Adi Tabib

2.0k total citations
14 papers, 349 citations indexed

About

Adi Tabib is a scholar working on Immunology, Neurology and Molecular Biology. According to data from OpenAlex, Adi Tabib has authored 14 papers receiving a total of 349 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Immunology, 5 papers in Neurology and 3 papers in Molecular Biology. Recurrent topics in Adi Tabib's work include Complement system in diseases (8 papers), Peripheral Neuropathies and Disorders (5 papers) and Phagocytosis and Immune Regulation (4 papers). Adi Tabib is often cited by papers focused on Complement system in diseases (8 papers), Peripheral Neuropathies and Disorders (5 papers) and Phagocytosis and Immune Regulation (4 papers). Adi Tabib collaborates with scholars based in Israel, United Kingdom and Germany. Adi Tabib's co-authors include Dror Mevorach, Orly Elpeleg, Zamir Shorer, Asaf Ta‐Shma, Avraham Shaag, Shamir Zenvirt, Yakov Fellig, Hanoch Goldshmidt, Bruria Ben‐Zeev and Yoram Nevo and has published in prestigious journals such as Blood, PLoS ONE and Annals of Neurology.

In The Last Decade

Adi Tabib

14 papers receiving 346 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Adi Tabib Israel 10 233 76 65 60 56 14 349
Aigli G. Vakrakou Greece 12 151 0.6× 19 0.3× 116 1.8× 115 1.9× 160 2.9× 40 462
Monica van der Vieren United States 8 352 1.5× 93 1.2× 31 0.5× 84 1.4× 104 1.9× 9 630
Will G. James Australia 10 440 1.9× 34 0.4× 21 0.3× 30 0.5× 111 2.0× 10 617
Chantal A. Mutsaers United Kingdom 8 99 0.4× 32 0.4× 31 0.5× 46 0.8× 300 5.4× 9 460
Jennyfer Zerbib France 16 142 0.6× 33 0.4× 17 0.3× 17 0.3× 310 5.5× 23 747
Simon Stritt Germany 10 63 0.3× 107 1.4× 12 0.2× 33 0.6× 79 1.4× 15 345
Samantha Loveless United Kingdom 10 120 0.5× 19 0.3× 68 1.0× 35 0.6× 172 3.1× 19 427
Tim J. Vyse United Kingdom 11 350 1.5× 21 0.3× 18 0.3× 30 0.5× 138 2.5× 17 567
Ségolène Pettré France 8 326 1.4× 20 0.3× 18 0.3× 13 0.2× 63 1.1× 10 513
Dingxin Pan United Kingdom 10 140 0.6× 42 0.6× 8 0.1× 50 0.8× 136 2.4× 14 356

Countries citing papers authored by Adi Tabib

Since Specialization
Citations

This map shows the geographic impact of Adi Tabib's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Adi Tabib with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Adi Tabib more than expected).

Fields of papers citing papers by Adi Tabib

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Adi Tabib. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Adi Tabib. The network helps show where Adi Tabib may publish in the future.

Co-authorship network of co-authors of Adi Tabib

This figure shows the co-authorship network connecting the top 25 collaborators of Adi Tabib. A scholar is included among the top collaborators of Adi Tabib based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Adi Tabib. Adi Tabib is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
1.
Eshed‐Eisenbach, Yael, Adi Tabib, Anya Vainshtein, et al.. (2023). Complement-membrane regulatory proteins are absent from the nodes of Ranvier in the peripheral nervous system. Journal of Neuroinflammation. 20(1). 245–245. 3 indexed citations
2.
Tabib, Adi, et al.. (2018). Prothrombotic mechanisms in patients with congenital p.Cys89Tyr mutation in CD59. Thrombosis Research. 168. 67–77. 7 indexed citations
3.
Kurolap, Alina, Tova Hershkovitz, Adi Tabib, et al.. (2018). Eculizumab Is Safe and Effective as a Long‐term Treatment for Protein‐losing Enteropathy Due to CD55 Deficiency. Journal of Pediatric Gastroenterology and Nutrition. 68(3). 325–333. 20 indexed citations
4.
Eshed‐Eisenbach, Yael, Adi Tabib, B. Paul Morgan, et al.. (2018). Molecular pathogenesis of human CD59 deficiency. Neurology Genetics. 4(6). e280–e280. 20 indexed citations
5.
Tabib, Adi, et al.. (2017). Demyelination, strokes, and eculizumab: Lessons from the congenital CD59 gene mutations. Molecular Immunology. 89. 69–72. 19 indexed citations
7.
Mevorach, Dror, Yackov Berkun, Asaf Ta‐Shma, et al.. (2016). Therapy with eculizumab for patients with CD59 p.Cys89Tyr mutation. Annals of Neurology. 80(5). 708–717. 38 indexed citations
8.
Ben‐Zeev, Bruria, Adi Tabib, Andreea Nissenkorn, et al.. (2015). Devastating recurrent brain ischemic infarctions and retinal disease in pediatric patients with CD59 deficiency. European Journal of Paediatric Neurology. 19(6). 688–693. 18 indexed citations
9.
Tabib, Adi, et al.. (2015). Apoptotic Cells Induce NF-κB and Inflammasome Negative Signaling. PLoS ONE. 10(3). e0122440–e0122440. 25 indexed citations
10.
Stepensky, Polina, Ann Saada, Marianne Cowan, et al.. (2013). The Thr224Asn mutation in the VPS45 gene is associated with the congenital neutropenia and primary myelofibrosis of infancy. Blood. 121(25). 5078–5087. 44 indexed citations
11.
Nevo, Yoram, Bruria Ben‐Zeev, Adi Tabib, et al.. (2012). CD59 deficiency is associated with chronic hemolysis and childhood relapsing immune-mediated polyneuropathy. Blood. 121(1). 129–135. 111 indexed citations
12.
Mevorach, Dror, et al.. (2010). What do we mean when we write “senescence,”“apoptosis,”“necrosis,” or “clearance of dying cells”?. Annals of the New York Academy of Sciences. 1209(1). 1–9. 24 indexed citations
13.
Tabib, Adi, Alon Krispin, Uriel Trahtemberg, et al.. (2009). Thrombospondin-1-N-Terminal Domain Induces a Phagocytic State and Thrombospondin-1-C-Terminal Domain Induces a Tolerizing Phenotype in Dendritic Cells. PLoS ONE. 4(8). e6840–e6840. 13 indexed citations
14.
Blaes, Nelly, et al.. (1988). [Establishment of a transformed line of arterial smooth muscle cells].. PubMed. 307(8). 499–503. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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