Aastha Mishra
- Genetics top 10%
- Molecular Biology
- Endocrine and Autonomic Systems top 10%
- Pulmonary and Respiratory Medicine
- Pathology and Forensic Medicine
- Co-authors
- Mohammad Zahid AshrafM. A. Qadar PashaGhulam MohammadRahul KumarSamantha KohliPerwez AlamArpana VibhutiManvendra K. Singh
- Topics
- High Altitude and Hypoxia (12 papers)Cancer, Hypoxia, and Metabolism (6 papers)Eicosanoids and Hypertension Pharmacology (4 papers)
- Journals
- Proceedings of the National Academy of SciencesSHILAP Revista de lepidopterologíaPLoS ONE
- Partner nations
- IndiaUnited StatesCanada
In The Last Decade
Aastha Mishra
25 papers receiving 478 citations
Peers
Comparison fields: 5 of 79
- Genetics 196
- Molecular Biology 108
- Endocrine and Autonomic Systems 84
- Pulmonary and Respiratory Medicine 81
- Pathology and Forensic Medicine 77
Countries citing papers authored by Aastha Mishra
This map shows the geographic impact of Aastha Mishra's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Aastha Mishra with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Aastha Mishra more than expected).
Fields of papers citing papers by Aastha Mishra
This network shows the impact of papers produced by Aastha Mishra. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Aastha Mishra. The network helps show where Aastha Mishra may publish in the future.
Co-authorship network of co-authors of Aastha Mishra
This figure shows the co-authorship network connecting the top 25 collaborators of Aastha Mishra. A scholar is included among the top collaborators of Aastha Mishra based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Aastha Mishra. Aastha Mishra is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 5 | |
| 3 | 1 | |
| 4 | 4 | |
| 5 | 7 | |
| 6 | 7 | |
| 7 | 9 | |
| 8 | 13 | |
| 9 | 5 | |
| 10 | 1 | |
| 11 | 35 | |
| 12 | 9 | |
| 13 | 23 | |
| 14 | 94 | |
| 15 | 31 | |
| 16 | 16 | |
| 17 | 42 | |
| 18 | 39 | |
| 19 | 24 | |
| 20 | 48 |
About Aastha Mishra
Aastha Mishra is a scholar working on Internal Medicine, Health Informatics and Endocrine and Autonomic Systems, having authored 30 papers that have together received 483 indexed citations. Recurring topics across this work include High Altitude and Hypoxia (12 papers), Cancer, Hypoxia, and Metabolism (6 papers) and Eicosanoids and Hypertension Pharmacology (4 papers). The work is most often cited by research in Endocrine and Autonomic Systems (84 citations), Genetics (196 citations) and Internal Medicine (23 citations). Aastha Mishra has collaborated with scholars based in India, United States and Canada. Frequent co-authors include Mohammad Zahid Ashraf, M. A. Qadar Pasha, Ghulam Mohammad, Rahul Kumar, Samantha Kohli, Perwez Alam, Arpana Vibhuti, Manvendra K. Singh, Desh Deepak and Bhawani Singh. Their work appears in journals such as Proceedings of the National Academy of Sciences, SHILAP Revista de lepidopterología and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.