Exosome-mediated genetic reprogramming of tumor-associated macrophages by exoASO-STAT6 leads to potent monotherapy antitumor activity

Abstract

loading...

About

This paper, published in 1950, received 185 indexed citations. Written by Sushrut Kamerkar, Charan Leng, Olga Burenkova, Su Chul Jang, Christine McCoy, Kelvin Zhang, Kevin Dooley, Tong Zi, Sílvia Sisó and William K. Dahlberg covering the research area of Molecular Biology and Immunology. It is primarily cited by scholars working on Molecular Biology (141 citations), Cancer Research (88 citations) and Immunology (65 citations). Published in Science Advances.

Countries where authors are citing Exosome-mediated genetic reprogramming of tumor-associated macrophages by exoASO-STAT6 leads to potent monotherapy antitumor activity

Since Specialization
Citations

This map shows the geographic impact of Exosome-mediated genetic reprogramming of tumor-associated macrophages by exoASO-STAT6 leads to potent monotherapy antitumor activity. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Exosome-mediated genetic reprogramming of tumor-associated macrophages by exoASO-STAT6 leads to potent monotherapy antitumor activity with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Exosome-mediated genetic reprogramming of tumor-associated macrophages by exoASO-STAT6 leads to potent monotherapy antitumor activity more than expected).

Fields of papers citing Exosome-mediated genetic reprogramming of tumor-associated macrophages by exoASO-STAT6 leads to potent monotherapy antitumor activity

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of Exosome-mediated genetic reprogramming of tumor-associated macrophages by exoASO-STAT6 leads to potent monotherapy antitumor activity. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the Exosome-mediated genetic reprogramming of tumor-associated macrophages by exoASO-STAT6 leads to potent monotherapy antitumor activity.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

This paper is also available at doi.org/10.1126/sciadv.abj7002.

Explore hit-papers with similar magnitude of impact

Rankless by CCL
2026