Akira Moh

2.5k total citations
16 papers, 1.2k citations indexed

About

Akira Moh is a scholar working on Molecular Biology, Oncology and Epidemiology. According to data from OpenAlex, Akira Moh has authored 16 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Oncology and 4 papers in Epidemiology. Recurrent topics in Akira Moh's work include Cytokine Signaling Pathways and Interactions (4 papers), Liver Disease Diagnosis and Treatment (3 papers) and Acute Myeloid Leukemia Research (2 papers). Akira Moh is often cited by papers focused on Cytokine Signaling Pathways and Interactions (4 papers), Liver Disease Diagnosis and Treatment (3 papers) and Acute Myeloid Leukemia Research (2 papers). Akira Moh collaborates with scholars based in United States, Japan and Singapore. Akira Moh's co-authors include Xin-Yuan Fu, Bin Gao, Xin‐Yuan Fu, Yi Zhou, George Kunos, Ogyi Park, Partha Mukhopadhyay, Fouad Lafdil, Norio Horiguchi and Pál Pacher and has published in prestigious journals such as Journal of Biological Chemistry, Genes & Development and Blood.

In The Last Decade

Akira Moh

15 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Akira Moh United States 13 502 311 248 204 179 16 1.2k
Vladislav Temkin United States 17 650 1.3× 616 2.0× 242 1.0× 220 1.1× 89 0.5× 24 1.5k
Chunbin Zou United States 22 1.0k 2.0× 364 1.2× 168 0.7× 271 1.3× 54 0.3× 52 1.5k
Hanna Rokita Poland 24 738 1.5× 437 1.4× 316 1.3× 148 0.7× 60 0.3× 68 1.4k
Pontus Ørning United States 13 1.5k 2.9× 738 2.4× 113 0.5× 240 1.2× 58 0.3× 16 1.9k
Ni Liu China 16 545 1.1× 408 1.3× 262 1.1× 72 0.4× 46 0.3× 20 1.3k
Roshan J. Thapa United States 15 1.2k 2.4× 885 2.8× 262 1.1× 290 1.4× 56 0.3× 19 1.7k
Terunobu Saito Japan 14 595 1.2× 257 0.8× 62 0.3× 80 0.4× 92 0.5× 33 1.3k
Claire Piquet‐Pellorce France 25 731 1.5× 1.1k 3.4× 290 1.2× 431 2.1× 103 0.6× 58 2.1k
Ki‐Jung Yun South Korea 22 320 0.6× 199 0.6× 198 0.8× 103 0.5× 71 0.4× 47 1.1k
Masumi Shimizu Japan 19 368 0.7× 785 2.5× 368 1.5× 229 1.1× 90 0.5× 71 1.5k

Countries citing papers authored by Akira Moh

Since Specialization
Citations

This map shows the geographic impact of Akira Moh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Akira Moh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Akira Moh more than expected).

Fields of papers citing papers by Akira Moh

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Akira Moh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Akira Moh. The network helps show where Akira Moh may publish in the future.

