Alan R. Godwin
- Molecular Biology top 5%
- Pathology and Forensic Medicine top 0.5%
- Cancer Research top 2%
- Oncology top 5%
- Genetics top 5%
- Co-authors
- R. Michael LiskayMario R. CapecchiRoni J. BollagJ. Russell LipfordSean M. BakerMary Kay LescoeChristian BronnerMichael Kane
- Topics
- Cell Adhesion Molecules Research (9 papers)Connective tissue disorders research (8 papers)CRISPR and Genetic Engineering (7 papers)
- Partner nations
- United StatesUnited KingdomGermany
In The Last Decade
Alan R. Godwin
36 papers receiving 2.9k citations
Hit Papers
Peers
Comparison fields: 5 of 104
- Molecular Biology 1.5k
- Pathology and Forensic Medicine 1.4k
- Cancer Research 906
- Oncology 881
- Genetics 596
Countries citing papers authored by Alan R. Godwin
This map shows the geographic impact of Alan R. Godwin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alan R. Godwin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alan R. Godwin more than expected).
Fields of papers citing papers by Alan R. Godwin
This network shows the impact of papers produced by Alan R. Godwin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alan R. Godwin. The network helps show where Alan R. Godwin may publish in the future.
Co-authorship network of co-authors of Alan R. Godwin
This figure shows the co-authorship network connecting the top 25 collaborators of Alan R. Godwin. A scholar is included among the top collaborators of Alan R. Godwin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alan R. Godwin. Alan R. Godwin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 2 | |
| 3 | 2 | |
| 4 | 6 | |
| 5 | 88 | |
| 6 | 11 | |
| 7 | 29 | |
| 8 | 5 | |
| 9 | 22 | |
| 10 | 57 | |
| 11 | 36 | |
| 12 | 79 | |
| 13 | 93 | |
| 14 | 79 | |
| 15 | 29 | |
| 16 | 20 | |
| 17 | 203 | |
| 18 | Mutation in the DNA mismatch repair gene homologue hMLH 1 is associated with hereditary non-polyposis colon cancerbreakdown → | 1605 |
| 19 | 21 | |
| 20 | 11 |
About Alan R. Godwin
Alan R. Godwin is a scholar working on Immunology and Allergy, Developmental Biology and Cancer Research, having authored 37 papers that have together received 3.0k indexed citations. Recurring topics across this work include Cell Adhesion Molecules Research (9 papers), Connective tissue disorders research (8 papers) and CRISPR and Genetic Engineering (7 papers). The work is most often cited by research in Pathology and Forensic Medicine (1.4k citations), Cancer Research (906 citations) and Oncology (881 citations). Alan R. Godwin has collaborated with scholars based in United States, United Kingdom and Germany. Frequent co-authors include R. Michael Liskay, Mario R. Capecchi, Roni J. Bollag, J. Russell Lipford, Sean M. Baker, Mary Kay Lescoe, Christian Bronner, Michael Kane, Magnus Nordenskj�ld and Richard D. Kolodner. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.