A. Mazo

775 total citations
19 papers, 577 citations indexed

About

A. Mazo is a scholar working on Molecular Biology, Oncology and Biotechnology. According to data from OpenAlex, A. Mazo has authored 19 papers receiving a total of 577 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 8 papers in Oncology and 6 papers in Biotechnology. Recurrent topics in A. Mazo's work include Cancer Research and Treatments (6 papers), Cancer-related Molecular Pathways (6 papers) and Virus-based gene therapy research (5 papers). A. Mazo is often cited by papers focused on Cancer Research and Treatments (6 papers), Cancer-related Molecular Pathways (6 papers) and Virus-based gene therapy research (5 papers). A. Mazo collaborates with scholars based in Spain, United States and France. A. Mazo's co-authors include Matilde E. Lleonart, Laura López-Vicente, S. Simonetti, Josep Castellví, Cleofé Romagosa, Santiago Ramón y Cajal, Josep Lluís Gelpí, Y. Fujii, Antonio Cortés and María Escribano and has published in prestigious journals such as Oncogene, Biochemical Journal and Annals of Oncology.

In The Last Decade

A. Mazo

19 papers receiving 569 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
A. Mazo Spain	10	247	242	114	89	86	19	577
Wan-Li Liu China	14	507 2.1×	342 1.4×	111 1.0×	85 1.0×	172 2.0×	18	868
Payel Bhanja United States	12	339 1.4×	202 0.8×	77 0.7×	46 0.5×	140 1.6×	25	782
Sally E. Dowen United Kingdom	9	336 1.4×	278 1.1×	105 0.9×	188 2.1×	179 2.1×	10	642
Patrick Roncarati Belgium	15	280 1.1×	251 1.0×	97 0.9×	197 2.2×	155 1.8×	21	766
Neil Kernohan United Kingdom	11	244 1.0×	210 0.9×	66 0.6×	64 0.7×	60 0.7×	22	528
Hermann-Josef Gröne Germany	9	228 0.9×	103 0.4×	62 0.5×	150 1.7×	64 0.7×	9	496
Ryan C. Chai Australia	12	299 1.2×	124 0.5×	74 0.6×	51 0.6×	91 1.1×	22	486
Andreas P. Kyriazis United States	15	270 1.1×	319 1.3×	149 1.3×	50 0.6×	151 1.8×	32	748
Ran-Yi Liu China	18	487 2.0×	298 1.2×	47 0.4×	130 1.5×	242 2.8×	26	882
Gustavo Nóriz Berardinelli Brazil	14	333 1.3×	328 1.4×	90 0.8×	62 0.7×	201 2.3×	35	696

Countries citing papers authored by A. Mazo

Since Specialization

Citations

This map shows the geographic impact of A. Mazo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A. Mazo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A. Mazo more than expected).

Fields of papers citing papers by A. Mazo

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A. Mazo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A. Mazo. The network helps show where A. Mazo may publish in the future.

Co-authorship network of co-authors of A. Mazo

This figure shows the co-authorship network connecting the top 25 collaborators of A. Mazo. A scholar is included among the top collaborators of A. Mazo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with A. Mazo. A. Mazo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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19 of 19 papers shown

Pérez‐Torras, Sandra, et al.. (2013). Concentrative nucleoside transporter 1 (hCNT1) promotes phenotypic changes relevant to tumor biology in a translocation-independent manner. Cell Death and Disease. 4(5). e648–e648. 23 indexed citations

Romagosa, Cleofé, S. Simonetti, Laura López-Vicente, et al.. (2011). p16Ink4a overexpression in cancer: a tumor suppressor gene associated with senescence and high-grade tumors. Oncogene. 30(18). 2087–2097. 345 indexed citations

Pérez‐Torras, Sandra, August Vidal, Jaume Adán, et al.. (2011). Dual effects of β3 integrin subunit expression on human pancreatic cancer models. Cellular Oncology. 34(4). 393–405. 5 indexed citations

