Adam J. Prus

727 total citations
44 papers, 541 citations indexed

About

Adam J. Prus is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Psychiatry and Mental health. According to data from OpenAlex, Adam J. Prus has authored 44 papers receiving a total of 541 indexed citations (citations by other indexed papers that have themselves been cited), including 37 papers in Cellular and Molecular Neuroscience, 26 papers in Molecular Biology and 11 papers in Psychiatry and Mental health. Recurrent topics in Adam J. Prus's work include Neurotransmitter Receptor Influence on Behavior (26 papers), Receptor Mechanisms and Signaling (23 papers) and Neuroscience and Neuropharmacology Research (18 papers). Adam J. Prus is often cited by papers focused on Neurotransmitter Receptor Influence on Behavior (26 papers), Receptor Mechanisms and Signaling (23 papers) and Neuroscience and Neuropharmacology Research (18 papers). Adam J. Prus collaborates with scholars based in United States, Poland and Bulgaria. Adam J. Prus's co-authors include Herbert Y. Meltzer, Joseph H. Porter, Jin Dai, Mei Huang, Zhu Li, Alan L. Pehrson, Todd M. Hillhouse, Lisa E. Baker, Amber LaCrosse and Junji Ichikawa and has published in prestigious journals such as Brain Research, Journal of Pharmacology and Experimental Therapeutics and Psychopharmacology.

In The Last Decade

Adam J. Prus

44 papers receiving 530 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Adam J. Prus United States 16 421 297 114 65 64 44 541
Virginia Lehmann-Masten United States 9 376 0.9× 213 0.7× 63 0.6× 66 1.0× 77 1.2× 9 497
Aleksandra Ewa Basak Türkiye 3 304 0.7× 206 0.7× 113 1.0× 72 1.1× 84 1.3× 5 560
Claudia Kriegebaum Germany 6 251 0.6× 133 0.4× 75 0.7× 79 1.2× 57 0.9× 8 458
Aaron D Logue United States 9 289 0.7× 189 0.6× 76 0.7× 49 0.8× 40 0.6× 9 401
G.G. Nomikos Sweden 7 471 1.1× 340 1.1× 56 0.5× 47 0.7× 85 1.3× 10 563
Ilya Sukhanov Russia 12 490 1.2× 433 1.5× 45 0.4× 40 0.6× 112 1.8× 34 641
Shlomit Flaisher-Grinberg United States 12 205 0.5× 140 0.5× 118 1.0× 73 1.1× 69 1.1× 17 477
M.C. Scorza Uruguay 5 362 0.9× 156 0.5× 108 0.9× 39 0.6× 84 1.3× 6 477
Elliot A. Loh Canada 8 510 1.2× 225 0.8× 48 0.4× 84 1.3× 124 1.9× 12 565
Bengt E. Hildebrand Sweden 9 600 1.4× 540 1.8× 92 0.8× 70 1.1× 75 1.2× 12 783

Countries citing papers authored by Adam J. Prus

Since Specialization
Citations

This map shows the geographic impact of Adam J. Prus's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Adam J. Prus with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Adam J. Prus more than expected).

Fields of papers citing papers by Adam J. Prus

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Adam J. Prus. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Adam J. Prus. The network helps show where Adam J. Prus may publish in the future.