Co-authorship network of co-authors of Akira Moh

This figure shows the co-authorship network connecting the top 25 collaborators of Akira Moh. A scholar is included among the top collaborators of Akira Moh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Akira Moh. Akira Moh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Sheng, Wanqiang, Fan Yang, Yi Zhou, et al.. (2014). STAT5 programs a distinct subset of GM-CSF-producing T helper cells that is essential for autoimmune neuroinflammation. Cell Research. 24(12). 1387–1402. 156 indexed citations
2.
Vinh, Dang, Jun Ueda, Daniel M. Messerschmidt, et al.. (2013). A genetic and developmental pathway from STAT3 to the OCT4–NANOG circuit is essential for maintenance of ICM lineages in vivo. Genes & Development. 27(12). 1378–1390. 142 indexed citations
3.
Mantel, Charlie, Steven Messina‐Graham, Akira Moh, et al.. (2012). Mouse hematopoietic cell–targeted STAT3 deletion: stem/progenitor cell defects, mitochondrial dysfunction, ROS overproduction, and a rapid aging–like phenotype. Blood. 120(13). 2589–2599. 107 indexed citations
4.
He, Ming, Zhiming Ouyang, Bryan Troxell, et al.. (2011). Cyclic di-GMP is Essential for the Survival of the Lyme Disease Spirochete in Ticks. PLoS Pathogens. 7(6). e1002133–e1002133. 106 indexed citations
5.
Li, Ping, Mark T. Nelson, Akira Moh, et al.. (2011). Osteoblast/osteocyte-specific inactivation of Stat3 decreases load-driven bone formation and accumulates reactive oxygen species. Bone. 49(3). 404–411. 80 indexed citations
6.
Miller, Andrew M., Hua Wang, Ogyi Park, et al.. (2010). Anti‐Inflammatory and Anti‐Apoptotic Roles of Endothelial Cell STAT3 in Alcoholic Liver Injury. Alcoholism Clinical and Experimental Research. 34(4). 719–725. 62 indexed citations
7.
Zhang, Wenjun, Rebecca J. Chan, Hanying Chen, et al.. (2009). Negative Regulation of Stat3 by Activating PTPN11 Mutants Contributes to the Pathogenesis of Noonan Syndrome and Juvenile Myelomonocytic Leukemia. Journal of Biological Chemistry. 284(33). 22353–22363. 49 indexed citations
8.
Fukuda, Seiji, Pratibha Singh, Akira Moh, et al.. (2009). Survivin mediates aberrant hematopoietic progenitor cell proliferation and acute leukemia in mice induced by internal tandem duplication of Flt3. Blood. 114(2). 394–403. 39 indexed citations
9.
Ichikawa, Shoji, Andrea H. Sorenson, Anthony M. Austin, et al.. (2009). Ablation of the Galnt3 Gene Leads to Low-Circulating Intact Fibroblast Growth Factor 23 (Fgf23) Concentrations and Hyperphosphatemia Despite Increased Fgf23 Expression. Endocrinology. 150(6). 2543–2550. 127 indexed citations
10.
Horiguchi, Norio, Lei Wang, Partha Mukhopadhyay, et al.. (2008). Cell Type–Dependent Pro- and Anti-Inflammatory Role of Signal Transducer and Activator of Transcription 3 in Alcoholic Liver Injury. Gastroenterology. 134(4). 1148–1158. 157 indexed citations
11.
Moh, Akira, Wenjun Zhang, Sidney Yu, et al.. (2008). STAT3 Sensitizes Insulin Signaling by Negatively Regulating Glycogen Synthase Kinase-3β. Diabetes. 57(5). 1227–1235. 41 indexed citations
12.
Węgrzyn, Joanna, Ramesh Potla, Yong‐Joon Chwae, et al.. (2008). 372 A novel function of STAT3 in cellular respiration. Cytokine. 43(3). 331–331. 1 indexed citations
13.
Fu, Xin‐Yuan, Wenjun Zhang, Scott Cooper, et al.. (2008). Potential roles of STAT3 in control of differentiation of hematopoietic and neuronal progenitors. Cell Research. 18(S1). S14–S14. 1 indexed citations
14.
Moh, Akira, Yoshiki Iwamoto, Arihiro Kano, et al.. (2007). Role of STAT3 in liver regeneration: survival, DNA synthesis, inflammatory reaction and liver mass recovery. Laboratory Investigation. 87(10). 1018–1028. 114 indexed citations
15.
Moh, Akira, Noriyuki Sakata, Shigeo Takebayashi, et al.. (2004). Increased Production of Urea Hydrogen Peroxide from Maillard Reaction and a UHP-Fenton Pathway Related to Glycoxidation Damage in Chronic Renal Failure. Journal of the American Society of Nephrology. 15(4). 1077–1085. 10 indexed citations
16.
Sakata, Noriyuki, Akira Moh, & Shigeo Takebayashi. (2002). Contribution of superoxide to reduced antioxidant activity of glycoxidative serum albumin. Heart and Vessels. 17(1). 22–29. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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