Agata Cieślak

905 total citations
16 papers, 492 citations indexed

About

Agata Cieślak is a scholar working on Public Health, Environmental and Occupational Health, Molecular Biology and Hematology. According to data from OpenAlex, Agata Cieślak has authored 16 papers receiving a total of 492 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Public Health, Environmental and Occupational Health, 8 papers in Molecular Biology and 6 papers in Hematology. Recurrent topics in Agata Cieślak's work include Acute Lymphoblastic Leukemia research (9 papers), Childhood Cancer Survivors' Quality of Life (4 papers) and Immune Cell Function and Interaction (4 papers). Agata Cieślak is often cited by papers focused on Acute Lymphoblastic Leukemia research (9 papers), Childhood Cancer Survivors' Quality of Life (4 papers) and Immune Cell Function and Interaction (4 papers). Agata Cieślak collaborates with scholars based in France, United States and Netherlands. Agata Cieślak's co-authors include Vahid Asnafi, Elizabeth Macintyre, Salvatore Spicuglia, Norbert Ifrah, Hervé Dombret, Mohamed Belhocine, Nicolas Boissel, Ludovic Lhermitte, Jonathan Bond and Arnaud Petit and has published in prestigious journals such as Journal of Clinical Investigation, The Journal of Experimental Medicine and Journal of Clinical Oncology.

In The Last Decade

Agata Cieślak

16 papers receiving 489 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Agata Cieślak France 12 248 184 173 110 105 16 492
Amélie Trinquand France 12 213 0.9× 178 1.0× 102 0.6× 121 1.1× 137 1.3× 23 462
Aurore Touzart France 12 199 0.8× 181 1.0× 143 0.8× 81 0.7× 100 1.0× 24 424
A. W. Langerak Netherlands 9 193 0.8× 169 0.9× 151 0.9× 116 1.1× 125 1.2× 13 462
Chiara Palmi Italy 10 360 1.5× 326 1.8× 156 0.9× 130 1.2× 51 0.5× 22 597
Cecilia Sheen United States 6 231 0.9× 280 1.5× 132 0.8× 120 1.1× 55 0.5× 9 569
Julia A. Meyer United States 8 292 1.2× 218 1.2× 336 1.9× 101 0.9× 37 0.4× 10 605
Frauke Bellos Germany 11 145 0.6× 254 1.4× 138 0.8× 98 0.9× 66 0.6× 21 428
Michael Hadler United States 7 261 1.1× 233 1.3× 255 1.5× 83 0.8× 53 0.5× 12 531
Renate Panzer‐Grümayer Austria 16 527 2.1× 404 2.2× 228 1.3× 144 1.3× 74 0.7× 35 766
Emanuela Giarin Italy 11 196 0.8× 291 1.6× 229 1.3× 72 0.7× 33 0.3× 16 470

Countries citing papers authored by Agata Cieślak

Since Specialization
Citations

This map shows the geographic impact of Agata Cieślak's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Agata Cieślak with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Agata Cieślak more than expected).

Fields of papers citing papers by Agata Cieślak

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Agata Cieślak. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Agata Cieślak. The network helps show where Agata Cieślak may publish in the future.