Co-authorship network of co-authors of Adam J. Prus

This figure shows the co-authorship network connecting the top 25 collaborators of Adam J. Prus. A scholar is included among the top collaborators of Adam J. Prus based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Adam J. Prus. Adam J. Prus is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Prus, Adam J., et al.. (2024). Discriminative stimulus properties of two training doses of gabapentin in rats: Substitution by pregabalin, diazepam, and pentobarbital.. Experimental and Clinical Psychopharmacology. 32(4). 485–495. 2 indexed citations
2.
Porter, Joseph H., Adam J. Prus, & Donald A. Overton. (2018). Drug Discrimination: Historical Origins, Important Concepts, and Principles. Current topics in behavioral neurosciences. 39. 3–26. 14 indexed citations
3.
Porter, Joseph H., Kevin A. Webster, & Adam J. Prus. (2017). Translational Value of Drug Discrimination with Typical and Atypical Antipsychotic Drugs. Current topics in behavioral neurosciences. 39. 193–212. 2 indexed citations
4.
Prus, Adam J., et al.. (2015). The antidepressant drugs fluoxetine and duloxetine produce anxiolytic-like effects in a schedule-induced polydipsia paradigm in rats. Behavioural Pharmacology. 26(5). 489–494. 4 indexed citations
5.
Keiser, Ashley A., et al.. (2014). Systemic administration of the neurotensin NTS₁-receptor agonist PD149163 improves performance on a memory task in naturally deficient male Brown Norway rats.. Experimental and Clinical Psychopharmacology. 22(6). 541–547. 17 indexed citations
6.
Hillhouse, Todd M., et al.. (2014). The quetiapine active metabolite N-desalkylquetiapine and the neurotensin NTS₁ receptor agonist PD149163 exhibit antidepressant-like effects on operant responding in male rats.. Experimental and Clinical Psychopharmacology. 22(6). 548–556. 4 indexed citations
7.
Hillhouse, Todd M. & Adam J. Prus. (2013). Effects of the neurotensin NTS1 receptor agonist PD149163 on visual signal detection in rats. European Journal of Pharmacology. 721(1-3). 201–207. 9 indexed citations
8.
Prus, Adam J., Todd M. Hillhouse, & Amber LaCrosse. (2013). Acute, but not repeated, administration of the neurotensin NTS1 receptor agonist PD149163 decreases conditioned footshock-induced ultrasonic vocalizations in rats. Progress in Neuro-Psychopharmacology and Biological Psychiatry. 49. 78–84. 27 indexed citations
9.
Holly, Elizabeth N., et al.. (2011). The neurotensin-1 receptor agonist PD149163 inhibits conditioned avoidance responding without producing catalepsy in rats. European Neuropsychopharmacology. 21(7). 526–531. 15 indexed citations
10.
Li, Zhu, Adam J. Prus, Jin Dai, & Herbert Y. Meltzer. (2009). Differential Effects of M1 and 5-Hydroxytryptamine1A Receptors on Atypical Antipsychotic Drug-Induced Dopamine Efflux in the Medial Prefrontal Cortex. Journal of Pharmacology and Experimental Therapeutics. 330(3). 948–955. 19 indexed citations
11.
Porter, Joseph H. & Adam J. Prus. (2008). Discriminative stimulus properties of atypical and typical antipsychotic drugs: a review of preclinical studies. Psychopharmacology. 203(2). 279–294. 20 indexed citations
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Prus, Adam J., Mei Huang, Zhu Li, Jin Dai, & Herbert Y. Meltzer. (2007). The neurotensin analog NT69L enhances medial prefrontal cortical dopamine and acetylcholine efflux: Potentiation of risperidone-, but not haloperidol-, induced dopamine efflux. Brain Research. 1184. 354–364. 22 indexed citations
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Prus, Adam J., et al.. (2005). Serotonin receptor mechanisms mediate the discriminative stimulus properties of the atypical antipsychotic clozapine in C57BL/6 mice. Psychopharmacology. 180(1). 49–56. 23 indexed citations
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Li, Zhu, Junji Ichikawa, Mei Huang, et al.. (2005). ACP-103, a 5-HT2A/2C inverse agonist, potentiates haloperidol-induced dopamine release in rat medial prefrontal cortex and nucleus accumbens. Psychopharmacology. 183(2). 144–153. 49 indexed citations
19.
Porter, Joseph H., Adam J. Prus, Robert E. Vann, & Stephen A. Varvel. (2004). Discriminative stimulus properties of the atypical antipsychotic clozapine and the typical antipsychotic chlorpromazine in a three-choice drug discrimination procedure in rats. Psychopharmacology. 178(1). 67–77. 20 indexed citations
20.
Goudie, Andrew J., Lisa E. Baker, Judith A. Smith, et al.. (2001). Common discriminative stimulus properties in rats of muscarinic antagonists, clozapine and the D 3 preferring antagonist PNU-99194A: an analysis of possible mechanisms. Behavioural Pharmacology. 12(5). 303–315. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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