Ajay Abraham

629 total citations
30 papers, 389 citations indexed

About

Ajay Abraham is a scholar working on Molecular Biology, Hematology and Genetics. According to data from OpenAlex, Ajay Abraham has authored 30 papers receiving a total of 389 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 15 papers in Hematology and 5 papers in Genetics. Recurrent topics in Ajay Abraham's work include Acute Myeloid Leukemia Research (10 papers), Chronic Myeloid Leukemia Treatments (7 papers) and Histone Deacetylase Inhibitors Research (6 papers). Ajay Abraham is often cited by papers focused on Acute Myeloid Leukemia Research (10 papers), Chronic Myeloid Leukemia Treatments (7 papers) and Histone Deacetylase Inhibitors Research (6 papers). Ajay Abraham collaborates with scholars based in United States, India and China. Ajay Abraham's co-authors include Poonkuzhali Balasubramanian, Vikram Mathews, Shaji R Velayudhan, Biju George, Alok Srivastava, Mammen Chandy, William Matsui, Kavitha M. Lakshmi, Aby Abraham and Partha Krishnamurthy and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Nature Communications.

In The Last Decade

Ajay Abraham

29 papers receiving 384 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ajay Abraham United States 10 247 172 86 68 53 30 389
Mengxia Yu China 15 282 1.1× 204 1.2× 90 1.0× 87 1.3× 41 0.8× 35 470
Qitian Mu China 11 209 0.8× 127 0.7× 61 0.7× 62 0.9× 33 0.6× 61 370
Xiongpeng Zhu China 9 207 0.8× 128 0.7× 73 0.8× 66 1.0× 32 0.6× 24 345
Mohamed Elhamri France 14 181 0.7× 323 1.9× 159 1.8× 91 1.3× 114 2.2× 44 507
Raquel Alves Portugal 13 196 0.8× 179 1.0× 63 0.7× 111 1.6× 25 0.5× 35 421
Laixi Bi China 12 199 0.8× 102 0.6× 81 0.9× 38 0.6× 26 0.5× 15 385
Shuqian Xu China 10 148 0.6× 189 1.1× 35 0.4× 102 1.5× 25 0.5× 20 381
N. H. Russell United Kingdom 12 255 1.0× 201 1.2× 129 1.5× 39 0.6× 60 1.1× 17 454
Florian Perner Germany 10 293 1.2× 238 1.4× 64 0.7× 124 1.8× 21 0.4× 26 471
Julie Turzanski United Kingdom 11 145 0.6× 119 0.7× 173 2.0× 45 0.7× 39 0.7× 15 342

Countries citing papers authored by Ajay Abraham

Since Specialization
Citations

This map shows the geographic impact of Ajay Abraham's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ajay Abraham with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ajay Abraham more than expected).

Fields of papers citing papers by Ajay Abraham

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ajay Abraham. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ajay Abraham. The network helps show where Ajay Abraham may publish in the future.

