Aidas Alaburda

911 total citations
30 papers, 718 citations indexed

About

Aidas Alaburda is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Cognitive Neuroscience. According to data from OpenAlex, Aidas Alaburda has authored 30 papers receiving a total of 718 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Cellular and Molecular Neuroscience, 12 papers in Molecular Biology and 12 papers in Cognitive Neuroscience. Recurrent topics in Aidas Alaburda's work include Neural dynamics and brain function (11 papers), Zebrafish Biomedical Research Applications (9 papers) and Neuroscience and Neural Engineering (8 papers). Aidas Alaburda is often cited by papers focused on Neural dynamics and brain function (11 papers), Zebrafish Biomedical Research Applications (9 papers) and Neuroscience and Neural Engineering (8 papers). Aidas Alaburda collaborates with scholars based in Lithuania, Denmark and Poland. Aidas Alaburda's co-authors include Jørn Hounsgaard, Jean‐Françóis Perrier, Rune W. Berg, Nanna MacAulay, Raúl E. Russo, Eiva Bernotienė, Ilona Uzielienè, Vitalij Novickij, Feliksas Ivanauskas and Gytis Svirskis and has published in prestigious journals such as Science, Journal of Neuroscience and The Journal of Physiology.

In The Last Decade

Aidas Alaburda

28 papers receiving 706 citations

Peers

Aidas Alaburda

CMN

Gytis Svirskis Lithuania

Frédéric Bretzner Canada

Carmelo Bellardita Sweden

Tuan V. Bui Canada

Jianren Song China

Roberto Leiras Sweden

Sabrina Tazerart France

Katherine R. Croce United States

Martin Hägglund Sweden

Vanessa Caldeira Sweden

Gytis Svirskis Lithuania

Aidas Alaburda

526 ×1.3

CMN

618 ×1.9

208 ×1.1

147 ×0.8

75 ×0.4

Citations per year, relative to Aidas Alaburda Aidas Alaburda (= 1×) peers Gytis Svirskis

Countries citing papers authored by Aidas Alaburda

Since Specialization

Citations

This map shows the geographic impact of Aidas Alaburda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Aidas Alaburda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Aidas Alaburda more than expected).

Fields of papers citing papers by Aidas Alaburda

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Aidas Alaburda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Aidas Alaburda. The network helps show where Aidas Alaburda may publish in the future.

Co-authorship network of co-authors of Aidas Alaburda

This figure shows the co-authorship network connecting the top 25 collaborators of Aidas Alaburda. A scholar is included among the top collaborators of Aidas Alaburda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Aidas Alaburda. Aidas Alaburda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Alaburda, Aidas, et al.. (2025). The Effect of TGF-β3 and IL-1β on L-Type Voltage-Operated Calcium Channels and Calcium Ion Homeostasis in Osteoarthritic Chondrocytes and Human Bone Marrow-Derived Mesenchymal Stem Cells During Chondrogenesis. Pharmaceutics. 17(3). 343–343. 1 indexed citations

Fijałkowski, Łukasz, Aidas Alaburda, Grzegorz Grześk, et al.. (2025). SGLT2 Inhibitors: From Molecular Mechanisms to Clinical Outcomes in Cardiology and Diabetology. Molecules. 30(15). 3112–3112. 1 indexed citations

Ivanauskas, Feliksas, et al.. (2024). Hysteresis and bistability in synaptic transmission modeled as a chain of biochemical reactions with a positive feedback. Nonlinear Analysis Modelling and Control. 746–761. 1 indexed citations

Kistamás, Kornél, Suchitra Muenthaisong, Aidas Alaburda, et al.. (2024). The Current State of Realistic Heart Models for Disease Modelling and Cardiotoxicity. International Journal of Molecular Sciences. 25(17). 9186–9186. 4 indexed citations

Ivanauskas, Feliksas, et al.. (2023). Mathematical Model of Synaptic Long-Term Potentiation as a Bistability in a Chain of Biochemical Reactions with a Positive Feedback. Acta Biotheoretica. 71(3). 16–16. 1 indexed citations

Uzielienè, Ilona, et al.. (2023). Electrical Stimulation in Cartilage Tissue Engineering. Bioengineering. 10(4). 454–454. 18 indexed citations

Alaburda, Aidas, et al.. (2023). Unveiling the Multifaceted Problems Associated with Dysrhythmia. International Journal of Molecular Sciences. 25(1). 263–263.

Uzielienè, Ilona, et al.. (2021). Effect of different types of electrical stimulation on intracellular calcium levels in human mesenchymal stem cells and chondrocytes. Osteoarthritis and Cartilage. 29. S408–S409. 1 indexed citations

Bernotienė, Eiva, et al.. (2019). The role of voltage-operated calcium channels in chondrogenic properties of human mesenchymal stem cells and chondrocytes. Osteoarthritis and Cartilage. 27. S202–S203. 1 indexed citations

10.

Ivanauskas, Feliksas, et al.. (2018). The “Memory” Effect in a Chain of Biochemical Reactions with a Positive Feedback is Enhanced by Substrate Saturation Described by Michaelis–Menten Kinetics. Bulletin of Mathematical Biology. 81(3). 919–935. 4 indexed citations

11.

Hounsgaard, Jørn, et al.. (2016). Irregular Firing and High-Conductance States in Spinal Motoneurons during Scratching and Swimming. Journal of Neuroscience. 36(21). 5799–5807. 13 indexed citations

12.

Alaburda, Aidas, et al.. (2016). The influence of increased membrane conductance on response properties of spinal motoneurons. Brain Research. 1648(Pt A). 110–118. 3 indexed citations

13.

Alaburda, Aidas, et al.. (2014). Dense Distributed Processing in a Hindlimb Scratch Motor Network. Journal of Neuroscience. 34(32). 10756–10764. 12 indexed citations

14.

Rukšėnas, Osvaldas, et al.. (2012). Gain of spinal motoneurons measured from square and ramp current pulses. Brain Research. 1450. 33–39. 2 indexed citations

15.

Hounsgaard, Jørn, et al.. (2012). Inhibition of motoneurons during the cutaneous silent period in the spinal cord of the turtle. Experimental Brain Research. 220(1). 23–28. 4 indexed citations

16.

Svirskis, Gytis, et al.. (2010). Persistent sodium current decreases transient gain in turtle motoneurons. Brain Research. 1373. 11–16. 9 indexed citations

17.

Perrier, Jean‐Françóis, Aidas Alaburda, & Jørn Hounsgaard. (2003). 5-HT1A receptors increase excitability of spinal motoneurons by inhibiting a TASK-1-like K+ current in the adult turtle. The Journal of Physiology. 548(2). 485–492. 43 indexed citations

18.

Perrier, Jean‐Françóis, Aidas Alaburda, & Jørn Hounsgaard. (2002). Spinal plasticity mediated by postsynaptic L-type Ca2+ channels. Brain Research Reviews. 40(1-3). 223–229. 80 indexed citations

19.

Alaburda, Aidas, Jean‐Françóis Perrier, & Jørn Hounsgaard. (2002). Mechanisms Causing Plateau Potentials in Spinal Motoneurones. Advances in experimental medicine and biology. 508. 219–226. 67 indexed citations

20.

Alaburda, Aidas, Jean‐Françóis Perrier, & Jørn Hounsgaard. (2002). An M‐like outward current regulates the excitability of spinal motoneurones in the adult turtle. The Journal of Physiology. 540(3). 875–881. 60 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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