Philadelphia-Positive (Ph+) Patients (Pts) Resistant to Imatinib (IM) by the Gimema Working Party on CML.
2005 ·Blood ·Simona Soverini, Sabrina Colarossi, Alessandra Gnani, Gianantonio Rosti, Fausto Castagnetti, Angela Poerio, Ilaria Iacobucci, Marilina Amabile, (unknown), Elisabetta Abruzzese, Ester Orlandi, Franca Radaelli, Fabrizio Ciccone, Mario Tiribelli, Clementina Caracciolo, Fabrizio Pane, Giuseppe Saglio, Michele Baccarani, Giovanni Martinelli
5

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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