Pérez‐Torras, Sandra, et al.. (2010). Dual Effects ofβ₃Integrin Subunit Expression on Human Pancreatic Cancer Models. Analytical Cellular Pathology. 33(5-6). 191–205. 2 indexed citations

Peñuelas, Silvia, Manel Cascalló, José Luís Hernández, et al.. (2004). Expression Profiles of a Human Pancreatic Cancer Cell Line upon Induction of Apoptosis Search for Modulators in Cancer Therapy. Oncology. 67(3-4). 277–290. 10 indexed citations

Calbó, Joaquim, et al.. (2004). The fate of pancreatic tumor cell lines following p16 overexpression depends on the modulation of CDK2 activity. Cell Death and Differentiation. 11(10). 1055–1065. 18 indexed citations

Calbó, Joaquim & A. Mazo. (2003). p16INK4a as a tumor suppressor with therapeutic applicability. Drugs of the Future. 28(2). 153–153. 2 indexed citations

Mercadé, Elena, Manel Cascalló, Meritxell Carrió, et al.. (2001). Treatment based on a combination of the CYP2B1/cyclophosphamide system and p53 delivery enhances tumour regression in human pancreatic cancer.. Annals of Oncology. 12(3). 379–388. 8 indexed citations

Cascalló, Manel, Elena Mercadé, Gabriel Capellá, et al.. (1999). Genetic background determines the response to adenovirus-mediated wild-type p53 expression in pancreatic tumor cells. Cancer Gene Therapy. 6(5). 428–436. 19 indexed citations

10.

Carrió, Meritxell, Anna Romagosa, Elena Mercadé, et al.. (1999). Enhanced pancreatic tumor regression by a combination of adenovirus and retrovirus-mediated delivery of the herpes simplex virus thymidine kinase gene. Gene Therapy. 6(4). 547–553. 38 indexed citations

11.

Langa, Fernando, et al.. (1993). Retention of the fetoacinar pancreatic (FAP) protein to the endoplasmic reticulum of tumor cells.. PubMed. 60(1). 115–21. 15 indexed citations

12.

Gelpí, Josep Lluís, et al.. (1992). Kinetic studies of the regulation of mitochondrial malate dehydrogenase by citrate. Biochemical Journal. 283(1). 289–297. 40 indexed citations

13.

Mazo, A., et al.. (1991). Expression of Fetoacinar Pancreatic (FAP) Protein in the Pancreatic Human Tumor Cell Line BxPC-3. Pancreas. 6(1). 37–45. 35 indexed citations

14.

Haro, Isabel, Josep Lluís Gelpí, A. Mazo, & Antonio Cortés. (1990). Kinetic Formulations for the Reduction of Ketomalonate by Lactate Dehydrogenase. Journal of enzyme inhibition. 3(3). 189–193. 1 indexed citations

15.

Mazo, A., et al.. (1990). Comparison of the kinetic behaviour of lactate dehydrogenase and cytosolic and mitochondrial malate dehydrogenase from guinea pig skeletal muscle. Journal of Molecular Catalysis. 58(2). 269–275. 1 indexed citations

16.

Mazo, A., et al.. (1990). Factors affecting L-malate activation of mitochondrial malate dehydrogenase from chicken liver.. PubMed. 20(1). 177–82. 1 indexed citations

17.

Gelpí, Josep Lluís, et al.. (1988). Purification of malate dehydrogenase from chicken liver mitochondria. existence of a small quantity of cytosolic isoenzyme. International Journal of Biochemistry. 20(9). 989–996. 2 indexed citations

18.

Mazo, A., et al.. (1987). Factors affecting malate dehydrogenase activity in freezing-thawing processes. Comparative Biochemistry and Physiology Part B Comparative Biochemistry. 88(2). 461–466. 2 indexed citations

19.

Mazo, A., et al.. (1980). Replication of transformation-defective mutants of the Prague strain of Rous sarcoma virus and isolation of a td mutant from duck-adapted PR-RSV-C.. PubMed. 26(1). 34–41. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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