Co-authorship network of co-authors of Ajay Abraham

This figure shows the co-authorship network connecting the top 25 collaborators of Ajay Abraham. A scholar is included among the top collaborators of Ajay Abraham based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ajay Abraham. Ajay Abraham is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Abraham, Ajay, et al.. (2024). Arid1a-dependent canonical BAF complex suppresses inflammatory programs to drive efficient germinal center B cell responses. Nature Immunology. 25(9). 1704–1717. 1 indexed citations
2.
Grody, Emanuelle I., Ajay Abraham, Vipul Shukla, & Yogesh Goyal. (2023). Toward a systems-level probing of tumor clonality. iScience. 26(5). 106574–106574. 3 indexed citations
3.
Abraham, Ajay, et al.. (2023). Loss of SWI/SNF Complex Subunit Arid1a in B Cells Promotes Inflammation and Perturbs Germinal Center B Cell Responses. Blood. 142(Supplement 1). 1400–1400. 1 indexed citations
4.
Shah, Mansi, Shaowei Qiu, Hui Li, et al.. (2022). Low c-Kit expression identifies primitive, therapy-resistant CML stem cells. JCI Insight. 8(1). 4 indexed citations
5.
Lopušná, Katarína, et al.. (2021). Dnmt3b catalytic activity is critical for its tumour suppressor function in lymphomagenesis and is associated with c-Met oncogenic signalling. EBioMedicine. 63. 103191–103191. 8 indexed citations
6.
Lopušná, Katarína, Jana Opavska, Ajay Abraham, et al.. (2021). Decreases in different Dnmt3b activities drive distinct development of hematologic malignancies in mice. Journal of Biological Chemistry. 296. 100285–100285. 8 indexed citations
7.
Lopušná, Katarína, Jana Opavska, Staci L. Haney, et al.. (2019). Catalytically inactive Dnmt3b rescues mouse embryonic development by accessory and repressive functions. Nature Communications. 10(1). 4374–4374. 26 indexed citations
8.
Abraham, Ajay, Shaowei Qiu, Balu K. Chacko, et al.. (2019). SIRT1 regulates metabolism and leukemogenic potential in CML stem cells. Journal of Clinical Investigation. 129(7). 2685–2701. 57 indexed citations
10.
Qiu, Shaowei, Chengcheng Yan, Andrew J. Paterson, et al.. (2019). Role of Autophagy in Resistance of FLT3-ITD AML Stem Cells to FLT3 TKI Treatment. Blood. 134(Supplement_1). 2548–2548. 1 indexed citations
11.
Abraham, Ajay, Shaowei Qiu, Balu K. Chacko, et al.. (2018). SIRT1 Mediates Enhanced Mitochondrial Oxidative Phosphorylation in Chronic Myelogenous Leukemia Stem Cells. Blood. 132(Supplement 1). 932–932. 2 indexed citations
13.
Panetta, John C., Ajay Abraham, Kavitha M. Lakshmi, et al.. (2016). Population pharmacokinetics of Daunorubicin in adult patients with acute myeloid leukemia. Cancer Chemotherapy and Pharmacology. 78(5). 1051–1058. 19 indexed citations
14.
Arunachalam, Arun Kumar, Kavitha M. Lakshmi, Ajay Abraham, et al.. (2016). Frequency of rare BCRABL1 fusion transcripts in chronic myeloid leukemia patients. International Journal of Laboratory Hematology. 39(3). 235–242. 28 indexed citations
15.
Abraham, Ajay, et al.. (2013). Harnessing Gene Expression Profiling In Search Of New Candidate Genes For Ara-C Resistance In Acute Myeloid Leukemia. Blood. 122(21). 1299–1299. 1 indexed citations
16.
Balasubramanian, Poonkuzhali, Ashok Kumar Jayavelu, Ajay Abraham, et al.. (2013). NPM1 Mutated AML Is Associated With Lower Expression Of Poor Prognostic Markers BAALC, ERG and MN1 In Adult Patients With Acute Myeloid Leukaemia. Blood. 122(21). 4945–4945. 1 indexed citations
17.
Abraham, Ajay, Wei Zhang, Shaji R Velayudhan, et al.. (2012). Carbonyl reductase 1 expression influences daunorubicin metabolism in acute myeloid leukemia. European Journal of Clinical Pharmacology. 68(12). 1577–1586. 26 indexed citations
18.
Abraham, Ajay, Wei Zhang, Shaji R Velayudhan, et al.. (2012). Cytidine Deaminase Genetic Variants Influence RNA Expression and Cytarabine Cytotoxicity in Acute Myeloid Leukemia. Pharmacogenomics. 13(3). 269–282. 39 indexed citations
19.
Abraham, Ajay, et al.. (2002). Assessment of faecal occult blood loss by qualitative and quantitative methods.. PubMed. 47(2). 491–4. 2 indexed citations
20.
Abraham, Ajay & C. K. Jacob. (2002). Severe hyponatraemia: current concepts on pathogenesis and treatment.. PubMed. 14(5). 277–83.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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