Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies
if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the
same subfield and year (this is the minimum needed to enter the top 1%, not the average
within it), or reaches the top citation threshold in at least one of its specific research
topics.
Inhibition of the glucose transporter SGLT2 with dapagliflozin in pancreatic alpha cells triggers glucagon secretion
2015538 citationsWilly Malaisse, Abdullah Sener et al.profile →
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late)
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Countries citing papers authored by Abdullah Sener
Since
Specialization
Citations
This map shows the geographic impact of Abdullah Sener's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Abdullah Sener with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Abdullah Sener more than expected).
This network shows the impact of papers produced by Abdullah Sener. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Abdullah Sener. The network helps show where Abdullah Sener may publish in the future.
Co-authorship network of co-authors of Abdullah Sener
This figure shows the co-authorship network connecting the top 25 collaborators of Abdullah Sener.
A scholar is included among the top collaborators of Abdullah Sener based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with Abdullah Sener. Abdullah Sener is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Malaisse, Willy, et al.. (2012). Role of anoctamin 1 (TMEM16A) as a volume regulated anion channel in insulin-producing cells. Diabetologia.1 indexed citations
3.
Shlyonsky, Vadim, Karim Louchami, Alain Boom, et al.. (2012). Does NAD(P)H oxidase-derived H2O2 participate in hypotonicity-induced insulin release by activating VRAC in ß-cells?. European Journal of Applied Physiology. 463. 377–390.1 indexed citations
4.
Malaisse, Willy, et al.. (2010). The metabolic syndrome of omega-3-depleted rats. VIII. Dietary lipid-induced liver steatosis. 3(3). 1–9.2 indexed citations
5.
Malaisse, Willy, Karim Louchami, Yaoyu Zhang, et al.. (2005). Perturbation of K+ fluxes in pancreatic islets from ω3-fatty-acid-depleted rats. Diabetes.2 indexed citations
6.
Courtois, Philippe, Abdullah Sener, & Willy Malaisse. (2001). Carbonic anhydrase activity in pancreatic islets. Diabetologia.1 indexed citations
7.
Malaisse, Willy, Remedios Ramı́rez, Philippe Courtois, Joanne Rasschaert, & Abdullah Sener. (2000). D-[³H]mannoheptulose uptake and phosphorylation in different cell types. European Journal of Applied Physiology. 441.1 indexed citations
8.
Ramı́rez, Remedios, et al.. (1995). THE COUPLING OF METABOLIC TO SECRETORY EVENTS IN PANCREATIC ISLETS: THE MITOCHONDRIAL REDOX STATE. Medical science research. 23(4). 249–250.1 indexed citations
9.
Giroix, Marie-Hélène, et al.. (1994). Pancreatic islet response to dicarboxylic acid esters in rats with type 2 diabetes. Diabetologia.1 indexed citations
10.
Fernández-Álvarez, Josefa, Ignacio Conget, Abdullah Sener, Ramón Gomis, & Willy Malaisse. (1993). Enzymatic, metabolic and secretory patterns in human islets of type 2 diabetics. Diabetologia.1 indexed citations
11.
Sener, Abdullah, et al.. (1992). Pancreatic islet FAD-linked glycerophosphate dehydrogenase activity in a model of B cell glucotoxicity. Medical science research. 20(19). 701–703.10 indexed citations
12.
Sener, Abdullah, et al.. (1988). D-glucose uptake by pancreatic islets from normal and hyperglycemic rats. Medical science research. 16. 1181–1182.2 indexed citations
13.
Carpinelli, Ângelo Rafael, Abdullah Sener, & Willy Malaisse. (1987). Positive feedback effect of ATP on glucose phosphorylation in pancreatic islets. Medical science research. 15. 481–482.5 indexed citations
Dufrane, Simon, Willy Malaisse, & Abdullah Sener. (1983). Aldose reductase activity in pancreatic islets. Diabetologia. 25. 152.3 indexed citations
16.
Malaisse, Willy, et al.. (1982). Metabolic response of pancreatic islets to the anomers of D-mannose. Diabetologia. 23(2). 185.4 indexed citations
17.
Malaisse, Willy, et al.. (1981). Glucose-induced activation of phosphofructokinase in pancreatic islets. Diabetes. 30. 115.2 indexed citations
18.
Malaisse, Willy & Abdullah Sener. (1980). Glutamate dehydrogenase in pancreatic islets: a receptor site for certain insulinotropic amino acids. Diabetologia. 19. 296–297.5 indexed citations
19.
Sener, Abdullah, Shoji Kawazu, & Willy Malaisse. (1978). Dissociation between the oxidation rate and insulinotropic capacity of lactate and pyruvate. Diabetologia. 15. 269.2 indexed citations
20.
Malaisse, Willy, Abdullah Sener, Shoji Kawazu, & Antônio C. Boschero. (1977). Nature of the coupling between metabolic and cationic events in pancreatic islets. Diabetologia. 13. 416.2 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.