A G Hinnebusch

4.3k citations

26 papers · 3.6k indexed · 1 hit paper · h-index 22

Molecular Biology top 2%
Cell Biology top 2%
Genetics top 10%
Plant Science top 10%
Immunology top 10%

Co-authors: Ronald C. Wek Thomas F. Donahue A. Mark Cigan Thomas Dever Lan Feng G R Fink Paul Miller Belinda M. Jackson
Topics: RNA and protein synthesis mechanisms (23 papers)RNA Research and Splicing (14 papers)RNA regulation and disease (11 papers)
Cited by: Molecular Biology Cell Biology Aging
Journals: Cell Proceedings of the National Academy of Sciences Nucleic Acids Research
Partner nations: United States Hungary Cameroon

In The Last Decade

A G Hinnebusch

26 papers receiving 3.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Phosphorylation of initiation factor 2α by protein kinase GCN2 mediates gene-specific translational control of GCN4 in yeast

1992 665 citations Thomas Dever, Lan Feng et al. Cell profile →

Peers

Countries citing papers authored by A G Hinnebusch

Since Specialization

Citations

This map shows the geographic impact of A G Hinnebusch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A G Hinnebusch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A G Hinnebusch more than expected).

Fields of papers citing papers by A G Hinnebusch

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A G Hinnebusch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A G Hinnebusch. The network helps show where A G Hinnebusch may publish in the future.

Co-authorship network of co-authors of A G Hinnebusch

This figure shows the co-authorship network connecting the top 25 collaborators of A G Hinnebusch. A scholar is included among the top collaborators of A G Hinnebusch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with A G Hinnebusch. A G Hinnebusch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Enhanced Interaction between Pseudokinase and Kinase Domains in Gcn2 stimulates eIF2α Phosphorylation in Starved Cells 2014 ·PLoS Genetics ·Sébastien Lageix,Stefan Rothenburg,Thomas Dever,A G Hinnebusch	29
2	An upstream ORF with non-AUG start codon is translated in vivo but dispensable for translational control of GCN4 mRNA 2011 ·Nucleic Acids Research ·Fan Zhang,A G Hinnebusch	26
3	Uncharged tRNA Activates GCN2 by Displacing the Protein Kinase Moiety from a Bipartite tRNA-Binding Domain 2000 ·Molecular Cell ·Jinsheng Dong,Hongfang Qiu,Minerva T. Garcia-Barrio,James T. Anderson,A G Hinnebusch	397
4	eIF2 independently binds two distinct eIF2B subcomplexes that catalyze and regulate guanine-nucleotide exchange 1998 ·Genes & Development ·Graham D. Pavitt,Kolluru V.A. Ramaiah,Scot R. Kimball,A G Hinnebusch	215
5	The eIF-2α kinases: regulators of protein synthesis in starvation and stress 1994 ·PubMed ·A G Hinnebusch	123
6	Requirements for intercistronic distance and level of eukaryotic initiation factor 2 activity in reinitiation on GCN4 mRNA vary with the downstream cistron. 1994 ·Molecular and Cellular Biology ·Chris M. Grant,Paul Miller,A G Hinnebusch	92
7	GCN1, a translational activator of GCN4 in Saccharomyces cerevisiae, is required for phosphorylation of eukaryotic translation initiation factor 2 by protein kinase GCN2. 1993 ·Molecular and Cellular Biology ·Matthew J. Marton,(unknown),A G Hinnebusch	113
8	Mammalian eukaryotic initiation factor 2 alpha kinases functionally substitute for GCN2 protein kinase in the GCN4 translational control mechanism of yeast. 1993 ·Proceedings of the National Academy of Sciences ·Thomas Dever,(unknown),Glen N. Barber,A. Mark Cigan,Lan Feng,Thomas F. Donahue,Irving M. London,Michael G. Katze,A G Hinnebusch	196
9	Translational regulation by the interferon-induced double-stranded-RNA-activated 68-kDa protein kinase. 1993 ·Proceedings of the National Academy of Sciences ·Glen N. Barber,Marlene Wambach,Min‐Liang Wong,Thomas Dever,A G Hinnebusch,Michael G. Katze	85
10	Phosphorylation of initiation factor 2α by protein kinase GCN2 mediates gene-specific translational control of GCN4 in yeastbreakdown → 1992 ·Cell ·Thomas Dever,Lan Feng,Ronald C. Wek,A. Mark Cigan,Thomas F. Donahue,A G Hinnebusch	665
11	Ribosome association of GCN2 protein kinase, a translational activator of the GCN4 gene of Saccharomyces cerevisiae. 1991 ·Molecular and Cellular Biology ·Manuel Ramírez,Ronald C. Wek,A G Hinnebusch	146
12	cis-Acting sequences involved in the translational control of GCN4 expression 1990 ·Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression ·Paul Miller,A G Hinnebusch	18
13	Transcriptional and Translational Regulation of Gene Expression in the General Control of Amino-Acid Biosynthesis in Saccharomyces cerevisiae 1990 ·Progress in nucleic acid research and molecular biology ·A G Hinnebusch	56
14	gcd12 mutations are gcn3-dependent alleles of GCD2, a negative regulator of GCN4 in the general amino acid control of Saccharomyces cerevisiae. 1989 ·Genetics ·Christopher J. Paddon,A G Hinnebusch	21
15	Amino acid sequence similarity between GCN3 and GCD2, positive and negative translational regulators of GCN4: evidence for antagonism by competition. 1989 ·Genetics ·Christopher J. Paddon,Ernest M. Hannig,A G Hinnebusch	32
16	Juxtaposition of domains homologous to protein kinases and histidyl-tRNA synthetases in GCN2 protein suggests a mechanism for coupling GCN4 expression to amino acid availability. 1989 ·Proceedings of the National Academy of Sciences ·Ronald C. Wek,Belinda M. Jackson,A G Hinnebusch	241
17	Sequences that surround the stop codons of upstream open reading frames in GCN4 mRNA determine their distinct functions in translational control. 1989 ·Genes & Development ·Paul Miller,A G Hinnebusch	104
18	Mutations in the structural genes for eukaryotic initiation factors 2 alpha and 2 beta of Saccharomyces cerevisiae disrupt translational control of GCN4 mRNA. 1989 ·Proceedings of the National Academy of Sciences ·N. P. Williams,A G Hinnebusch,Thomas F. Donahue	90
19	A synthetic HIS4 regulatory element confers general amino acid control on the cytochrome c gene (CYC1) of yeast. 1985 ·Proceedings of the National Academy of Sciences ·A G Hinnebusch,Giovanna Lucchini,Gerald R. Fink	135
20	Evidence for translational regulation of the activator of general amino acid control in yeast. 1984 ·Proceedings of the National Academy of Sciences ·A G Hinnebusch	415

About A G Hinnebusch

A G Hinnebusch is a scholar working on Molecular Biology, Genetics and Cell Biology, having authored 26 papers that have together received 3.6k indexed citations. Recurring topics across this work include RNA and protein synthesis mechanisms (23 papers), RNA Research and Splicing (14 papers) and RNA regulation and disease (11 papers). The work is most often cited by research in Molecular Biology (3.3k citations), Cell Biology (660 citations) and Aging (51 citations). A G Hinnebusch has collaborated with scholars based in United States, Hungary and Cameroon. Frequent co-authors include Ronald C. Wek, Thomas F. Donahue, A. Mark Cigan, Thomas Dever, Lan Feng, G R Fink, Paul Miller, Belinda M. Jackson, Jinsheng Dong and James T. Anderson. Their work appears in journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

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