Abdul Basit Baba

924 total citations · 1 hit paper
12 papers, 671 citations indexed

About

Abdul Basit Baba is a scholar working on Molecular Biology, Oncology and Plant Science. According to data from OpenAlex, Abdul Basit Baba has authored 12 papers receiving a total of 671 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 4 papers in Oncology and 4 papers in Plant Science. Recurrent topics in Abdul Basit Baba's work include Cytokine Signaling Pathways and Interactions (3 papers), Phytochemical compounds biological activities (3 papers) and Retinoids in leukemia and cellular processes (2 papers). Abdul Basit Baba is often cited by papers focused on Cytokine Signaling Pathways and Interactions (3 papers), Phytochemical compounds biological activities (3 papers) and Retinoids in leukemia and cellular processes (2 papers). Abdul Basit Baba collaborates with scholars based in India and Serbia. Abdul Basit Baba's co-authors include Siddavaram Nagini, Kowshik Jaganathan, K. Kavitha, Madhulika Dixit, Ifra Mushtaq, Dil Afroze, Bilal Rah, Gh Rasool Bhat, Hemant Giri and Indranil Chattopadhyay and has published in prestigious journals such as PLoS ONE, Food and Chemical Toxicology and Biochimica et Biophysica Acta (BBA) - General Subjects.

In The Last Decade

Abdul Basit Baba

12 papers receiving 660 citations

Hit Papers

Transforming Growth Factor-Beta (TGF-β) Signaling in Canc... 2022 2026 2023 2024 2022 40 80 120
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Transforming Growth Factor-Beta (TGF-β) Signaling in Cancer-A Betrayal Within
    2022 139 citations Abdul Basit Baba, Bilal Rah et al. Frontiers in Pharmacology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Abdul Basit Baba India 10 343 154 138 132 82 12 671
Ping Pei United States 16 359 1.0× 145 0.9× 133 1.0× 115 0.9× 49 0.6× 32 783
Youn Hee Joung South Korea 16 425 1.2× 84 0.5× 158 1.1× 216 1.6× 74 0.9× 22 818
Haiming Ding United States 15 514 1.5× 92 0.6× 79 0.6× 162 1.2× 45 0.5× 30 898
Ming‐Ting Lee Taiwan 11 339 1.0× 72 0.5× 138 1.0× 153 1.2× 62 0.8× 19 712
Desanka Výbohová Slovakia 17 288 0.8× 83 0.5× 101 0.7× 112 0.8× 49 0.6× 30 649
Syed Sultan Beevi India 14 420 1.2× 152 1.0× 102 0.7× 51 0.4× 65 0.8× 35 845
J. Wahler United States 12 218 0.6× 70 0.5× 81 0.6× 187 1.4× 59 0.7× 15 512
Dong Yeok Shin South Korea 14 383 1.1× 97 0.6× 61 0.4× 76 0.6× 73 0.9× 25 632
Jiawen Yu China 13 505 1.5× 74 0.5× 104 0.8× 78 0.6× 60 0.7× 27 819
Kousuke Ishino Japan 16 272 0.8× 49 0.3× 137 1.0× 77 0.6× 85 1.0× 32 676

Countries citing papers authored by Abdul Basit Baba

Since Specialization
Citations

This map shows the geographic impact of Abdul Basit Baba's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Abdul Basit Baba with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Abdul Basit Baba more than expected).

Fields of papers citing papers by Abdul Basit Baba

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Abdul Basit Baba. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Abdul Basit Baba. The network helps show where Abdul Basit Baba may publish in the future.

Co-authorship network of co-authors of Abdul Basit Baba

This figure shows the co-authorship network connecting the top 25 collaborators of Abdul Basit Baba. A scholar is included among the top collaborators of Abdul Basit Baba based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Abdul Basit Baba. Abdul Basit Baba is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Baba, Abdul Basit, et al.. (2024). The Neem Limonoid Nimbolide Modulates Key Components of the DNA Damage Response Signalling in Cellular and Animal Models of Oral Squamous Cell Carcinoma. Current Pharmaceutical Biotechnology. 26(3). 428–442. 1 indexed citations
2.
Rah, Bilal, Rafiq A. Rather, Gh Rasool Bhat, et al.. (2022). JAK/STAT Signaling: Molecular Targets, Therapeutic Opportunities, and Limitations of Targeted Inhibitions in Solid Malignancies. Frontiers in Pharmacology. 13. 821344–821344. 98 indexed citations
3.
Baba, Abdul Basit, et al.. (2022). Transforming Growth Factor-Beta (TGF-β) Signaling in Cancer-A Betrayal Within. Frontiers in Pharmacology. 13. 791272–791272. 139 indexed citations breakdown →
4.
Baba, Abdul Basit, et al.. (2022). Nimbolide, a Neem Limonoid, Inhibits Angiogenesis in Breast Cancer by Abrogating Aldose Reductase Mediated IGF-1/PI3K/Akt Signalling. Anti-Cancer Agents in Medicinal Chemistry. 22(14). 2619–2636. 13 indexed citations
5.
Rather, Rafiq A., Ifra Mushtaq, Ajaz Ahmad Malik, et al.. (2021). Helicobacter pylori Subdues Cytokine Signaling to Alter Mucosal Inflammation via Hypermethylation of Suppressor of Cytokine Signaling 1 Gene During Gastric Carcinogenesis. Frontiers in Oncology. 10. 604747–604747. 19 indexed citations
6.
Tanagala, Kranthi Kiran Kishore, Abdul Basit Baba, Kowshik Jaganathan, G. Bhanuprakash Reddy, & Siddavaram Nagini. (2019). Gedunin, A Neem Limonoid in Combination with Epalrestat Inhibits Cancer Hallmarks by Attenuating Aldose Reductase-Driven Oncogenic Signaling in SCC131 Oral Cancer Cells. Anti-Cancer Agents in Medicinal Chemistry. 18(14). 2042–2052. 20 indexed citations
7.
Jaganathan, Kowshik, Abdul Basit Baba, Gordana Joksiċ, et al.. (2019). Activation of PI3K/Akt/NF-kB Signaling Mediates Swedish Snus Induced Proliferation and Apoptosis Evasion in the Rat Forestomach: Modulation by Blueberry. Anti-Cancer Agents in Medicinal Chemistry. 20(1). 59–69. 11 indexed citations
8.
Baba, Abdul Basit, et al.. (2018). Impact of stainless-steel welding fumes on proteins and non-coding RNAs regulating DNA damage response in the respiratory tract of Sprague-Dawley rats. Journal of Toxicology and Environmental Health. 81(24). 1231–1245. 3 indexed citations
9.
Baba, Abdul Basit, et al.. (2017). Blueberry and malvidin inhibit cell cycle progression and induce mitochondrial-mediated apoptosis by abrogating the JAK/STAT-3 signalling pathway. Food and Chemical Toxicology. 109(Pt 1). 534–543. 45 indexed citations
10.
11.
Jaganathan, Kowshik, et al.. (2014). Astaxanthin Inhibits JAK/STAT-3 Signaling to Abrogate Cell Proliferation, Invasion and Angiogenesis in a Hamster Model of Oral Cancer. PLoS ONE. 9(10). e109114–e109114. 128 indexed citations
12.
Kavitha, K., et al.. (2013). Astaxanthin inhibits NF-κB and Wnt/β-catenin signaling pathways via inactivation of Erk/MAPK and PI3K/Akt to induce intrinsic apoptosis in a hamster model of oral cancer. Biochimica et Biophysica Acta (BBA) - General Subjects. 1830(10). 4433–4444. 167 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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