Hui‐Chen Ku

Quercetin and Epigallocatechin Gallate in the Prevention and Treatment of Obesity: From Molecular to Clinical Studies

The Epidemiology of Obesity: A Big Picture

Adipocytes under assault: Environmental disruption of adipose physiology

Laboratory, Epidemiological, and Human Intervention Studies Show That Tea (Camellia sinensis) May Be Useful in the Prevention of Obesity ,

NRF2 and NF-қB interplay in cerebrovascular and neurodegenerative disorders: Molecular mechanisms and possible therapeutic approaches

Epigallocatechin-3-gallate suppresses cell proliferation and promotes apoptosis and autophagy in oral cancer SSC-4 cells

Defining roles of specific reactive oxygen species (ROS) in cell biology and physiology

Epigallocatechin-3-Gallate Improves Acne in Humans by Modulating Intracellular Molecular Targets and Inhibiting P. acnes

The antioxidant and pro-oxidant activities of green tea polyphenols: A role in cancer prevention

Plant-derived immunomodulators: an insight on their preclinical evaluation and clinical trials

Physiological Implications of Hydrogen Sulfide: A Whiff Exploration That Blossomed

PGC-1α, Inflammation, and Oxidative Stress: An Integrative View in Metabolism

EDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting Chemicals

Apoptosis: A Target for Anticancer Therapy

The development of anticancer ruthenium(ii) complexes: from single molecule compounds to nanomaterials

Epigallocatechin-3-gallate prevents TNF-α-induced NF-κB activation thereby upregulating ABCA1 via the Nrf2/Keap1 pathway in macrophage foam cells

Therapeutic Potential of Quercetin: New Insights and Perspectives for Human Health

Improving the Anticancer Efficacy of Laminin Receptor-Specific Therapeutic Ruthenium Nanoparticles (RuBB-Loaded EGCG-RuNPs) via ROS-Dependent Apoptosis in SMMC-7721 Cells

ACS67, a Hydrogen Sulfide–Releasing Derivative of Latanoprost Acid, Attenuates Retinal Ischemia and Oxidative Stress to RGC-5 Cells in Culture

Endocrine Disruptors: From Endocrine to Metabolic Disruption

Green tea (−)-epigallocatechin gallate suppresses IGF-I and IGF-II stimulation of 3T3-L1 adipocyte glucose uptake via the glucose transporter 4, but not glucose transporter 1 pathway

Green Tea Epigallocatechin Gallate Inhibits Insulin Stimulation of Adipocyte Glucose Uptake via the 67-Kilodalton Laminin Receptor and AMP-Activated Protein Kinase Pathways

Octylphenol stimulates resistin gene expression in 3T3-L1 adipocytes via the estrogen receptor and extracellular signal-regulated kinase pathways

Green tea (−)-epigallocatechin gallate inhibits insulin stimulation of 3T3-L1 preadipocyte mitogenesis via the 67-kDa laminin receptor pathway

PGC-1α as a Pivotal Factor in Lipid and Metabolic Regulation

The effects of green tea (–)‐epigallocatechin‐3‐gallate on reactive oxygen species in 3T3‐L1 preadipocytes and adipocytes depend on the glutathione and 67 kDa laminin receptor pathways

Green tea (–)‐epigallocatechin gallate inhibits IGF‐I and IGF‐IIstimulation of 3T3‐L1 preadipocyte mitogenesis via the 67‐kDa laminin receptor, but not AMP‐activated protein kinase pathway