A. A. Cole

1.6k total citations
27 papers, 1.3k citations indexed

About

A. A. Cole is a scholar working on Rheumatology, Cancer Research and Surgery. According to data from OpenAlex, A. A. Cole has authored 27 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Rheumatology, 8 papers in Cancer Research and 6 papers in Surgery. Recurrent topics in A. A. Cole's work include Osteoarthritis Treatment and Mechanisms (18 papers), Protease and Inhibitor Mechanisms (8 papers) and Cell Adhesion Molecules Research (6 papers). A. A. Cole is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (18 papers), Protease and Inhibitor Mechanisms (8 papers) and Cell Adhesion Molecules Research (6 papers). A. A. Cole collaborates with scholars based in United States, Germany and United Kingdom. A. A. Cole's co-authors include Klaus E. Kuettner, Linda M. Walters, Susan Chubinskaya, Katalin Mikecz, Dale R. Sumner, K. Huch, Karen A. Hasty, Gabriella Cs‐Szabó, Christopher B. Forsyth and Alan J. Grodzinsky and has published in prestigious journals such as Annals of the New York Academy of Sciences, Cellular and Molecular Life Sciences and The Journals of Gerontology Series A.

In The Last Decade

A. A. Cole

26 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
A. A. Cole United States 17 717 318 268 219 215 27 1.3k
G. Weseloh Germany 20 1.2k 1.7× 338 1.1× 377 1.4× 161 0.7× 118 0.5× 46 1.7k
Harumoto Yamada Japan 22 518 0.7× 614 1.9× 433 1.6× 157 0.7× 93 0.4× 76 1.6k
Sander M. Botter Switzerland 16 473 0.7× 211 0.7× 424 1.6× 108 0.5× 178 0.8× 34 1.1k
Veronica Ulici United States 20 581 0.8× 266 0.8× 509 1.9× 135 0.6× 93 0.4× 35 1.2k
A. E. M. Holthuysen Netherlands 23 1.1k 1.5× 195 0.6× 556 2.1× 123 0.6× 56 0.3× 26 1.8k
J.S. Kuliwaba Australia 23 656 0.9× 398 1.3× 821 3.1× 534 2.4× 312 1.5× 44 2.0k
Lynda O’Rear United States 15 365 0.5× 293 0.9× 593 2.2× 104 0.5× 147 0.7× 22 1.4k
Danka Grčević Croatia 27 383 0.5× 261 0.8× 1.0k 3.9× 282 1.3× 178 0.8× 84 1.9k
Robert Dinser Germany 16 779 1.1× 229 0.7× 474 1.8× 52 0.2× 54 0.3× 32 1.7k
Gary Gibson United States 16 449 0.6× 221 0.7× 711 2.7× 420 1.9× 133 0.6× 27 1.5k

Countries citing papers authored by A. A. Cole

Since Specialization
Citations

This map shows the geographic impact of A. A. Cole's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A. A. Cole with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A. A. Cole more than expected).

Fields of papers citing papers by A. A. Cole

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A. A. Cole. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A. A. Cole. The network helps show where A. A. Cole may publish in the future.