Co-authorship network of co-authors of Agata Cieślak

This figure shows the co-authorship network connecting the top 25 collaborators of Agata Cieślak. A scholar is included among the top collaborators of Agata Cieślak based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Agata Cieślak. Agata Cieślak is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Cieślak, Agata, Mourad Amrane, Renaud Vincentelli, et al.. (2024). RANKL treatment restores thymic function and improves T cell–mediated immune responses in aged mice. Science Translational Medicine. 16(776). eadp3171–eadp3171. 9 indexed citations
2.
Cabannes‐Hamy, Aurélie, Guillaume P. Andrieu, Agata Cieślak, et al.. (2023). PHF6 -altered T-ALL Harbor Epigenetic Repressive Switch at Bivalent Promoters and Respond to 5-Azacitidine and Venetoclax. Clinical Cancer Research. 30(1). 94–105. 3 indexed citations
3.
Touzart, Aurore, Anand Mayakonda, Joschka Hey, et al.. (2021). Epigenetic analysis of patients with T-ALL identifies poor outcomes and a hypomethylating agent-responsive subgroup. Science Translational Medicine. 13(595). 23 indexed citations
4.
Belhocine, Mohamed, Mathieu Simonin, Agata Cieślak, et al.. (2021). Dynamics of broad H3K4me3 domains uncover an epigenetic switch between cell identity and cancer-related genes. Genome Research. 32(7). 1328–1342. 17 indexed citations
5.
Andrieu, Guillaume P., Milena Kohn, Mathieu Simonin, et al.. (2021). PRC2 loss of function confers a targetable vulnerability to BET proteins in T-ALL. Blood. 138(19). 1855–1869. 22 indexed citations
6.
Hoareau‐Aveilla, Coralie, Marie Tosolini, Patrick Villarèse, et al.. (2020). ALK-transformed mature T lymphocytes restore early thymus progenitor features. Journal of Clinical Investigation. 130(12). 6395–6408. 9 indexed citations
7.
Cieślak, Agata, Guillaume Charbonnier, Melania Tesio, et al.. (2020). Blueprint of human thymopoiesis reveals molecular mechanisms of stage-specific TCR enhancer activation. The Journal of Experimental Medicine. 217(9). 49 indexed citations
8.
Amirache, Fouzia, Camille Lévy, Caroline Costa, et al.. (2019). Baboon envelope LVs efficiently transduced human adult, fetal, and progenitor T cells and corrected SCID-X1 T-cell deficiency. Blood Advances. 3(3). 461–475. 22 indexed citations
9.
Villarèse, Patrick, Amélie Trinquand, Sandrine Le Noir, et al.. (2017). TCRα rearrangements identify a subgroup of NKL-deregulated adult T-ALLs associated with favorable outcome. Leukemia. 32(1). 61–71. 19 indexed citations
10.
Boëlle, Pierre‐Yves, Caroline Deswarte, Salvatore Spicuglia, et al.. (2017). Homeobox protein TLX3 activates miR-125b expression to promote T-cell acute lymphoblastic leukemia. Blood Advances. 1(12). 733–747. 30 indexed citations
11.
Bond, Jonathan, Carlos Graux, Ludovic Lhermitte, et al.. (2017). Early Response–Based Therapy Stratification Improves Survival in Adult Early Thymic Precursor Acute Lymphoblastic Leukemia: A Group for Research on Adult Acute Lymphoblastic Leukemia Study. Journal of Clinical Oncology. 35(23). 2683–2691. 116 indexed citations
13.
Dussiau, Charles, Amélie Trinquand, Ludovic Lhermitte, et al.. (2015). Targeting IRAK1 in T-Cell acute lymphoblastic leukemia. Oncotarget. 6(22). 18956–18965. 16 indexed citations
14.
Bond, Jonathan, Tony Marchand, Aurore Touzart, et al.. (2015). An Early Thymic Precursor Phenotype Predicts Outcome Exclusively in HOXA-Overexpressing Adult T-ALL: A GRAALL Study. Blood. 126(23). 808–808. 3 indexed citations
15.
Abdelali, Raouf Ben, Anne Roggy, Thibaut Leguay, et al.. (2014). SET-NUP214 is a recurrent γδ lineage-specific fusion transcript associated with corticosteroid/chemotherapy resistance in adult T-ALL. Blood. 123(12). 1860–1863. 36 indexed citations
16.
Callens, Céline, Frédéric Baleydier, Étienne Lengliné, et al.. (2012). Clinical Impact of NOTCH1 and/or FBXW7 Mutations, FLASH Deletion, and TCR Status in Pediatric T-Cell Lymphoblastic Lymphoma. Journal of Clinical Oncology. 30(16). 1966–1973. 75 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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