Co-authorship network of co-authors of A. A. Cole

This figure shows the co-authorship network connecting the top 25 collaborators of A. A. Cole. A scholar is included among the top collaborators of A. A. Cole based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with A. A. Cole. A. A. Cole is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Plaas, Anna, et al.. (2011). The relationship between fibrogenic TGFβ1 signaling in the joint and cartilage degradation in post-injury osteoarthritis. Osteoarthritis and Cartilage. 19(9). 1081–1090. 52 indexed citations
2.
Patwari, Parth, et al.. (2009). Potent inhibition of cartilage biosynthesis by coincubation with joint capsule through an IL‐1‐independent pathway. Scandinavian Journal of Medicine and Science in Sports. 19(4). 528–535. 18 indexed citations
3.
HAKOBYAN, N., et al.. (2008). Experimental haemophilic arthropathy in a mouse model of a massive haemarthrosis: gross, radiological and histological changes. Haemophilia. 14(4). 804–809. 77 indexed citations
4.
Aurich, Matthias, Wolfgang Eger, Bernd Rolauffs, et al.. (2006). Sprunggelenkchondrozyten besitzen eine höhere Interleukin-1-Resistenz als Kniegelenkchondrozyten. Der Orthopäde. 35(7). 784–790. 11 indexed citations
5.
Cole, A. A., et al.. (2005). Chondrocytes, synoviocytes and dermal fibroblasts all express PH-20, a hyaluronidase active at neutral pH. Arthritis Research & Therapy. 7(4). R756–68. 25 indexed citations
6.
Fichter, Michael, et al.. (2005). Collagen degradation products modulate matrix metalloproteinase expression in cultured articular chondrocytes. Journal of Orthopaedic Research®. 24(1). 63–70. 79 indexed citations
7.
Kuettner, Klaus E. & A. A. Cole. (2005). Cartilage degeneration in different human joints. Osteoarthritis and Cartilage. 13(2). 93–103. 169 indexed citations
8.
Forsyth, Christopher B., A. A. Cole, Gillian Murphy, et al.. (2005). Increased Matrix Metalloproteinase-13 Production With Aging by Human Articular Chondrocytes in Response to Catabolic Stimuli. The Journals of Gerontology Series A. 60(9). 1118–1124. 100 indexed citations
9.
Cole, A. A., et al.. (2003). Comparison of the catabolic effects of fibronectin fragments in human knee and ankle cartilages. Osteoarthritis and Cartilage. 11(7). 538–547. 30 indexed citations
10.
Kerin, Alex J., Parth Patwari, Klaus E. Kuettner, A. A. Cole, & Alan J. Grodzinsky. (2002). Molecular basis of osteoarthritis: biomechanical aspects. Cellular and Molecular Life Sciences. 59(1). 27–35. 70 indexed citations
11.
Han, Bo, et al.. (2002). Early alterations in the collagen meshwork and lesions in the ankles are associated with spontaneous osteoarthritis in guinea-pigs. Osteoarthritis and Cartilage. 10(10). 778–784. 25 indexed citations
12.
Mollenhauer, J., Matthias Aurich, Z. Zhong, et al.. (2002). Diffraction-enhanced X-ray imaging of articular cartilage. Osteoarthritis and Cartilage. 10(3). 163–171. 115 indexed citations
13.
Muehleman, Carol, Holger Koepp, Wolfgang Eger, et al.. (2002). Bone density of the human talus does not increase with the cartilage degeneration score. The Anatomical Record. 266(2). 81–86. 16 indexed citations
14.
Cole, A. A. & Klaus E. Kuettner. (2002). Molecular basis for differences between human joints. Cellular and Molecular Life Sciences. 59(1). 19–26. 42 indexed citations
15.
16.
Muehleman, Carol, et al.. (1997). 1997 William J. Stickel Gold Award. Morphological and biochemical properties of metatarsophalangeal joint cartilage. Journal of the American Podiatric Medical Association. 87(10). 447–459. 11 indexed citations
17.
Cole, A. A., et al.. (1997). Tetracycline derivatives inhibit cartilage degradation in cultured embryonic chick tibiae. Research in Veterinary Science. 63(1). 11–14. 7 indexed citations
18.
Cole, A. A., et al.. (1994). Doxycycline Inhibition of Cartilage Matrix Degradationa. Annals of the New York Academy of Sciences. 732(1). 414–415. 2 indexed citations
19.
Cole, A. A., Klaus E. Kuettner, & Thomas Schmid. (1992). Metalloproteinases in regions of the embryonic chick tibiotarsus.. PubMed. 1. 393–4.
20.
Cole, A. A., et al.. (1989). Are perivascular cells in cartilage canals chondrocytes?. PubMed. 165. 1–8. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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